Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Nawal S. Gouda"'
Autor:
Amira M. Sultan, Nawal S. Gouda
Publikováno v:
Journal of Pure and Applied Microbiology, Vol 16, Iss 2, Pp 1179-1186 (2022)
Vancomycin resistant enterococci are challenging bacteria as they are difficult to be eradicated. Toxin-antitoxin (TA) systems are genetic elements located in most prokaryotic genomes. The mazEF TA system is harbored by a plasmid among Enterococcus f
Externí odkaz:
https://doaj.org/article/a198c2f98d134619a87c13508ef4b982
Autor:
Nouran B. AbdAllah, Eman A. Toraih, Essam Al Ageeli, Hala Elhagrasy, Nawal S. Gouda, Manal S. Fawzy, Ghada M. Helal
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-14 (2021)
Abstract Toll-like receptor (TLR) family signature has been implicated in sepsis etiopathology. We aimed to evaluate the genetic profile of TLR pathway-related key genes; the myeloid differentiation protein 88 (MYD88), IL1 receptor-associated kinase
Externí odkaz:
https://doaj.org/article/cb878c7b4af8494cbf9939490a57a6c2
Autor:
Abdelhady R. Abdel‐Gawad, Sameerah Shaheen, Nouf A. Babteen, Eman A. Toraih, Rami M. Elshazli, Manal S. Fawzy, Nawal S. Gouda
Publikováno v:
Immunity, Inflammation and Disease, Vol 8, Iss 4, Pp 595-604 (2020)
Abstract Objective MicroRNAs are large family clusters of small noncoding RNAs that implicated in genetic and epigenetic regulation of several immunological processes and pathways. As an epigenetic modifier, the microRNA 17‐92 cluster host gene (MI
Externí odkaz:
https://doaj.org/article/8469b8d3a4384bee97314c458619f60e
Autor:
Shymaa Ahmed Maher, Nader Ali Ismail, Eman A. Toraih, Alaa H. Habib, Nawal S. Gouda, Amal H. A. Gomaa, Manal S. Fawzy, Ghada M. Helal
Publikováno v:
Biomolecules, Vol 12, Iss 5, p 602 (2022)
Alopecia areata (AA) is a type of immune-mediated alopecia. Recent studies have suggested microRNAs’ (miRNAs) implication in several cellular processes, including epidermal and hair follicle biology. Single nucleotide polymorphisms (SNPs) can modif
Externí odkaz:
https://doaj.org/article/ec6d4c23705b49d9a5f60ec4b6138ff0
Autor:
Nawal S Gouda, Basem Salama
Publikováno v:
Medical Science. 26:1-8
Autor:
Basma Shouman, Abd Elazeez Shabaan, Nawal S. Gouda, Hesham Abdel-Hady, Manal M. Nomir, Nehad Nasef, Islam Nour, Mayada Sabry Zeid, Heba Eldegla, Noha Mostafa Mahmoud
Publikováno v:
African Journal of Microbiology Research. 15:286-294
Carbapenems are group of extended-spectrum β-lactam antimicrobials frequently used for treating multidrug-resistant Gram-negative bacilli (GNB) infections. This study aimed at detecting and characterizing carbapenem resistance (CR) genes among GNB i
Autor:
Nawal S. Gouda, Sameerah Shaheen, Rami M. Elshazli, Eman A. Toraih, Manal S. Fawzy, Nouf Abubakr Babteen, Abdelhady Ragab Abdel-Gawad
Publikováno v:
Immunity, Inflammation and Disease, Vol 8, Iss 4, Pp 595-604 (2020)
Immunity, Inflammation and Disease
Immunity, Inflammation and Disease
Objective MicroRNAs are large family clusters of small noncoding RNAs that implicated in genetic and epigenetic regulation of several immunological processes and pathways. As an epigenetic modifier, the microRNA 17‐92 cluster host gene (MIR17HG) ha
Publikováno v:
African Journal of Clinical and Experimental Microbiology. 21:88-96
Background : Studies have reported relationship between chronic Helicobacter pylori infection and coronary artery disease (CAD). The cytotoxin-associated gene A product (CagA) is an immunodominant protein which indicates infection with virulent H. py
Autor:
Nawal S. Gouda, Ghada M. Helal, Manal S. Fawzy, Hala F. Elhagrasy, Eman A. Toraih, Nouran B. AbdAllah, Essam Al Ageeli
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-14 (2021)
Scientific Reports
Scientific Reports
Toll-like receptor (TLR) family signature has been implicated in sepsis etiopathology. We aimed to evaluate the genetic profile of TLR pathway-related key genes; the myeloid differentiation protein 88 (MYD88), IL1 receptor-associated kinase 1 (IRAK1)
Publikováno v:
Journal of Clinical Laboratory Analysis
Background Cytotoxic T‐lymphocyte‐associated protein 4 (CTLA‐4) is one of the essential brakes expressed on T cells that prevent T‐cell hyperactivation‐associated autoimmune disorders. Several CTLA4 polymorphisms were implicated in the regu