Zobrazeno 1 - 10
of 58
pro vyhledávání: '"Natia, Qipshidze"'
Autor:
Kelm, Natia Qipshidze1,2 (AUTHOR), Solinger, Jane C.1 (AUTHOR), Piell, Kellianne M.1 (AUTHOR), Cole, Marsha P.1,2 (AUTHOR) marcie.cole@louisville.edu
Publikováno v:
International Journal of Molecular Sciences. Jul2023, Vol. 24 Issue 13, p11208. 10p.
Publikováno v:
Redox Biology, Vol 2, Iss C, Pp 1-7 (2014)
According to the CDC, the most common type of heart disease is coronary artery disease, which commonly leads to myocardial infarction (MI). Therapeutic approaches to lessen the resulting cardiovascular injury associated with MI are limited. Recently,
Externí odkaz:
https://doaj.org/article/70c46eec7ca24a27968b229e2bbc5a09
Publikováno v:
International Journal of Biological Sciences, Vol 8, Iss 4, Pp 430-441 (2012)
Exogenous hydrogen sulfide (H2S) leads to down-regulation of inflammatory responses and provides myocardial protection during acute ischemia/reperfusion injury; however its role during chronic heart failure (CHF) due to myocardial infarction (MI) is
Externí odkaz:
https://doaj.org/article/ea4b828f15fe4c9e82cad5f25719b4de
Akademický článek
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Autor:
F. Armaghan, Thomas P. Vacek, Natia Qipshidze, Srikanth Givvimani, Soumi Kundu, Suresh C. Tyagi, Nithya Narayanan, Sathnur Pushpakumar
Publikováno v:
Archives of Physiology and Biochemistry. 119:65-74
Pressure overload induces cardiac extracellular matrix (ECM) remodelling and results in heart failure. ECM remodelling by matrix metalloproteinases (MMPs) is primarily regulated by their target inhibitors, tissue inhibitor of matrix metalloproteinase
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::068fc82b12c96fb47fdea2af9e0c9edb
https://doi.org/10.3109/13813455.2012.755548
https://doi.org/10.3109/13813455.2012.755548
Autophagy mechanism of right ventricular remodeling in murine model of pulmonary artery constriction
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 302:H688-H696
Although right ventricular failure (RVF) is the hallmark of pulmonary arterial hypertension (PAH), the mechanism of RVF is unclear. Development of PAH-induced RVF is associated with an increased reactive oxygen species (ROS) production. Increases in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9264ba98f32d66262606a5865b97fea8
https://doi.org/10.1152/ajpheart.00777.2011
https://doi.org/10.1152/ajpheart.00777.2011
Publikováno v:
International Journal of Biological Sciences
Exogenous hydrogen sulfide (H2S) leads to down-regulation of inflammatory responses and provides myocardial protection during acute ischemia/reperfusion injury; however its role during chronic heart failure (CHF) due to myocardial infarction (MI) is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f31592a9ae5b711c177d51c9f12c7d14
https://doi.org/10.7150/ijbs.3632
https://doi.org/10.7150/ijbs.3632
Autor:
Charu Munjal, Srikanth Givvimani, Poulami Basu, Utpal Sen, Paras Kumar Mishra, Natia Qipshidze, Suresh C. Tyagi
Publikováno v:
Archives of Physiology and Biochemistry. 117:270-282
In the present study we tested the hypothesis whether hyperhomocysteinemia, an elevated homocysteine level, induces venous phenotype in artery. To test our hypothesis, we employed wild type (WT) and cystathionine β-synthase heterozygous (+/-) (CBS+/
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9802d285023ee584306ebad0fd50c3ad
https://doi.org/10.3109/13813455.2011.599844
https://doi.org/10.3109/13813455.2011.599844
Autor:
Sathnur Pushpakumar, Jonathan C. Vacek, Madhavi Kandel, Naria Metreveli, Suresh C. Tyagi, Charu Munjal, Neetu Tyagi, Natia Qipshidze
Publikováno v:
Journal of Orthopaedic Research. 29:1511-1516
Deficiencies in folate lead to increased serum concentrations of homocysteine (Hcy), which is known as hyperhomocysteinemia (HHcy), is associated with bone disorders. Although, homocysteine (Hcy) accumulates collagen in bone and contribute to decreas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::68165a0f8e647df2b4e289c334b3304d
https://doi.org/10.1002/jor.21415
https://doi.org/10.1002/jor.21415
Autor:
Jonathan C. Vacek, Srikanth Givvimani, Suresh C. Tyagi, Soumi Kundu, Natia Qipshidze, Neetu Tyagi, Shray Kapoor
Publikováno v:
Neurochemistry International. 56:301-307
An elevated level of homocysteine (Hcy), known as hyperhomocysteinemia (HHcy), was associated with neurovascular diseases. At physiological levels, hydrogen sulfide (H(2)S) protected the neurovascular system. Because Hcy was also a precursor of hydro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a5948b8fb4cdf57d22ca739c91781f23
https://doi.org/10.1016/j.neuint.2009.11.002
https://doi.org/10.1016/j.neuint.2009.11.002