Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Nandagopal Venkataprasad"'
Publikováno v:
Tuberculosis. 86:349-356
Summary Setting M. tuberculosis (MTB) lose virulence during prolonged culture on artificial media. This loss of virulence is associated with a change in colony morphology. Several studies suggested that trehalose 6,6′ dimycolate (TDM or cord factor
Autor:
Gilles Klopman, Michael R. Jacobs, Jerrold J. Ellner, Nandagopal Venkataprasad, John L. Johnson
Publikováno v:
Journal of Antimicrobial Chemotherapy. 40:841-845
The ability to inhibit the in-vitro growth of mycobacteria within human monocytes is a useful screening assay for novel chemotherapeutic agents. In this study the MICs of a panel of new quinolones were determined by the broth microdilution method for
Publikováno v:
International Archives of Allergy and Immunology. 114:23-29
Immunotherapy as an adjunct to chemotherapy is of interest for optimizing therapeutic regimens for tuberculosis. In this context, we investigated the influence and mode of action ofglucosaminylmuramyl dipeptide (GMDP) in mouse experimental models. In
Autor:
Gilles Klopman, Michael R. Jacobs, Jerrold J. Ellner, Nandagopal Venkataprasad, Anthony J. Pearson, Hiroe Shiratsuchi
Publikováno v:
Journal of Antimicrobial Chemotherapy. 37:491-500
Mycobacterium avium frequently causes disseminated infection in advanced AIDS. Some quinolones including ciprofloxacin and sparfloxacin have anti-M. avium activity in cell-free systems in vitro. Acidic conditions within macrophages and variable intra
Autor:
Nandagopal Venkataprasad
Publikováno v:
Annals of the New York Academy of Sciences. 832
These results show that given a source of mouse tissue macrophage, there is differential mycobacterial growth with different species of mycobacteria. This suggests that each species of mycobacteria generates a differential response in a given tissue
Publikováno v:
The Journal of infectious diseases. 174(4)
Prostanoids, including prostaglandin E2 (PGE2), suppress macrophage effector functions against Mycobacterium tuberculosis. PGE2 production by monocytes infected with Mycobacterium avium complex (MAC) and its effects on intracellular mycobacterial gro