Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Neha Diwanji"'
Autor:
Neha Diwanji, Andreas Bergmann
Publikováno v:
Nature Communications, Vol 11, Iss 1, Pp 1-14 (2020)
The molecular mechanisms regulating macrophage recruitment to tumors are unclear. Here, the authors use a Drosophila overgrowth model to show how damaged basement membranes recruit macrophages to undead tissue, via an interdependent effect of reactiv
Externí odkaz:
https://doaj.org/article/93c2f6af999545048616780f10a3bd53
Autor:
Neha Diwanji, Andreas Bergmann
Publikováno v:
Fly, Vol 11, Iss 1, Pp 46-52 (2017)
Apoptosis-induced proliferation (AiP) maintains tissue homeostasis following massive stress-induced cell death. During this phenomenon, dying cells induce proliferation of the surviving cells to compensate for the tissue loss, and thus restore organ
Externí odkaz:
https://doaj.org/article/77b69f97ab094ed1ae6aad3ac19380a6
Autor:
Neha Diwanji, Edward Cochran, Michael Gorgievski, Thomas Prod’homme, Yuxiao Wang, Bruce McCreedy, Daniel Getts
Publikováno v:
Regular and Young Investigator Award Abstracts.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::83f1704188231969888c1d18a07f6f20
https://doi.org/10.1136/jitc-2022-sitc2022.0393
https://doi.org/10.1136/jitc-2022-sitc2022.0393
Autor:
Michele Gerber, Thomas Prod'homme, Yuxiao Wang, Kyong-Rim Kieffer-Kwon, Neha Diwanji, Josephine D'Alessandro, Xing Du, Edward Cochran, Michael Gorgievski, Zheng Ling, Nicholas King, Siddhartha Mukherjee, Daniel Getts
Publikováno v:
Cancer Research. 83:CT131-CT131
For many patients with T cell lymphomas (TCL), sustained clinical benefit with standard treatment options is elusive. Myeloid cells, such as monocytes, dendritic cells and macrophages, readily accumulate in tumors, whereby they support tumor progress
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c25a7397d35260cb173426271806fd0f
https://doi.org/10.1158/1538-7445.am2023-ct131
https://doi.org/10.1158/1538-7445.am2023-ct131
Autor:
Neha Diwanji, Kyong-Rim Kieffer-Kwon, Thomas Prod’homme, Bruce McCreedy, Yuxiao Wang, Daniel Getts
Publikováno v:
Cancer Research. 82:LB005-LB005
For the majority of patients with advanced solid tumors, sustained clinical benefit with immunotherapy has yet to be achieved. Myeloid cells, including monocytes and macrophages are the primary orchestrators of immune responses and are found to accum
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ab0ec09d3803f1f028188606fc5e89d2
https://doi.org/10.1158/1538-7445.am2022-lb005
https://doi.org/10.1158/1538-7445.am2022-lb005
Autor:
Neha, Diwanji, Andreas, Bergmann
Publikováno v:
Advances in experimental medicine and biology. 1167
Apoptosis has long been regarded as a tumor suppressor mechanism and evasion from apoptosis is considered to be one hallmark of cancer. However, this principle is not always consistent with clinical data which often illustrate a correlation between a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b5ee2755da8abea6ad1836ec900e938f
https://pubmed.ncbi.nlm.nih.gov/31520349
https://pubmed.ncbi.nlm.nih.gov/31520349
Autor:
Andreas Bergmann, Neha Diwanji
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9783030236281
Apoptosis has long been regarded as a tumor suppressor mechanism and evasion from apoptosis is considered to be one hallmark of cancer. However, this principle is not always consistent with clinical data which often illustrate a correlation between a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7bb7fb4bc96cb18aa672fab7744454ad
https://doi.org/10.1007/978-3-030-23629-8_4
https://doi.org/10.1007/978-3-030-23629-8_4
Autor:
Andreas Bergmann, Neha Diwanji
Publikováno v:
Fly
Apoptosis-induced proliferation (AiP) maintains tissue homeostasis following massive stress-induced cell death. During this phenomenon, dying cells induce proliferation of the surviving cells to compensate for the tissue loss, and thus restore organ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1064390cec6d8063caeaf49747450841
https://doi.org/10.1080/19336934.2016.1222997
https://doi.org/10.1080/19336934.2016.1222997
Autor:
Andreas Bergmann, Neha Diwanji
Apoptosis-induced compensatory proliferation (AiP) is a form of compensatory proliferation that is triggered by apoptotic cell death to maintain tissue homeostasis. As such, AiP is essential for many tissue repair processes including regeneration. Th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1c1f90f08d1d0027fe260e0195d05ed
https://europepmc.org/articles/PMC5756134/
https://europepmc.org/articles/PMC5756134/
Autor:
Yun Fan, Jillian L. Lindblad, Alla Amcheslavsky, Meghana Tare, Kalpana Makhijani, Caitlin E. Fogarty, Neha Diwanji, Andreas Bergmann, Katja Brückner
Summary Apoptosis-induced proliferation (AiP) is a compensatory mechanism to maintain tissue size and morphology following unexpected cell loss during normal development, and may also be a contributing factor to cancer and drug resistance. In apoptot
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73049ecd7686d1fb55ebf550e3f2b90d
https://europepmc.org/articles/PMC4765900/
https://europepmc.org/articles/PMC4765900/