Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Natsuko Kumamoto"'
Autor:
Masato Sawada, Mami Matsumoto, Keishi Narita, Natsuko Kumamoto, Shinya Ugawa, Sen Takeda, Kazunobu Sawamoto
Publikováno v:
Bio-Protocol, Vol 10, Iss 22 (2020)
Neuronal migration is a critical step for the development of neuronal circuits in the brain. Immature new neurons (neuroblasts) generated in the postnatal ventricular-subventricular zone (V-SVZ) show a remarkable potential to migrate for a long dista
Externí odkaz:
https://doaj.org/article/a7f05a7827ae4d26880ad9f6468e39e9
Autor:
Daisuke Ieda, Yutaka Negishi, Tomomi Miyamoto, Yoshikazu Johmura, Natsuko Kumamoto, Kohji Kato, Ichiro Miyoshi, Makoto Nakanishi, Shinya Ugawa, Hisashi Oishi, Shinji Saitoh
Publikováno v:
PLoS ONE, Vol 15, Iss 8, p e0237814 (2020)
Schaaf-Yang syndrome (SYS) is a neurodevelopmental disorder caused by truncating variants in the paternal allele of MAGEL2, located in the Prader-Willi critical region, 15q11-q13. Although the phenotypes of SYS overlap those of Prader-Willi syndrome
Externí odkaz:
https://doaj.org/article/f3848521228e4611b929283e23d91d29
Publikováno v:
Nutrients, Vol 13, Iss 11, p 3749 (2021)
In addition to the taste receptors corresponding to the six basic taste qualities—sweet, salty, sour, bitter, umami, and fatty—another type of taste receptor, calcium-sensing receptor (CaSR), is found in taste-bud cells. CaSR is called the ‘kok
Externí odkaz:
https://doaj.org/article/4b21c44fd2eb497d991c106c2fe83a3f
Autor:
Masaya Watanabe, Takashi Ueda, Yasuhiro Shibata, Natsuko Kumamoto, Shoichi Shimada, Shinya Ugawa
Publikováno v:
PLoS ONE, Vol 10, Iss 5, p e0127572 (2015)
The Cav3.2 isoform of the T-type calcium channel is expressed in primary sensory neurons of the dorsal root ganglion (DRG), and these channels contribute to nociceptive and neuropathic pain in rats. However, there are conflicting reports on the roles
Externí odkaz:
https://doaj.org/article/4a94de76530f42cdb4405d8580160e12
Autor:
Ko Miyoshi, Kyosuke Kasahara, Shinki Murakami, Mika Takeshima, Natsuko Kumamoto, Asako Sato, Ikuko Miyazaki, Shinsuke Matsuzaki, Toshikuni Sasaoka, Taiichi Katayama, Masato Asanuma
Publikováno v:
PLoS ONE, Vol 9, Iss 5, p e97918 (2014)
In the rodent brain, certain G protein-coupled receptors and adenylyl cyclase type 3 are known to localize to the neuronal primary cilium, a primitive sensory organelle protruding singly from almost all neurons. A recent chemical screening study demo
Externí odkaz:
https://doaj.org/article/c1bd7b14dc8f47c2ae472cdde31326da
Autor:
Tsuyoshi Hattori, Shoko Shimizu, Yoshihisa Koyama, Hisayo Emoto, Yuji Matsumoto, Natsuko Kumamoto, Kohei Yamada, Hironori Takamura, Shinsuke Matsuzaki, Taiichi Katayama, Masaya Tohyama, Akira Ito
Publikováno v:
PLoS ONE, Vol 9, Iss 2, p e88506 (2014)
Disrupted-in-schizophrenia 1 (DISC1) is a gene disrupted by a translocation, t(1;11) (q42.1;q14.3), that segregates with major psychiatric disorders, including schizophrenia, recurrent major depression and bipolar affective disorder, in a Scottish fa
Externí odkaz:
https://doaj.org/article/8765681b92ae450e9c43867f6ae9b463
Autor:
Hiroaki Okuda, Ryusuke Kuwahara, Shinsuke Matsuzaki, Shingo Miyata, Natsuko Kumamoto, Tsuyoshi Hattori, Shoko Shimizu, Kohei Yamada, Keisuke Kawamoto, Ryota Hashimoto, Masatoshi Takeda, Taiichi Katayama, Masaya Tohyama
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8773 (2010)
BACKGROUND: An accumulating body of evidence suggests that Dtnbp1 (Dysbindin) is a key susceptibility gene for schizophrenia. Using the yeast-two-hybrid screening system, we examined the candidate proteins interacting with Dysbindin and revealed one
Externí odkaz:
https://doaj.org/article/f0a3043d11644344b9c97bc06f180ba0
Autor:
Kohei Yamada, Shinsuke Matsuzaki, Tsuyoshi Hattori, Ryusuke Kuwahara, Manabu Taniguchi, Hitoshi Hashimoto, Norihito Shintani, Akemichi Baba, Natsuko Kumamoto, Kazuo Yamada, Takeo Yoshikawa, Taiichi Katayama, Masaya Tohyama
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8596 (2010)
Pituitary adenylate cyclase-activating polypeptide (PACAP) is involved in multiple brain functions. To clarify the cause of abnormal behavior in PACAP deficient-mice, we attempted the identification of genes whose expression was altered in the dentat
Externí odkaz:
https://doaj.org/article/d68b3558a9d64d7cab3602a6687ff67a
Publikováno v:
NeuroReport. 34:238-248
Autor:
Yasuhiro Shibata, Natsuko Kumamoto, Eisuke Sakuma, Yusuke Ishida, Takashi Ueda, Shoichi Shimada, Shinya Ugawa
Publikováno v:
Biochemical and biophysical research communications. 610
Specific amino acid substitutions in degenerin mechano-gated channels (DEGs) of C. elegans convert these channels into constitutively active mutants that induce the degeneration of neurons where DEGs are expressed. Acid-sensing ion channel-2a (ASIC2a