Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Nathaly Espitia Pinzon"'
Autor:
Nathaly Espitia Pinzon, Hanneke van Mierlo, Jenny C. de Jonge, John J. P. Brevé, John G. J. M. Bol, Benjamin Drukarch, Anne-Marie van Dam, Wia Baron
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Demyelinated lesions of the central nervous system are characteristic for multiple sclerosis (MS). Remyelination is not very effective, particular at later stages of the disease, which results in a chronic neurodegenerative character with worsening o
Externí odkaz:
https://doaj.org/article/a38802668228440e9bce933d457eae6c
Autor:
Nathaly Espitia Pinzon, Esther Stroo, Bert A 't Hart, John G J M Bol, Benjamin Drukarch, Jan Bauer, Anne-Marie van Dam
Publikováno v:
PLoS ONE, Vol 9, Iss 6, p e100574 (2014)
Infiltration of leukocytes is a major pathological event in white matter lesion formation in the brain of multiple sclerosis (MS) patients. In grey matter lesions, less infiltration of these cells occur, but microglial activation is present. Thus far
Externí odkaz:
https://doaj.org/article/f4884f653e4e41fbaaae4fdc706b01e8
Autor:
Benjamin Drukarch, Hanneke van Mierlo, John J. P. Brevé, John G.J.M. Bol, Wia Baron, Jenny C. de Jonge, Anne-Marie van Dam, Nathaly Espitia Pinzon
Publikováno v:
Espitia Pinzon, N, van Mierlo, H, de Jonge, J C, Brevé, J J P, Bol, J G J M, Drukarch, B, van Dam, A-M & Baron, W 2019, ' Tissue transglutaminase promotes early differentiation of oligodendrocyte progenitor cells ', Frontiers in Cellular Neuroscience, vol. 13, 281 . https://doi.org/10.3389/fncel.2019.00281
Frontiers in cellular neuroscience, 13:281. Frontiers Media SA
Frontiers in Cellular Neuroscience, 13:281. Frontiers Media S.A.
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Frontiers in cellular neuroscience, 13:281. Frontiers Media SA
Frontiers in Cellular Neuroscience, 13:281. Frontiers Media S.A.
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Demyelinated lesions of the central nervous system are characteristic for multiple sclerosis (MS). Remyelination is not very effective, particular at later stages of the disease, which results in a chronic neurodegenerative character with worsening o
Autor:
Benjamin Drukarch, Anne-Marie van Dam, John G.J.M. Bol, Nathaly Espitia Pinzon, John J. P. Brevé, Wia Baron
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Journal of Neuroinflammation, 14(1). BioMed Central
Journal of neuroinflammation, 14:260. BMC
Espitia Pinzón, N, Brevé, J J P, Bol, J G J M, Drukarch, B, Baron, W & van Dam, A-M 2017, ' Tissue transglutaminase in astrocytes is enhanced by inflammatory mediators and is involved in the formation of fibronectin fibril-like structures ', Journal of Neuroinflammation, vol. 14, no. 1, pp. 260 . https://doi.org/10.1186/s12974-017-1031-2
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Journal of Neuroinflammation, 14(1). BioMed Central
Journal of neuroinflammation, 14:260. BMC
Espitia Pinzón, N, Brevé, J J P, Bol, J G J M, Drukarch, B, Baron, W & van Dam, A-M 2017, ' Tissue transglutaminase in astrocytes is enhanced by inflammatory mediators and is involved in the formation of fibronectin fibril-like structures ', Journal of Neuroinflammation, vol. 14, no. 1, pp. 260 . https://doi.org/10.1186/s12974-017-1031-2
