Zobrazeno 1 - 10
of 55
pro vyhledávání: '"Nathalie Auphan"'
Autor:
Franca Cassol, Loriane Portal, Sylvie Richelme, Mathieu Dupont, Yannick Boursier, Maria Arechederra, Nathalie Auphan-Anezin, Lionel Chasson, Caroline Laprie, Samantha Fernandez, Laure Balasse, Fabienne Lamballe, Rosanna Dono, Benjamin Guillet, Toby Lawrence, Christian Morel, Flavio Maina
Publikováno v:
iScience, Vol 21, Iss , Pp 68-83 (2019)
Summary: Computed tomography is a powerful medical imaging modality for longitudinal studies in cancer to follow neoplasia progression and evaluate anticancer therapies. Here, we report the generation of a photon-counting micro-computed tomography (P
Externí odkaz:
https://doaj.org/article/4ac8f497746640ab8599f6ebf470943e
Autor:
Jennifer Bintz, Analía Meilerman Abuelafia, François Gerbe, Elodie Baudoin, Nathalie Auphan-Anezin, Emmanuelle Sidot, Philippe Jay, Juan Iovanna
Publikováno v:
Biology, Vol 9, Iss 10, p 341 (2020)
TUFT cells have been described as strong modulators of inflammatory cells in several tissues including pancreas. TUFT cells, also known as DCLK1+ cells, are dependent of the transcriptional factor POU2F3. Several works report DCLK1+ cells in early st
Externí odkaz:
https://doaj.org/article/409ce77e683f4d0da865b758b33ff221
Autor:
Saïdi M Soudja, Sandrine Henri, Marielle Mello, Lionel Chasson, Amandine Mas, Maria Wehbe, Nathalie Auphan-Anezin, Lee Leserman, Benoît Van den Eynde, Anne-Marie Schmitt-Verhulst
Publikováno v:
PLoS ONE, Vol 6, Iss 7, p e22639 (2011)
Migration of dendritic cells (DC) from the tumor environment to the T cell cortex in tumor-draining lymph nodes (TDLN) is essential for priming naïve T lymphocytes (TL) to tumor antigen (Ag). We used a mouse model of induced melanoma in which simila
Externí odkaz:
https://doaj.org/article/1f7425e84e524386a019f0d136cc58e1
Autor:
Christophe Verthuy, Toby Lawrence, Bernard Malissen, Karine Crozat, Ammar Sabir Cheema, Pierre Milpied, Ghita Ghislat, Thien Phong Vu Manh, Marc Dalod, Noudjoud Attaf, Nathalie Auphan-Anezin, Chuang Dong, Pedro J. Ballester, Elodie Baudoin
Publikováno v:
Science Immunology
Science Immunology, 2021, 6 (61), pp.eabg3570. ⟨10.1126/sciimmunol.abg3570⟩
Science Immunology, American Association for the Advancement of Science, 2021, 6 (61), pp.eabg3570. ⟨10.1126/sciimmunol.abg3570⟩
Science Immunology, American Association for the Advancement of Science, 2021, 6 (61), ⟨10.1126/sciimmunol.abg3570⟩
Science Immunology, 2021, 6 (61), pp.eabg3570. ⟨10.1126/sciimmunol.abg3570⟩
Science Immunology, American Association for the Advancement of Science, 2021, 6 (61), pp.eabg3570. ⟨10.1126/sciimmunol.abg3570⟩
Science Immunology, American Association for the Advancement of Science, 2021, 6 (61), ⟨10.1126/sciimmunol.abg3570⟩
International audience; Conventional type 1 dendritic cells (cDC1s) are critical for antitumor immunity. They acquire antigens from dying tumor cells and cross-present them to CD8 + T cells, promoting the expansion of tumor-specific cytotoxic T cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63e86aba87d83672cfdd6f9a20b76280
https://hal-amu.archives-ouvertes.fr/hal-03329415/document
https://hal-amu.archives-ouvertes.fr/hal-03329415/document
Autor:
Christophe Verthuy, Pedro J. Ballester, Bernard Malissen, Karine Crozat, Elodie Baudoin, Toby Lawrence, Pierre Milpied, Noudjoud Attaf, Thien-Phong Vu Manh, Nathalie Auphan-Anezin, Marc Dalod, Chuang Dong, Ammar Sabir Cheema, Ghita Ghislat
Conventional type 1 dendritic cells (cDC1s) are critical for anti-tumor immunity. They acquire antigens from dying tumor cells and cross-present them to CD8+ T cells, promoting the expansion of tumor-specific cytotoxic T cells. However, the signaling
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::376dd9511c61eb6060b67f75a5f088b6
https://doi.org/10.1101/2020.12.28.424621
https://doi.org/10.1101/2020.12.28.424621
Autor:
