Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Naomi J. Logsdon"'
Autor:
Sunad Rangarajan, Victor J. Thannickal, Naomi J. Logsdon, Samuel R. Smith, Ashish Kurundkar, Deepali Kurundkar, Jessy S. Deshane, Jaroslaw W. Zmijewski, Yan Y. Sanders, K.G. Dsouza, Mohammad Rehan, Karen Bernard, Diptiman Chanda
Publikováno v:
Nature aging
Aging is a risk factor for progressive fibrotic disorders involving diverse organ systems, including the lung. Idiopathic pulmonary fibrosis, an age-associated degenerative lung disorder, is characterized by persistence of apoptosis-resistant myofibr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f627c2f0e97d41fe6cd46da83953ef79
https://pubmed.ncbi.nlm.nih.gov/34386777
https://pubmed.ncbi.nlm.nih.gov/34386777
Autor:
Samuel B. Hutcheson, Tejaswini Kulkarni, Hai T. Vo, Dominik Kentrup, Joao A. de Andrade, Jarrod W. Barnes, Sadis Matalon, Christian Faul, Naomi J. Logsdon, Carol Farver, Deepali Kurundkar, Jaleesa M. Garth, Stefanie Krick, Morgan L. Locy, Scott Helton, Victor J. Thannickal, R. Denson, Zhihong Yu, Gwendalyn D. King, Dawn Duncan
Publikováno v:
Am J Physiol Lung Cell Mol Physiol
Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive fibrosing interstitial pneumonia that mainly affects the elderly. Several reports have demonstrated that aging is involved in the underlying pathogenic mechanisms of IPF. α-Klotho (KL) ha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a15210a1cfbafd22baa8df09e738d866
https://doi.org/10.1152/ajplung.00246.2018
https://doi.org/10.1152/ajplung.00246.2018
Autor:
Victor J. Thannickal, Mohammad Rehan, Ashish Kurundkar, Diptiman Chanda, Deepali Kurundkar, Jaroslaw W. Zmijewski, K.G. Dsouza, Karen Bernard, Naomi J. Logsdon
Publikováno v:
B20. THERAPEUTICS "2020" IN LUNG DISEASE.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ec8892d8b81fe6501fd6df68d5a3d2ba
https://doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2785
https://doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2785
Autor:
Kosuke Kato, Yoon Joo Shin, Victor J. Thannickal, Louise Hecker, Johnny M. Trinh, Mohamed Mohamed, Skye P. Rounseville, Kenneth S. Knox, Adam Knox, Sunny Palumbo, Joseph D. Irish, Kareem Ahmad, Sydney R. Rummel, Naomi J. Logsdon, Deepali Kurundkar
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology
Am J Respir Cell Mol Biol
Am J Respir Cell Mol Biol
Idiopathic pulmonary fibrosis (IPF) is a fatal age-associated disease with no cure. Although IPF is widely regarded as a disease of aging, the cellular mechanisms that contribute to this age-associated predilection remain elusive. In this study, we s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36eda1282936e6e8c03b89671996f9fe
https://pubmed.ncbi.nlm.nih.gov/31978311
https://pubmed.ncbi.nlm.nih.gov/31978311
Autor:
Meghan K Eberhardt, W L William Chang, Naomi J Logsdon, Yujuan Yue, Mark R Walter, Peter A Barry
Publikováno v:
PLoS ONE, Vol 7, Iss 5, p e37931 (2012)
BackgroundConsiderable evidence has accumulated that multiple viruses, bacteria, and protozoa manipulate interleukin-10 (IL-10)-mediated signaling through the IL-10 receptor (IL-10R) in ways that could enable establishment of a persistent microbial i
Externí odkaz:
https://doaj.org/article/410cfbf39d024c269da64e8987589c51
Publikováno v:
PLoS ONE, Vol 6, Iss 11, p e28127 (2011)
Human cytomegalovirus (HCMV) expresses a viral ortholog (CMVIL-10) of human cellular interleukin-10 (cIL-10). Despite only ∼26% amino acid sequence identity, CMVIL-10 exhibits comparable immunosuppressive activity with cIL-10, attenuates HCMV antiv
Externí odkaz:
https://doaj.org/article/8c17b5e8586c4d19afdd7d42380cce20
Publikováno v:
C62. FIBROBLAST BIOLOGY.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::453dbccd7fe428a2617eac1d06ef67d7
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5334
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5334
Publikováno v:
C62. FIBROBLAST BIOLOGY.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5c9edff99fd8891bd766620e243f12ea
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5343
https://doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5343
Autor:
Sunad Rangarajan, Naomi J. Logsdon, Benjamin P. Persons, Jaroslaw W. Zmijewski, Victor M. Darley-Usmar, Karen Bernard, Saranya Ravi, Gang Liu, Victor J. Thannickal, Kasturi Mitra, Na Xie
Publikováno v:
Journal of Biological Chemistry. 290:25427-25438
Contraction is crucial in maintaining the differentiated phenotype of myofibroblasts. Contraction is an energy-dependent mechanism that relies on the production of ATP by mitochondria and/or glycolysis. Although the role of mitochondrial biogenesis i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7285878f7cd3608a6ac51b8047d94972
https://doi.org/10.1074/jbc.m115.646984
https://doi.org/10.1074/jbc.m115.646984
Publikováno v:
BIOS. 85:218-223
Despite the increased incidence of fibrotic diseases with advancing age, little is known about the role of aging in these pathologies. Recent studies have proposed that senescence is an anti-fibrotic mechanism that promotes fibrosis resolution throug
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e6018b77f1ecdcbd1771f2da9370a2b2
https://doi.org/10.1893/0005-3155-85.4.218
https://doi.org/10.1893/0005-3155-85.4.218