Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Natasha J. Anstey"'
Autor:
Natasha J. Anstey, Vijayakumar Kapgal, Shashank Tiwari, Thomas C. Watson, Anna K. H. Toft, Owen R. Dando, Felicity H. Inkpen, Paul S. Baxter, Zrinko Kozić, Adam D. Jackson, Xin He, Mohammad Sarfaraz Nawaz, Aiman Kayenaat, Aditi Bhattacharya, David J. A. Wyllie, Sumantra Chattarji, Emma R. Wood, Oliver Hardt, Peter C. Kind
Publikováno v:
Molecular Autism, Vol 13, Iss 1, Pp 1-19 (2022)
Abstract Background Mutations in the postsynaptic transmembrane protein neuroligin-3 are highly correlative with autism spectrum disorders (ASDs) and intellectual disabilities (IDs). Fear learning is well studied in models of these disorders, however
Externí odkaz:
https://doaj.org/article/3d9e90cbc8e047a6a47ce223c07a8fd8
Autor:
Sam A. Booker, Laura Simões de Oliveira, Natasha J. Anstey, Zrinko Kozic, Owen R. Dando, Adam D. Jackson, Paul S. Baxter, Lori L. Isom, Diane L. Sherman, Giles E. Hardingham, Peter J. Brophy, David J.A. Wyllie, Peter C. Kind
Publikováno v:
Cell Reports, Vol 32, Iss 6, Pp 107988- (2020)
Summary: Cellular hyperexcitability is a salient feature of fragile X syndrome animal models. The cellular basis of hyperexcitability and how it responds to changing activity states is not fully understood. Here, we show increased axon initial segmen
Externí odkaz:
https://doaj.org/article/43e9885c0bb64e499d98b5c2d2ae7c83
Autor:
Natasha J. Anstey, Vijayakumar Kapgal, Shashank Tiwari, Thomas C. Watson, Anna K. H. Toft, Owen R. Dando, Felicity H. Inkpen, Paul S. Baxter, Zrinko Kozić, Adam D. Jackson, Xin He, Mohammad Sarfaraz Nawaz, Aiman Kayenaat, Aditi Bhattacharya, David J. A. Wyllie, Sumantra Chattarji, Emma R. Wood, Oliver Hardt, Peter C. Kind
Publikováno v:
Anstey, N J, Kapgal, V, Tiwari, S, Watson, T C, Toft, A K H, Dando, O R, Inkpen, F H, Baxter, P S, Kozic, Z, Jackson, A D, He, X, Nawaz, M S, Kayenaat, A, Bhattacharya, A, Wyllie, D J A, Chattarji, S, Wood, E R, Hardt, O & Kind, P C 2022, ' Imbalance of flight-freeze responses and their cellular correlates in the Nlgn3-/y rat model of autism ', Molecular Autism, vol. 13, 34 . https://doi.org/10.1186/s13229-022-00511-8
BackgroundMutations in the postsynaptic transmembrane protein neuroligin-3 are highly correlative with autism spectrum disorders (ASDs) and intellectual disabilities (IDs). Fear learning is well studied in models of these disorders, however differenc
Autor:
Natasha J, Anstey, Vijayakumar, Kapgal, Shashank, Tiwari, Thomas C, Watson, Anna K H, Toft, Owen R, Dando, Felicity H, Inkpen, Paul S, Baxter, Zrinko, Kozić, Adam D, Jackson, Xin, He, Mohammad Sarfaraz, Nawaz, Aiman, Kayenaat, Aditi, Bhattacharya, David J A, Wyllie, Sumantra, Chattarji, Emma R, Wood, Oliver, Hardt, Peter C, Kind
Publikováno v:
Molecular autism. 13(1)
Mutations in the postsynaptic transmembrane protein neuroligin-3 are highly correlative with autism spectrum disorders (ASDs) and intellectual disabilities (IDs). Fear learning is well studied in models of these disorders, however differences in fear
Autor:
Oliver Hardt, Alfredo Gonzalez-Sulser, I. Buller-Peralta, Jacqueline Smith, S. Biswal, Michael A. Cousin, Nicholas Perentos, Daisy Arkell, V. Kapgal, Peter C. Kind, M. S. Nawaz, Danai Katsanevaki, Emma R. Wood, Sally M. Till, Natasha J. Anstey, Thomas C Watson, S. Tiwari, L. Mizen, Matt Jones, Sumantra Chattarji
Pathogenic variants in SYNGAP1 are one of the most common genetic causes of nonsyndromic intellectual disability (ID) and are considered a risk for autism spectrum disorder (ASD). SYNGAP1 encodes a synaptic GTPase activating protein that modulates th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2b55f8a496b2614deb02d23f736d9fcf
https://doi.org/10.1101/2020.10.14.339192
https://doi.org/10.1101/2020.10.14.339192
Autor:
David J. A. Wyllie, Sumantra Chattarji, Thomas C Watson, Zrinko Kozic, Natasha J. Anstey, Adam D. Jackson, Emma R. Wood, Xin He, Shashank Tiwari, Aiman Kayenaat, Peter C. Kind, Owen Dando, Aditi Bhattacharya, Oliver Hardt, Mohammad Sarfaraz Nawaz, Anna Kh Toft, Felicity H Inkpen, Vijayakumar Kapgal, Paul Baxter
SummaryMutations in the postsynaptic transmembrane protein neuroligin-3 are highly correlative with autism spectrum disorders (ASDs) and intellectual disabilities (IDs). Fear learning is well studied in models of these disorders, however differences
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c27a22c8c7a06ee869fde6d839c64ba3
https://doi.org/10.1101/2020.08.27.267880
https://doi.org/10.1101/2020.08.27.267880
Autor:
Giles E. Hardingham, Sam A. Booker, Peter J. Brophy, Lori L. Isom, Peter C. Kind, Natasha J. Anstey, Owen Dando, Paul Baxter, Zrinko Kozic, Diane L. Sherman, David J. A. Wyllie, Adam D. Jackson, Laura Simões de Oliveira
Publikováno v:
Cell Reports
Cell Reports, Vol 32, Iss 6, Pp 107988-(2020)
Booker, S, Simões De Oliveira, L, Anstey, N, Kozic, Z, Dando, O, Jackson, A, Baxter, P, Isom, L L, Sherman, D, Hardingham, G, Brophy, P, Wyllie, D & Kind, P 2020, ' Input-output relationship of CA1 pyramidal neurons reveals intact homeostatic mechanisms in a mouse model of Fragile X Syndrome ', Cell Reports . https://doi.org/10.1016/j.celrep.2020.107988
Cell Reports, Vol 32, Iss 6, Pp 107988-(2020)
Booker, S, Simões De Oliveira, L, Anstey, N, Kozic, Z, Dando, O, Jackson, A, Baxter, P, Isom, L L, Sherman, D, Hardingham, G, Brophy, P, Wyllie, D & Kind, P 2020, ' Input-output relationship of CA1 pyramidal neurons reveals intact homeostatic mechanisms in a mouse model of Fragile X Syndrome ', Cell Reports . https://doi.org/10.1016/j.celrep.2020.107988
Summary Cellular hyperexcitability is a salient feature of fragile X syndrome animal models. The cellular basis of hyperexcitability and how it responds to changing activity states is not fully understood. Here, we show increased axon initial segment