Nathalie Auphan-Anezin, Marcello Delfini, Morgane Moulin, Toby Lawrence, Noushine Mossadegh-Keller, Anders Etzerodt, Søren K. Moestrup, Thomas Koed Doktor, Marc Bajénoff, Michael H. Sieweke
Publikováno v:
Etzerodt, A, Moulin, M, Doktor, T K, Delfini, M, Mossadegh-Keller, N, Bajenoff, M, Sieweke, M H, Moestrup, S K, Auphan-Anezin, N & Lawrence, T 2020, ' Tissue-resident macrophages in omentum promote metastatic spread of ovarian cancer ', The Journal of Experimental Medicine, vol. 217, no. 4, e20191869 . https://doi.org/10.1084/jem.20191869
Etzerodt, A, Moulin, M, Doktor, T K, Delfini, M, Mossadegh-Keller, N, Bajenoff, M, Sieweke, M H, Moestrup, S K, Auphan-Anezin, N & Lawrence, T 2020, ' Tissue-resident macrophages in omentum promote metastatic spread of ovarian cancer ', The Journal of experimental medicine, vol. 217, no. 4, e20191869 . https://doi.org/10.1084/jem.20191869
Journal of Experimental Medicine
Journal of Experimental Medicine, 2020, 217 (4), ⟨10.1084/jem.20191869⟩
Journal of Experimental Medicine, Rockefeller University Press, 2020, 217 (4), ⟨10.1084/jem.20191869⟩
The Journal of Experimental Medicine
Etzerodt, A, Moulin, M, Doktor, T K, Delfini, M, Mossadegh-Keller, N, Bajenoff, M, Sieweke, M H, Moestrup, S K, Auphan-Anezin, N & Lawrence, T 2020, ' Tissue-resident macrophages in omentum promote metastatic spread of ovarian cancer ', The Journal of experimental medicine, vol. 217, no. 4, e20191869 . https://doi.org/10.1084/jem.20191869
Journal of Experimental Medicine
Journal of Experimental Medicine, 2020, 217 (4), ⟨10.1084/jem.20191869⟩
Journal of Experimental Medicine, Rockefeller University Press, 2020, 217 (4), ⟨10.1084/jem.20191869⟩
The Journal of Experimental Medicine
Etzerodt et al. identify a unique subset of CD163+ Tim4+ tissue-resident macrophages in omentum that are maintained independently of bone marrow–derived monocytes and have a specific role in the metastatic spread of ovarian cancer cells.
Exper
Exper
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ae2879b4b746bf5f6e178a9fce14d38b
https://findresearcher.sdu.dk:8443/ws/files/170050190/jem_20191869.pdf
https://findresearcher.sdu.dk:8443/ws/files/170050190/jem_20191869.pdf
Autor:
Fabienne Lamballe, Christian Morel, L Portal, M. Dupont, Nathalie Auphan-Anezin, F. Cassol, Toby Lawrence, Samantha Fernandez, Yannick Boursier, Rosanna Dono, Caroline Laprie, Lionel Chasson, Laure Balasse, Sylvie Richelme, Flavio Maina, María Arechederra, Benjamin Guillet
Publikováno v:
iScience
iScience, Elsevier, 2019, 21, pp.68-83. ⟨10.1016/j.isci.2019.10.015⟩
iScience, Vol 21, Iss, Pp 68-83 (2019)
iScience, 2019, 21, pp.68-83. ⟨10.1016/j.isci.2019.10.015⟩
iScience, Elsevier, 2019, 21, pp.68-83. ⟨10.1016/j.isci.2019.10.015⟩
iScience, Vol 21, Iss, Pp 68-83 (2019)
iScience, 2019, 21, pp.68-83. ⟨10.1016/j.isci.2019.10.015⟩
Summary Computed tomography is a powerful medical imaging modality for longitudinal studies in cancer to follow neoplasia progression and evaluate anticancer therapies. Here, we report the generation of a photon-counting micro-computed tomography (PC
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6be3b76b5f4145e043d19e208c72784a
https://hal.archives-ouvertes.fr/hal-02368834/document
https://hal.archives-ouvertes.fr/hal-02368834/document
Autor:
Nathalie Auphan-Anezin, Jan Valečka, Evelina Gatti, Philippe Pierre, Marisa Reverendo, Voahirana Camosseto, Christine Polte, Rafael J. Argüello, Zoya Ignatova
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, ⟨10.1073/pnas.1904396116⟩
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, 116 (45), pp.22721-22729. ⟨10.1073/pnas.1904396116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, 116 (45), pp.22721-22729. ⟨10.1073/pnas.1904396116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, ⟨10.1073/pnas.1904396116⟩
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, ⟨10.1073/pnas.1904396116⟩
