Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Naomi C. Boisvert"'
Autor:
Eldjonai Kamto, Melica Novakovic, Naomi C. Boisvert, Khaled S. Abd-Elrahman, Jean-Francois Thibodeau, Stephen S. G. Ferguson, Chet E. Holterman, Christopher R.J. Kennedy
Publikováno v:
Antioxidants & Redox Signaling. 30:1817-1830
Oxidative stress associated with a proinflammatory state occurs in endothelial dysfunction, hypertension, chronic kidney disease, and diabetes. The NADPH oxidase (Nox) family of reactive oxygen species (ROS) generating enzymes is implicated in these
Autor:
Karolynn J Hsu, Anthony Carter, Khaled S. Abd-Elrahman, Christopher R.J. Kennedy, Stephen S. G. Ferguson, Jean-Francois Thibodeau, Naomi C. Boisvert, Gregory O. Cron, Ying He, Chet E. Holterman
Publikováno v:
Antioxidants & Redox Signaling. 25:642-656
Cyclooxygenase inhibition by non-steroidal anti-inflammatory drugs is contraindicated in hypertension, as it may reduce glomerular filtration rate (GFR) and renal blood flow. However, the identity of the specific eicosanoid and receptor underlying th
Autor:
Chet E. Holterman, Douglas A. Gray, Wanling Pan, R. Todd Alexander, Naomi C. Boisvert, Josée Coulombe, Chris R. J. Kennedy, Alexey Gutsol
Publikováno v:
American journal of physiology. Renal physiology. 315(2)
Loss of ubiquitin COOH-terminal hydrolase L1 (UCHL1), a deubiquitinating enzyme required for neuronal function, led to hyperphosphatemia accompanied by phosphaturia in mice, while calcium homeostasis remained intact. We therefore investigated the mec
Autor:
Gregory O. Cron, Luciano da Fontoura Costa, Alex Gutsol, Cesar H. Comin, Christopher R.J. Kennedy, Douglas A. Gray, Anthony Carter, Richard L. Hébert, Naomi C. Boisvert, Chet E. Holterman, Jean-Francois Thibodeau, Eldjonai Kamto, Baptiste Lacoste, Rania Nasrallah
Publikováno v:
Repositório Institucional da USP (Biblioteca Digital da Produção Intelectual)
Universidade de São Paulo (USP)
instacron:USP
Universidade de São Paulo (USP)
instacron:USP
Neuronal ubiquitin C-terminal hydrolase L1 (UCHL1) is a deubiquitinating enzyme that maintains intracellular ubiquitin pools and promotes axonal transport. Uchl1 deletion in mice leads to progressive axonal degeneration, affecting the dorsal root gan