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pro vyhledávání: '"Nathaniel M Byers"'
Autor:
Nathaniel M Byers, Paul L Burns, Olga Stuchlik, Matthew S Reed, Jeremy P Ledermann, Jan Pohl, Ann M Powers
Publikováno v:
PLoS Neglected Tropical Diseases, Vol 17, Iss 1, p e0011028 (2023)
Chikungunya virus (CHIKV) and the closely related onyong-nyong virus (ONNV) are arthritogenic arboviruses that have caused significant, often debilitating, disease in millions of people. However, despite their kinship, they are vectored by different
Externí odkaz:
https://doaj.org/article/2bc7b1fe58474bf2976fd23e296bc986
Publikováno v:
Viruses, Vol 11, Iss 3, p 225 (2019)
The global burden of arboviral diseases and the limited success in controlling them calls for innovative methods to understand arbovirus infections. Metabolomics has been applied to detect alterations in host physiology during infection. This approac
Externí odkaz:
https://doaj.org/article/34d7816e4d584767a9381969092b9630
Publikováno v:
Viruses
Viruses, Vol 11, Iss 3, p 225 (2019)
Viruses, Vol 11, Iss 3, p 225 (2019)
The global burden of arboviral diseases and the limited success in controlling them calls for innovative methods to understand arbovirus infections. Metabolomics has been applied to detect alterations in host physiology during infection. This approac
Publikováno v:
Journal of Virology. 90:533-544
Baculovirus-encoded inhibitor of apoptosis (IAP) proteins likely evolved from their host cell IAP homologs, which function as critical regulators of cell death. Despite their striking relatedness to cellular IAPs, including the conservation of two ba
Publikováno v:
Journal of Virology. 89:4481-4493
Inhibitor-of-apoptosis (IAP) proteins are key regulators of the innate antiviral response by virtue of their capacity to respond to signals affecting cell survival. In insects, wherein the host IAP provides a primary restriction to apoptosis, diverse
Publikováno v:
Journal of virology. 87(23)
The DNA damage response (DDR) of a host organism represents an effective antiviral defense that is frequently manipulated and exploited by viruses to promote multiplication. We report here that the large DNA baculoviruses, which require host DDR acti