Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Natalia Milduberger"'
Autor:
Alina E. Perrone, Mariana Pinillo, Marcela S. Rial, Marisa Fernández, Natalia Milduberger, Carolina González, Patricia L. Bustos, Laura E. Fichera, Susana A. Laucella, María Cecilia Albareda, Jacqueline Bua
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 15, p 11875 (2023)
Cyclophilins (CyPs) are a family of enzymes involved in protein folding. Trypanosoma cruzi, the causative agent of Chagas disease, has a 19-kDa cyclophilin, TcCyP19, that was found to be secreted in parasite stages of the CL Brener clone and recogniz
Externí odkaz:
https://doaj.org/article/327a1d97b16041009de3dceda3b3499b
Autor:
Patricia L. Bustos, Natalia Milduberger, Bibiana J. Volta, Alina E. Perrone, Susana A. Laucella, Jacqueline Bua
Publikováno v:
Frontiers in Microbiology, Vol 10 (2019)
Trypanosoma cruzi is the protozoan unicellular parasite that causes Chagas disease. It can be transmitted from infected mothers to their babies via the connatal route, thus being able to perpetuate even in the absence of Triatomine insect vectors. Ch
Externí odkaz:
https://doaj.org/article/39bc0f0ceb2d4bb0a25844c9779b8628
Publikováno v:
Biomolecules, Vol 8, Iss 4, p 132 (2018)
Trypanosoma cruzi is the etiological agent of Chagas disease. It affects eight million people worldwide and can be spread by several routes, such as vectorborne transmission in endemic areas and congenitally, and is also important in non-endemic regi
Externí odkaz:
https://doaj.org/article/910f012317e042cca0e08a6505a4dcf9
Autor:
Bibiana Julieta Volta, María Ailén Natale, Natalia Milduberger, Jacqueline Bua, Alina Elizabeth Perrone, Susana A. Laucella, Patricia Bustos, Carolina González
Publikováno v:
The Journal of Infectious Diseases. 224:1086-1095
Background Trypanosoma cruzi, the causative agent of Chagas disease, can be transmitted to the offspring of infected women, which constitutes an epidemiologically significant parasite transmission route in nonendemic areas. It is relevant to evaluate
Autor:
Jacqueline Bua, Natalia Milduberger, Miriam Postan, Patricia Bustos, Alina Elizabeth Perrone, Carolina González
Publikováno v:
Experimental parasitology. 220
Trypanosoma cruzi is the causative agent of Chagas disease, which is endemic in Latin America and around the world through mother to child transmission. The heart is the organ most frequently affected in the chronic stage of the human infection and d
Autor:
Alicia Graciela Fuchs, Alina Elizabeth Perrone, Patricia Laura Bustos, Natalia Milduberger, Jacqueline Bua
Publikováno v:
Biomolecules, Vol 8, Iss 4, p 132 (2018)
Biomolecules
Biomolecules
Trypanosoma cruzi is the etiological agent of Chagas disease. It affects eight million people worldwide and can be spread by several routes, such as vectorborne transmission in endemic areas and congenitally, and is also important in non-endemic regi
Autor:
Bibiana Julieta Volta, Patricia Laura Bustos, Alina Elizabeth Perrone, Natalia Milduberger, Jacqueline Bua
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Cell Death Discovery
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
Cell Death Discovery
Mitochondria have an important role in energy production, homeostasis and cell death. The opening of the mitochondrial permeability transition pore (mPTP) is considered one of the key events in apoptosis and necrosis, modulated by cyclophilin D (CyPD
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8424f6a13d26b87b2a355da1128c980b
https://www.nature.com/articles/cddiscovery201692
https://www.nature.com/articles/cddiscovery201692
Autor:
Miriam Postan, Natalia Milduberger, Alina Elizabeth Perrone, Patricia Laura Bustos, Jacqueline Búa
Cyclosporin A (CsA) specifically inhibits the mitochondrial permeability transition pore (mPTP). Opening of the mPTP, which is triggered by high levels of matrix [Ca2+] and/or oxidative stress, leads to mitochondrial dysfunction and thus to cell deat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f33b9413de54f95b13cdc40ce3d00572
https://www.cambridge.org/core/journals/parasitology/article/oxidative-stress-damage-in-the-protozoan-parasite-trypanosoma-cruzi-is-inhibited-by-cyclosporin-a/8658985A3CBDD72F637749304ACD2B55
https://www.cambridge.org/core/journals/parasitology/article/oxidative-stress-damage-in-the-protozoan-parasite-trypanosoma-cruzi-is-inhibited-by-cyclosporin-a/8658985A3CBDD72F637749304ACD2B55
Publikováno v:
Immunochemistry & Immunopathology.
Protein samples can be challenging to analyze due to the presence of high-abundance proteins masking lowabundance proteins of interest, such as biomarkers and novel physiological mediators. Cyclophilins are chaperones involved in the cis/trans isomer