Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Nancy L. Derevjanik"'
Autor:
Melissa A. Vinores, Diane A Klein, Barbara Detrick, Yun Wang, John J. Hooks, Nancy L. Derevjanik, Stanley A. Vinores, Albert Shi
Publikováno v:
Journal of Neuroimmunology
Intraocular coronavirus inoculation results in a biphasic retinal disease in susceptible mice (BALB/c) characterized by an acute inflammatory response, followed by retinal degeneration associated with autoimmune reactivity. Resistant mice (CD-1), whe
Autor:
Nancy L. Derevjanik, Peter A. Campochiaro, Donald J. Zack, William J. LaRochelle, Haruhiko Yamada, Sean F. Hackett, Naoyuki Okamoto, Man Seong Seo, Melissa A. Vinores, Eri Yamada, Stanley A. Vinores
Publikováno v:
The American Journal of Pathology. 157:995-1005
Expression of platelet-derived growth factor (PDGF)-A and PDGF-B is increased in patients with proliferative retinopathies in which traction retinal detachments occur. Previous studies have demonstrated that increased expression of PDGF-A in the reti
Autor:
Hiroaki Ozaki, Stanley A. Vinores, Naoyuki Okamoto, Peter A. Campochiaro, Nancy L. Derevjanik
Publikováno v:
Documenta Ophthalmologica. 97:217-228
PurposeTo determine the mechanism of blood-retinal barrier (BRB) dysfunction in human and experimental specimens using immunocytochemistry. Methods Extravascular albumin was localized in clinical specimens and retinas from transgenic mice that overex
Autor:
Stanley A. Vinores, Takao Tobe, Chi Chao Chan, Bo Peng, Nancy L. Derevjanik, Charlie Chiu, Jeremy Mahlow, J. D. Luna, Peter A. Campochiaro
Publikováno v:
Journal of Neuroscience Research. 49:268-280
Experimental autoimmune uveoretinitis (EAU) induced in Lewis rats by immunization with S-antigen is a model of human uveitis. By using immunocytochemical staining for albumin, relatively minor blood-retinal barrier (BRB) breakdown was initially shown
Publikováno v:
The Histochemical Journal. 26:655-665
Post-surgical macular oedema results from blood-retinal barrier breakdown, but it is not accompanied by structural abnormalities in the retinal vessels or retinal pigmented epithelium. Previous studies, using horseradish peroxidase in a primate model
Publikováno v:
Brain research. Developmental brain research. 140(2)
Platelet-derived growth factor (PDGF) is necessary for the normal development of the retinal vasculature and its overexpression is likely to contribute to proliferative retinal disorders, such as proliferative vitreoretinopathy. Transgenic mice that
Autor:
Nancy L, Derevjanik, Stanley A, Vinores, Wei-Hong, Xiao, Keisuke, Mori, Tara, Turon, Tyler, Hudish, Steve, Dong, Peter A, Campochiaro
Publikováno v:
Investigative ophthalmologyvisual science. 43(7)
The purpose of this study was to develop and characterize a quantitative assay of blood-retinal barrier (BRB) function in mice and to determine the effect of several purported vasopermeability factors on the BRB.Adult C57BL/6J mice were treated with
Autor:
Nancy L. Derevjanik, Man Seong Seo, John D. Ash, Naoyuki Okamoto, Peter A. Campochiaro, Wei Hong Xiao, T. Hudish, E.F. Wawrousek, Stanley A. Vinores
Publikováno v:
General pharmacology. 35(5)
Following chronic ischemia, vascular endothelial growth factor (VEGF) is induced primarily in the ganglion cell layer of the retina. This often results in neovascularization (NV) that originates from the vascular bed closest to the ganglion cell laye
Publikováno v:
Journal of neuroimmunology. 114(1-2)
Endothelial-monocyte-activating polypeptide (EMAP) is a proinflammatory cytokine and a mediator of programmed endothelial cell death. To gain insight into its possible functions during retinal development and degeneration, the cellular distribution o
Autor:
Naoyuki Okamoto, Stanley A. Vinores, Nancy L. Derevjanik, Peter A. Campochiaro, Melissa A. Vinores
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9781461368953
Neovascularization (NV) causes visual deficits in ocular disorders such as diabetic retinopathy, age-related macular degeneration, and retinopathy of prematurity. An understanding of the angiogenic factors promoting this abnormal vascular growth is n
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::80156da7a5ffeb48bd34c26b0e8b5806
https://doi.org/10.1007/978-1-4615-4221-6_11
https://doi.org/10.1007/978-1-4615-4221-6_11