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Autor:
Nathaly Espitia Pinzón, John J. P. Brevé, John G. J. M. Bol, Benjamin Drukarch, Wia Baron, Anne-Marie van Dam
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Abstract Background During multiple sclerosis (MS) lesion formation, inflammatory mediators are produced by microglial cells and invading leukocytes. Subsequently, hypertrophic astrocytes fill the lesion and produce extracellular matrix (ECM) protein
Externí odkaz:
https://doaj.org/article/5439e290a360424b93f60c75943997ea
