Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Natalie L Patterson"'
Autor:
Bianca C Bernardo, Geeta Sapra, Natalie L Patterson, Nelly Cemerlang, Helen Kiriazis, Tomomi Ueyama, Mark A Febbraio, Julie R McMullen
Publikováno v:
PLoS ONE, Vol 10, Iss 12, p e0145173 (2015)
Previous animal studies had shown that increasing heat shock protein 70 (Hsp70) using a transgenic, gene therapy or pharmacological approach provided cardiac protection in models of acute cardiac stress. Furthermore, clinical studies had reported ass
Externí odkaz:
https://doaj.org/article/f28c025ac4ac4f7b95bb31a103964121
Autor:
Jayne Manning, Sam Manna, Eileen M Dunne, Viktoria Bongcaron, Casey L Pell, Natalie L Patterson, Sacha D Kuil, Poshmaal Dhar, David Goldblatt, E Kim Mulholland, Paul V Licciardi, Roy M Robins-Browne, Richard Malley, Odilia Wijburg, Catherine Satzke
Pneumococcal Conjugate Vaccines (PCVs) have substantially reduced the burden of disease caused byStreptococcus pneumoniae(the pneumococcus). However, protection is limited to vaccine serotypes, and when administered to children who are colonized with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c7c50364ea7ca82c3dd5a34bdb35a886
https://doi.org/10.1101/2023.04.05.534641
https://doi.org/10.1101/2023.04.05.534641
Autor:
Natalie L. Patterson, Xiao-Jun Du, Xiao-Ming Gao, Sally S. Nguyen, Julie R. McMullen, Jenny Y. Y. Ooi, Yow Keat Tham, Helen Kiriazis, Bianca C. Bernardo, Yidan Su, Colleen J. Thomas, Ruby C.Y. Lin
Publikováno v:
Scientific Reports
Expression of miR-154 is upregulated in the diseased heart and was previously shown to be upregulated in the lungs of patients with pulmonary fibrosis. However, the role of miR-154 in a model of sustained pressure overload-induced cardiac hypertrophy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a36ec32c70cbffc0ac23c5b23fb25550
Autor:
Natalie L. Patterson, Logan Kanagaratnam, David Whalley, Ferdinand Tabas, N. Mughal, Wen Kai Wong, Alex L. Huang
Publikováno v:
Heart, Lung and Circulation. 27:812-818
Atrial fibrillation is common and management by pharmacotherapy is limited by modest efficacy and significant toxicities. Pulmonary vein isolation (PVI) is a safe and effective alternative in select patients with atrial fibrillation. However, prolong
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c6d827c5412f425a721a96ed7eadec54
https://doi.org/10.1016/j.hlc.2017.07.005
https://doi.org/10.1016/j.hlc.2017.07.005
Publikováno v:
Physiological Reviews. 98:419-475
The benefits of exercise on the heart are well recognized, and clinical studies have demonstrated that exercise is an intervention that can improve cardiac function in heart failure patients. This has led to significant research into understanding th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6fdc88b5f2300d30a04d8a7acb6405f6
https://doi.org/10.1152/physrev.00043.2016
https://doi.org/10.1152/physrev.00043.2016
Autor:
Junichi Sadoshima, Natalie L. Patterson, Jenny Y. Y. Ooi, Aya Matsumoto, Julie R. McMullen, Saloni Singla, Bianca C. Bernardo, Ruby C.Y. Lin, Yow Keat Tham, Susanna Obad, Helen Kiriazis
Publikováno v:
The Journal of Physiology. 594:5959-5974
Key points MicroRNA (miRNA)-based therapies are in development for numerous diseases, including heart disease. Currently, very limited basic information is available on the regulation of specific miRNAs in male and female hearts in settings of diseas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5cf6f3946322707be71e44cafd1fbedc
https://doi.org/10.1113/jp272512
https://doi.org/10.1113/jp272512
The IGF1-PI3K-Akt Signaling Pathway in Mediating Exercise-Induced Cardiac Hypertrophy and Protection
Publikováno v:
Advances in experimental medicine and biology. 1000
Regular physical activity or exercise training can lead to heart enlargement known as cardiac hypertrophy. Cardiac hypertrophy is broadly defined as an increase in heart mass. In adults, cardiac hypertrophy is often considered a poor prognostic sign
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::412e2f4aba082275a3ae2139c9151486
https://pubmed.ncbi.nlm.nih.gov/29098623
https://pubmed.ncbi.nlm.nih.gov/29098623
Autor:
Julie R. McMullen, Natalie A. Mellett, Jenny Y. Y. Ooi, Kevin Huynh, Peter J. Meikle, Yow Keat Tham, Natalie L. Patterson, Helen Kiriazis, Junichi Sadoshima, Aya Matsumoto, Darren C. Henstridge
Publikováno v:
Biochimica et biophysica acta. Molecular and cell biology of lipids. 1863(3)
Cardiac myocyte membranes contain lipids which remodel dramatically in response to heart growth and remodeling. Lipid species have both structural and functional roles. Physiological and pathological cardiac remodeling have very distinct phenotypes,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53a5cda0d445613b9833772df91a1a82
https://pubmed.ncbi.nlm.nih.gov/29217479
https://pubmed.ncbi.nlm.nih.gov/29217479
The IGF1-PI3K-Akt Signaling Pathway in Mediating Exercise-Induced Cardiac Hypertrophy and Protection
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9789811043031
Regular physical activity or exercise training can lead to heart enlargement known as cardiac hypertrophy. Cardiac hypertrophy is broadly defined as an increase in heart mass. In adults, cardiac hypertrophy is often considered a poor prognostic sign
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0196cb99553699ae7271b0c8909fcd25
https://doi.org/10.1007/978-981-10-4304-8_12
https://doi.org/10.1007/978-981-10-4304-8_12
Publikováno v:
Future medicinal chemistry. 8(18)
Heat shock proteins are a family of proteins that are produced by cells in response to exposure to stressful conditions. The best studied heat shock protein is HSP70, which is known to act as a molecular chaperone to maintain cellular homeostasis and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d58ca7f444aab533860a8c829055b62
https://pubmed.ncbi.nlm.nih.gov/27845575
https://pubmed.ncbi.nlm.nih.gov/27845575