Zobrazeno 1 - 10
of 60
pro vyhledávání: '"Naomichi, Nishimura"'
Publikováno v:
Journal of Nutritional Science and Vitaminology. 68:104-111
To investigate whether the oral intake of slowly digestible α-glucan (SDG) could have a trophic (i.e., thickening) effect on their ileal mucosae, for 10 d, rats were given control (non-SDG), 10% isomaltodextrin (IMD) or 10% resistant maltodextrin (R
Publikováno v:
British Journal of Nutrition. 128:1509-1517
The brain is rich in long-chain PUFA, which play an essential role in its development and functions. Here, we examined the impact of maternal n-3 PUFA intake deficiency during gestation and lactation on the development of glial cells in the pup’s d
Autor:
Naomichi, Nishimura *, Hiroki, Tanabe, Misato, Adachi, Tatsuro, Yamamoto, Michihiro, Fukushima
Publikováno v:
In The Journal of Nutrition December 2013 143(12):1943-1951
Publikováno v:
Journal of Nutritional Science and Vitaminology. 66:331-338
Pectin enhances mucin secretion in the rat small intestine. However, what structural features of pectin to stimulate mucin secretion remain unclear. The study aimed to clarify active constituents of pectin using a human goblet cell line, HT29-MTX. Va
Publikováno v:
The British journal of nutrition. 128(8)
The brain is rich in long-chain PUFA, which play an essential role in its development and functions. Here, we examined the impact of maternal
Publikováno v:
British Journal of Nutrition. 123(6):619-626
To investigate whether oral intake of highly branched α-glucan isomaltodextrin (IMD) could stimulate ileal glucagon-like peptide-1 (GLP-1) secretion, we examined (1) the digestibility of IMD, (2) the digestion and absorption rates of IMD, in rat sma
Publikováno v:
British Journal of Nutrition. 123:537-544
We investigated whether non-digestible saccharide fermentation-derived hydrogen molecules (H2) in rat colon could improve the in vivo reduction–oxidation (redox) balance via regeneration of α-tocopherol, by assessing their effect on hydroxyl radic
Autor:
Miki Umeki, Naomichi Nishimura, Yasuko Matsuyama, Hiroaki Oda, Satoshi Mochizuki, Saiko Ikeda, Shumin Sun, Fumiaki Hanzawa
Publikováno v:
Chronobiology International. 36:826-837
Excessive sucrose intake, known as fructose toxicity, leads to fatty liver, hyperlipidemia, and metabolic syndrome. Circadian disorders also contribute to metabolic syndrome. Here, we investigated the effect of excessive sucrose intake on circadian r
Autor:
Satoshi Mochizuki, Naomichi Nishimura, Shumin Sun, Saiko Ikeda, Fumiaki Hanzawa, Yuki Araki, Miki Umeki, Hiroaki Oda, Takaaki Kojima
Publikováno v:
The Journal of nutritional biochemistry. 93
Excess sucrose intake has been found to be a major factor in the development of metabolic syndrome, especially in promoting nonalcoholic fatty liver disease. The excess fructose is believed to targets the liver to promote de novo lipogenesis, as desc
Publikováno v:
International Journal of Human Culture Studies. 2019:147-154