Zobrazeno 1 - 10
of 48
pro vyhledávání: '"Nasim, Md. Talat"'
Yes
Aberrant transforming growth factor-β (TGF-β) signaling activation is linked to pulmonary arterial hypertension (PAH). BMPR2 mutations perturb the balance between bone morphogenetic protein (BMP) and TGF-β pathways, leading to vascular re
Aberrant transforming growth factor-β (TGF-β) signaling activation is linked to pulmonary arterial hypertension (PAH). BMPR2 mutations perturb the balance between bone morphogenetic protein (BMP) and TGF-β pathways, leading to vascular re
Externí odkaz:
http://hdl.handle.net/10454/18507
Autor:
Habas, Khaled S.A., Nganwuchu, Chinyere C., Shahzad, F., Gopalan, Rajendran C., Haque, M., Rahman, Sayeeda, Majumder, A.A., Nasim, Md. Talat
Yes
Introduction. Coronavirus disease 2019 (COVID-19) was first detected in China in December, 2019, and declared as a pandemic by the World Health Organization (WHO) on March 11, 2020. The current management of COVID-19 is based generally on su
Introduction. Coronavirus disease 2019 (COVID-19) was first detected in China in December, 2019, and declared as a pandemic by the World Health Organization (WHO) on March 11, 2020. The current management of COVID-19 is based generally on su
Externí odkaz:
http://hdl.handle.net/10454/17989
Autor:
Chowdhury, H.M., Sharmin, N., Yuzbasioglu Baran, M., Long, L., Morrell, N.W., Trembath, R.C., Nasim, Md. Talat
Yes
Pulmonary Arterial Hypertension (PAH) is a devastating cardiovascular disorder characterised by the remodelling of pre-capillary pulmonary arteries. The vascular remodelling observed in PAH patients results from excessive proliferation and a
Pulmonary Arterial Hypertension (PAH) is a devastating cardiovascular disorder characterised by the remodelling of pre-capillary pulmonary arteries. The vascular remodelling observed in PAH patients results from excessive proliferation and a
Externí odkaz:
http://hdl.handle.net/10454/16885
Autor:
Chowdhury, H.M., Siddiqui, M.A., Kanneganti, S., Sharmin, N., Chowdhury, M.W., Nasim, Md. Talat
Yes
Attempts have been made to treat nonsense-associated genetic disorders by chemical agents and hence an improved mechanistic insight into the decoding of readthrough signals is essential for the identification and characterisation of factors
Attempts have been made to treat nonsense-associated genetic disorders by chemical agents and hence an improved mechanistic insight into the decoding of readthrough signals is essential for the identification and characterisation of factors
Externí odkaz:
http://hdl.handle.net/10454/13989
Autor:
Hu, J., Khodadadi-Jamayran, A., Mao, M., Shah, K., Yang, Z., Nasim, Md. Talat, Wang, Z., Jiang, H.
Yes
Alternative splicing of pre-mRNAs significantly contributes to the complexity of gene expression in higher organisms, but the regulation of the splice site selection remains incompletely understood. We have previously demonstrated that a chr
Alternative splicing of pre-mRNAs significantly contributes to the complexity of gene expression in higher organisms, but the regulation of the splice site selection remains incompletely understood. We have previously demonstrated that a chr
Externí odkaz:
http://hdl.handle.net/10454/10168
Autor:
Lento, S., Brioschi, M., Barcella, S., Nasim, Md. Talat, Ghilardi, S., Barbieri, S.S., Tremoli, E., Banfi, C.
Yes
It has long been known that Tissue Factor (TF) plays a role in blood coagulation and has a direct thrombotic action that is closely related to cardiovascular risk, but it is becoming increasingly clear that it has a much wider range of biolo
It has long been known that Tissue Factor (TF) plays a role in blood coagulation and has a direct thrombotic action that is closely related to cardiovascular risk, but it is becoming increasingly clear that it has a much wider range of biolo
Externí odkaz:
http://hdl.handle.net/10454/8001
Yes
This data article is related to the research article entitled Proteomics of Tissue Factor silencing in cardiomyocytic cells reveals a new role for this coagulation factor in splicing machinery control by Lento et al [1]. Tissue Factor (TF) i
This data article is related to the research article entitled Proteomics of Tissue Factor silencing in cardiomyocytic cells reveals a new role for this coagulation factor in splicing machinery control by Lento et al [1]. Tissue Factor (TF) i
Externí odkaz:
http://hdl.handle.net/10454/7983
Autor:
Ogo, T., Chowdhury, H.M., Yang, J., Long, T., Li, X., Torres Cleuven, Y.N., Morrell, N.W., Schermuly, R.T., Trembath, R.C., Nasim, Md. Talat
Yes
Heterozygous loss of function mutations in the type II bone morphogenetic protein receptor (BMPR-II), a member of the transforming growth factor (TGF-β) receptor family, underlie the majority of familial cases of pulmonary arterial hyperten
Heterozygous loss of function mutations in the type II bone morphogenetic protein receptor (BMPR-II), a member of the transforming growth factor (TGF-β) receptor family, underlie the majority of familial cases of pulmonary arterial hyperten
Externí odkaz:
http://hdl.handle.net/10454/7969
Yes
Pulmonary arterial hypertension (PAH) is a rare cardiovascular disorder caused by narrowing of blood vessels in the lung and in the absence of therapy leads to right heart failure and death. No cure for this devastating disorder is known. Th
Pulmonary arterial hypertension (PAH) is a rare cardiovascular disorder caused by narrowing of blood vessels in the lung and in the absence of therapy leads to right heart failure and death. No cure for this devastating disorder is known. Th
Externí odkaz:
http://hdl.handle.net/10454/7971
Yes
Pulmonary arterial hypertension (PAH) is a cardiovascular disorder associated with enhanced proliferation and suppressed apoptosis of pulmonary arterial smooth muscle cells (PASMCs). Heterozygous mutations in the type II receptor for bone mo
Pulmonary arterial hypertension (PAH) is a cardiovascular disorder associated with enhanced proliferation and suppressed apoptosis of pulmonary arterial smooth muscle cells (PASMCs). Heterozygous mutations in the type II receptor for bone mo
Externí odkaz:
http://hdl.handle.net/10454/6115