Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Navneeta Pathak"'
Autor:
Veronica G. Beaudry, Rebecca A. Ihrie, Suzanne B. R. Jacobs, Bichchau Nguyen, Navneeta Pathak, Eunice Park, Laura D. Attardi
Publikováno v:
Dermatology Research and Practice, Vol 2010 (2010)
Epithelial wound closure is a complex biological process that relies on the concerted action of activated keratinocytes and dermal fibroblasts to resurface and close the exposed wound. Modulation of cell-cell adhesion junctions is thought to facilita
Externí odkaz:
https://doaj.org/article/16657c900fef40eb80e337e43439dd06
Autor:
Bichchau Nguyen, Suzanne B.R. Jacobs, Navneeta Pathak, Rebecca A. Ihrie, Veronica G. Beaudry, Laura D. Attardi, Eunice J. Park
Publikováno v:
Dermatology Research and Practice, Vol 2010 (2010)
Dermatology Research and Practice
Dermatology Research and Practice
Epithelial wound closure is a complex biological process that relies on the concerted action of activated keratinocytes and dermal fibroblasts to resurface and close the exposed wound. Modulation of cell-cell adhesion junctions is thought to facilita
Publikováno v:
American Journal of Medical Genetics Part A. :1952-1957
Ankyloblepharon Ectodermal Dysplasia and Cleft Lip/Palate (AEC) or Hay-Wells Syndrome is an autosomal dominant disorder characterized by a variety of phenotypes in ectodermal derivatives, including severe skin erosions, ankyloblepharon, coarse and wi
Publikováno v:
Proceedings of the National Academy of Sciences. 105:1215-1220
The contribution of transcriptional activation to the p53 effector functions critical for tumor suppression, apoptosis and cellular senescence, remains unclear because of p53's ability to regulate diverse cellular processes in a transactivation-indep
Publikováno v:
Cell Death & Differentiation. 14:1374-1385
p53 plays a central role in neuronal cell death resulting from acute injury or disease. To define the pathway by which p53 triggers apoptosis, we used microarray analysis to identify p53 target genes specifically upregulated during apoptosis but not
Autor:
Shuo Han, Colleen A. Brady, Veronica G. Beaudry, Thomas M. Johnson, Navneeta Pathak, Kathryn T. Bieging-Rolett, Laura D. Attardi
Publikováno v:
Oncogene
The p53 tumor suppressor is a stress sensor, driving cell cycle arrest or apoptosis in response to DNA damage or oncogenic signals. p53 activation by oncogenic signals relies on the p19(Arf) tumor suppressor, while p53 activation downstream of acute
The ability to degrade extracellular matrix is critical for tumor cells to invade and metastasize. Recent studies show that tumor cells utilize specialized actin-based membrane protrusions termed invadopodia to perform matrix degradation. Invadopodia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e1b7ee5cf816bb2ba429f0d2923198c
https://europepmc.org/articles/PMC3969876/
https://europepmc.org/articles/PMC3969876/
Autor:
Shashwati Basak, Philipp Kaldis, Amato J. Giaccia, Suzanne B.R. Jacobs, Navneeta Pathak, Qi Zeng, Adam J. Krieg, Laura D. Attardi
The p53 tumor suppressor restricts tumorigenesis through the transcriptional activation of target genes involved in cell-cycle arrest and apoptosis. Here, we identify Prl-3 (phosphatase of regenerating liver-3) as a p53-inducible gene. Whereas previo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d6c2dcbad9e8c4fd72a14fe69c328862
https://europepmc.org/articles/PMC3951836/
https://europepmc.org/articles/PMC3951836/