Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Naomi Kunichika"'
Autor:
Akihiko Taniguchi, Katsuyuki Kiura, Masaomi Marukawa, Nobuaki Miyahara, Naomi Kunichika, Shoichi Kuyama, Yoshinobu Maeda, Goro Kimura
Publikováno v:
Respiratory Investigation. 57:566-573
Background The prevalence of Mycobacterium avium complex (MAC) pulmonary disease (PD) is increasing significantly in Japan. Among the patterns of MAC-PD, a solitary pulmonary nodule (SPN) is less common and often resembles lung cancer. The aim of thi
Publikováno v:
Nihon Naika Gakkai Zasshi. 107:1056-1069
Autor:
Shen Zhang, Ying Yu, Jason X.-J. Yuan, Carmelle V. Remillard, Naomi Kunichika, Oleksandr Platoshyn
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 287:L962-L969
Transient receptor potential (TRP) cation channels are a critical pathway for Ca2+entry during pulmonary artery (PA) smooth muscle contraction. However, whether canonical TRP (TRPC) subunits and which TRP channel isoforms are involved in store deplet
Autor:
Ori Ben-Yehuda, Hideki Kunichika, Stephane Lafitte, Barry Peters, Anthony N. DeMaria, Naomi Kunichika
Publikováno v:
Journal of the American College of Cardiology. 43(2):276-283
Objectives We assessed the effect of glycoprotein IIb/IIIa inhibition (GPI) on microvascular flow after coronary occlusion/reperfusion using quantitative myocardial contrast echocardiography (QMCE). Background Platelets may play a major role in the d
Autor:
Anthony N. DeMaria, Naomi Kunichika, Hisashi Masugata, Paul L. Wolf, Hideki Kunichika, Barry Peters, Bruno Cotter
Publikováno v:
Journal of the American College of Cardiology. 42:552-557
Objectives We examined whether delayed post-injection imaging of a new ultrasound contrast agent (BR-14) could produce prolonged opacification and hyperenhancement of myocardium subjected to coronary occlusion/reperfusion. Background We hypothesized
Publikováno v:
Journal of the American Society of Echocardiography. 15:1165-1169
Patients with chronic obstructive pulmonary disease (COPD) are difficult to assess by conventional transthoracic echocardiography (TTE) because of emphysematous lungs or mediastinal deviation. We hypothesized that superior vena cava (SVC) flow is rel
Autor:
Hiroyasu Takeyama, Mine Harada, Ryosuke Eda, Yoshiki Umemori, Kiyoshi Makihata, Tomoyuki Murakami, Keisuke Aoe, Naomi Kunichika, Nobuaki Miyahara, Kazuo Murakami
Publikováno v:
Internal Medicine. 40:786-790
A 27-year-old man with a primary pulmonary lymphoma of large B-cell type is described. Symptoms involved both the upper and lower respiratory tract. A chest roentgenogram showed a dense mass with cavitation. Transbronchial biopsy specimens revealed n
Autor:
Mine Harada, Tadashi Maeda, Hiroyasu Takeyama, Shigeki Makihara, Keisuke Aoe, Naomi Kunichika, Ryosuke Eda, Hiroyuki Kohara, Nobuaki Miyahara
Publikováno v:
Internal Medicine. 38:974-978
We report the transient spontaneous disappearance of a mucocele due to bronchial atresia. Two years before presentation, a chest radiograph showed a hyperlucent right upper lung and a mucocele near the right hilum. A chest radiograph taken 1 year lat
Autor:
Ying Yu, Judd W. Landsberg, Jason X.-J. Yuan, Patricia A. Thistlethwaite, Oleksandr Platoshyn, Ivana Fantozzi, Carmelle V. Remillard, Naomi Kunichika, Lewis J. Rubin, Donna D. Tigno
Pulmonary vascular medial hypertrophy caused by excessive pulmonary artery smooth muscle cell (PASMC) proliferation is a major cause for the elevated pulmonary vascular resistance in patients with idiopathic pulmonary arterial hypertension (IPAH). In
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc355aa50cbe4058068bac063d871474
https://europepmc.org/articles/PMC518765/
https://europepmc.org/articles/PMC518765/
Autor:
Hideki Kunichika, Ying Yu, Judd W. Landsberg, Patricia A. Thistlethwaite, Naomi Kunichika, Lewis J. Rubin, Jason X.-J. Yuan
Publikováno v:
American journal of respiratory and critical care medicine. 170(10)
Bosentan, a dual endothelin receptor blocker, has been used clinically to treat idiopathic pulmonary arterial hypertension (IPAH). However, the mechanism of its antiproliferative effect on pulmonary artery smooth muscle cells (PASMCs) remains unclear