Background During multiple sclerosis (MS) lesion formation, inflammatory mediators are produced by microglial cells and invading leukocytes. Subsequently, hypertrophic astrocytes fill the lesion and produce extracellular matrix (ECM) proteins that to
Autor:
John G.J.M. Bol, Anne Marie Van Dam, Wia Baron, Benjamin Drukarch, Berta Sanz-Morello, John J. P. Brevé, Jan Bauer, Nathaly Espitia Pinzon
Publikováno v:
Scientific Reports
Scientific Reports, 7:40995. Nature Publishing Group
Espitia Pinzon, N, Sanz-Morello, B, Brevé, J J P, Bol, J G J M, Drukarch, B, Bauer, J, Baron, W & Van Dam, A M 2017, ' Astrocyte-derived tissue Transglutaminase affects fibronectin deposition, but not aggregation, during cuprizone-induced demyelination ', Scientific Reports, vol. 7, 40995 . https://doi.org/10.1038/srep40995
Scientific Reports, 7:40995. Nature Publishing Group
Espitia Pinzon, N, Sanz-Morello, B, Brevé, J J P, Bol, J G J M, Drukarch, B, Bauer, J, Baron, W & Van Dam, A M 2017, ' Astrocyte-derived tissue Transglutaminase affects fibronectin deposition, but not aggregation, during cuprizone-induced demyelination ', Scientific Reports, vol. 7, 40995 . https://doi.org/10.1038/srep40995
Astrogliosis as seen in Multiple Sclerosis (MS) develops into astroglial scarring, which is beneficial because it seals off the site of central nervous system (CNS) damage. However, astroglial scarring also forms an obstacle that inhibits axon outgro
Autor:
Anne-Marie van Dam, Jan Bauer, Bert A. 't Hart, Esther Stroo, John G.J.M. Bol, Nathaly Espitia Pinzon, Benjamin Drukarch
Publikováno v:
PLoS One (print), 9(6). Public Library of Science
Espitia Pinzon, N, Stroo, E, 't Hart, B A, Bol, J G J M, Drukarch, B, Bauer, J & van Dam, A-M 2014, ' Tissue transglutaminase in marmoset experimental multiple sclerosis : discrepancy between white and grey matter ', PLoS ONE, vol. 9, no. 6, pp. e100574 . https://doi.org/10.1371/journal.pone.0100574
PLoS ONE, 9(6). Public Library of Science
PLoS ONE
PLoS ONE, 9(6):e100574. PUBLIC LIBRARY SCIENCE
PLoS ONE, Vol 9, Iss 6, p e100574 (2014)
Espitia Pinzon, N, Stroo, E, 't Hart, B A, Bol, J G J M, Drukarch, B, Bauer, J & van Dam, A-M 2014, ' Tissue transglutaminase in marmoset experimental multiple sclerosis : discrepancy between white and grey matter ', PLoS ONE, vol. 9, no. 6, pp. e100574 . https://doi.org/10.1371/journal.pone.0100574
PLoS ONE, 9(6). Public Library of Science
PLoS ONE
PLoS ONE, 9(6):e100574. PUBLIC LIBRARY SCIENCE
PLoS ONE, Vol 9, Iss 6, p e100574 (2014)
Infiltration of leukocytes is a major pathological event in white matter lesion formation in the brain of multiple sclerosis (MS) patients. In grey matter lesions, less infiltration of these cells occur, but microglial activation is present. Thus far
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1a8d3cf641cd3aab465fd9f74b0e313b
https://pure.eur.nl/en/publications/edfc7f5e-d77d-4395-b3df-a72c6666ff53
https://pure.eur.nl/en/publications/edfc7f5e-d77d-4395-b3df-a72c6666ff53
Autor:
Nathaly Espitia Pinzón, John J. P. Brevé, John G. J. M. Bol, Benjamin Drukarch, Wia Baron, Anne-Marie van Dam
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Abstract Background During multiple sclerosis (MS) lesion formation, inflammatory mediators are produced by microglial cells and invading leukocytes. Subsequently, hypertrophic astrocytes fill the lesion and produce extracellular matrix (ECM) protein
Externí odkaz:
https://doaj.org/article/5439e290a360424b93f60c75943997ea