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Proceedings of the National Academy of Sciences of the United States of America, National Academy of Sciences, 2019, 116 (45), pp.22721-22729. ⟨10.1073/pnas.1904396116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, 116 (45), pp.22721-22729. ⟨10.1073/pnas.1904396116⟩
Proceedings of the National Academy of Sciences of the United States of America, 2019, ⟨10.1073/pnas.1904396116⟩
International audience; Exposure to microbe-associated molecular patterns (MAMPs) causes dendritic cells (DCs) to undergo a remarkable activation process characterized by changes in key biochemical mechanisms. These enhance antigen processing and pre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8be354bedd4ef791b99a25abc9c0ace7
https://hal.archives-ouvertes.fr/hal-02363598
https://hal.archives-ouvertes.fr/hal-02363598
Autor:
Nathalie Auphan-Anezin, Marion Massé, Thomas Ulas, Juan Rodriguez-Vita, Pieter Goossens, Christophe Verthuy, Anders Etzerodt, Marc Dalod, Magali Bebien, Olivia Rastoin, Martin R Turner, Joachim L. Schultze, Olympia Papantonopoulou, Miranda Van Eck, Toby Lawrence, Thien Phong Vu Manh, Victoire Gouirand
Publikováno v:
Cell Metabolism
Cell Metabolism, Elsevier, 2019, 29 (6), pp.1376-1389. ⟨10.1016/j.cmet.2019.02.016⟩
Goossens, P, Rodriguez-Vita, J, Etzerodt, A, Masse, M, Rastoin, O, Gouirand, V, Ulas, T, Papantonopoulou, O, Van Eck, M, Auphan-Anezin, N, Bebien, M, Verthuy, C, Vu Manh, T P, Turner, M, Dalod, M, Schultze, J L & Lawrence, T 2019, ' Membrane Cholesterol Efflux Drives Tumor-Associated Macrophage Reprogramming and Tumor Progression ', CELL METABOLISM, vol. 29, no. 6, pp. 1376-1389.e4 . https://doi.org/10.1016/j.cmet.2019.02.016
Cell Metabolism, Elsevier, 2019, 29 (6), pp.1376-1389.e4. ⟨10.1016/j.cmet.2019.02.016⟩
Goossens, P, Rodriguez-Vita, J, Etzerodt, A, Masse, M, Rastoin, O, Gouirand, V, Ulas, T, Papantonopoulou, O, Van Eck, M, Auphan-Anezin, N, Bebien, M, Verthuy, C, Vu Manh, T P, Turner, M, Dalod, M, Schultze, J L & Lawrence, T 2019, ' Membrane Cholesterol Efflux Drives Tumor-Associated Macrophage Reprogramming and Tumor Progression ', Cell Metabolism, vol. 29, no. 6, pp. 1376-1389.e4 . https://doi.org/10.1016/j.cmet.2019.02.016
Cell Metabolism, 2019, 29 (6), pp.1376-1389. ⟨10.1016/j.cmet.2019.02.016⟩
Cell Metabolism, 29(6), 1376-1389.e4. Cell Press
Cell metabolism 29(6), 1376-1389.e4 (2019). doi:10.1016/j.cmet.2019.02.016
Cell Metabolism, Elsevier, 2019, 29 (6), pp.1376-1389. ⟨10.1016/j.cmet.2019.02.016⟩
Goossens, P, Rodriguez-Vita, J, Etzerodt, A, Masse, M, Rastoin, O, Gouirand, V, Ulas, T, Papantonopoulou, O, Van Eck, M, Auphan-Anezin, N, Bebien, M, Verthuy, C, Vu Manh, T P, Turner, M, Dalod, M, Schultze, J L & Lawrence, T 2019, ' Membrane Cholesterol Efflux Drives Tumor-Associated Macrophage Reprogramming and Tumor Progression ', CELL METABOLISM, vol. 29, no. 6, pp. 1376-1389.e4 . https://doi.org/10.1016/j.cmet.2019.02.016
Cell Metabolism, Elsevier, 2019, 29 (6), pp.1376-1389.e4. ⟨10.1016/j.cmet.2019.02.016⟩
Goossens, P, Rodriguez-Vita, J, Etzerodt, A, Masse, M, Rastoin, O, Gouirand, V, Ulas, T, Papantonopoulou, O, Van Eck, M, Auphan-Anezin, N, Bebien, M, Verthuy, C, Vu Manh, T P, Turner, M, Dalod, M, Schultze, J L & Lawrence, T 2019, ' Membrane Cholesterol Efflux Drives Tumor-Associated Macrophage Reprogramming and Tumor Progression ', Cell Metabolism, vol. 29, no. 6, pp. 1376-1389.e4 . https://doi.org/10.1016/j.cmet.2019.02.016
Cell Metabolism, 2019, 29 (6), pp.1376-1389. ⟨10.1016/j.cmet.2019.02.016⟩
Cell Metabolism, 29(6), 1376-1389.e4. Cell Press
Cell metabolism 29(6), 1376-1389.e4 (2019). doi:10.1016/j.cmet.2019.02.016
Macrophages possess intrinsic tumoricidal activity, yet tumor-associated macrophages (TAMs) rapidly adopt an alternative phenotype within the tumor microenvironment that is marked by tumor-promoting immunosuppressive and trophic functions. The mechan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d90bee64f16c403c31b709e0d5a1a96b
https://hal.archives-ouvertes.fr/hal-02359441/document
https://hal.archives-ouvertes.fr/hal-02359441/document