Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Natsuko Hada"'
Publikováno v:
Pharmacology Research & Perspectives, Vol 11, Iss 3, Pp n/a-n/a (2023)
Abstract Ceramide is a central molecule of sphingolipid metabolism and is involved in the development of non‐alcoholic fatty liver disease (NAFLD) and non‐alcoholic steatohepatitis (NASH). It has already been reported that the inhibition of serin
Externí odkaz:
https://doaj.org/article/9f8c96cc6ff847988da6e427c6e0f9de
Autor:
Misato Kobayashi, Tamio Ohno, Natsuko Hada, Masato Fujiyoshi, Masako Kuga, Masahiko Nishimura, Atsushi Murai, Fumihiko Horio
Publikováno v:
Journal of Lipid Research, Vol 51, Iss 12, Pp 3463-3469 (2010)
Each abdominal fat depot, such as mesenteric or epididymal, differently contributes to the development of insulin resistance. The aim of this study was to identify the genetic regions that contribute to fat accumulation in epididymal/mesenteric fat a
Externí odkaz:
https://doaj.org/article/cff98f59de314539930ca5d6ea6b2ab5
Autor:
Yoshiyuki Sakamaki, Asao Katsume, Chiaki Tanaka, Tsuneo Ito, Masayuki Sudoh, Masahiko Matsumoto, Akiko Uno, Natsuko Hada, Toshihiko Shiga
Publikováno v:
International Journal of Experimental Pathology
Non-alcoholic steatohepatitis (NASH) is a progressive fibrotic disease, the pathogenesis of which has not been fully elucidated. One of the most common models used in NASH research is a nutritional model where NASH is induced by feeding a diet defici
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a052173cc9a4fc99124052cdbd33544
https://doi.org/10.1111/iep.12008
https://doi.org/10.1111/iep.12008
Autor:
Masato Fujiyoshi, Masahiko Nishimura, Atsushi Murai, Misato Kobayashi, Fumihiko Horio, Tamio Ohno, Masako Kuga, Natsuko Hada
Publikováno v:
Journal of Lipid Research, Vol 51, Iss 12, Pp 3463-3469 (2010)
Each abdominal fat depot, such as mesenteric or epididymal, differently contributes to the development of insulin resistance. The aim of this study was to identify the genetic regions that contribute to fat accumulation in epididymal/mesenteric fat a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::00e28456984a220ab81d45352d7b0d29
https://doi.org/10.1194/jlr.m009563
https://doi.org/10.1194/jlr.m009563
Autor:
Atsushi Murai, Kazuhiko Umeshita, Misato Kobayashi, Masahiko Nishimura, Hiromi Hoshino, Tamio Ohno, Fumihiko Horio, Natsuko Hada, Takashi Ozawa
Publikováno v:
Journal of Nutritional Science and Vitaminology. 55:257-263
The interaction between causative genes and diet is known to influence the onset of obesity and diabetes in humans, although it has remained difficult to identify diabetogenic gene(s) because humans are genetically and environmentally heterogeneous.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c317e8675d320864ef887571f1e68ed
https://doi.org/10.3177/jnsv.55.257
https://doi.org/10.3177/jnsv.55.257
Autor:
William Alazawi, Masayuki Sudoh, Michinori Kohara, Nobuo Shimma, Atsunori Ohta, Mikio Arisawa, Yuko Aoki, Asao Katsume, Natsuko Hada, Hitoshi Yoshino, Hiroshi Ohmori, Yuichi Hirata, Hiroshi Sakamoto, Kazumi Morikawa, Takuo Tsukuda, Motooki Ashihara, Yusuke Ohmori, Koichi Okamoto, Hideyuki Konishi, Graham R. Foster
Publikováno v:
Scientific Reports
Most acute hepatitis C virus (HCV) infections become chronic and some progress to liver cirrhosis or hepatocellular carcinoma. Standard therapy involves an interferon (IFN)-α-based regimen, and efficacy of therapy has been significantly improved by
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9277b180c8f39e1a1ef387254794377b
https://pubmed.ncbi.nlm.nih.gov/22355771
https://pubmed.ncbi.nlm.nih.gov/22355771
Autor:
Masato Fujiyoshi, Masako Kuga, Fumihiko Horio, Masahiko Nishimura, Akira Ishikawa, Misato Kobayashi, Tamio Ohno, Natsuko Hada, Shizufumi Ebihara
Publikováno v:
Physiological genomics. 35(1)
The SMXA-5 recombinant inbred strain, which was established from nondiabetic parental SM/J and A/J mice, develops diabetic phenotypes such as impaired glucose tolerance. The combination of diabetogenic genes in the SM/J and A/J genomes impairs glucos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9362322b79a9c0cc857f24e2eb9d6c6
https://pubmed.ncbi.nlm.nih.gov/18628340
https://pubmed.ncbi.nlm.nih.gov/18628340