Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Nadine Steinbronn"'
Autor:
Stefan Lange, Rüdiger C. Braun-Dullaeus, Nadine Steinbronn, Clementine Fischaleck, Doreen Hollenbach, Ruth H. Strasser, Annette Ebner, Christof Weinbrenner, Bernd Ebner
Publikováno v:
Canadian Journal of Cardiology. 31:76-83
Background The myocardial infarct size can be reduced by pharmacological postconditioning using cardioprotective agents. Neuregulin-1β is a potential candidate, but previous studies in an isolated heart model of ischemia and reperfusion displayed co
Autor:
Nadine Steinbronn, Ruth H. Strasser, Sonja Schallenberg, Marcus Gereke, Robert Geffers, Dunja Bruder, Cathleen Petzold, Tim Sparwasser, Karsten Kretschmer
Publikováno v:
European Journal of Immunology. 44:3632-3645
Under physiological conditions, studies on the biology of naturally induced Foxp3(+) Treg cells of intra- and extrathymic origin have been hampered by the lack of unambiguous markers to discriminate the mature progeny of such developmental Treg-cell
Autor:
Anja Hofmann, R. A. Catar, Undine Schubert, Nadine Steinbronn, Aljoscha Brandt, Coy Brunssen, Annika Frenzel, Claudia Goettsch, Stefan R. Bornstein, Ruth H. Strasser, Winfried Goettsch, Barbara Ludwig, Henning Morawietz, Gregor Muller, Heike Langbein
Publikováno v:
Hormone and Metabolic Research. 47:509-515
Obesity is a well-known risk factor of atherosclerosis and heart failure. In the human heart, a local endothelin system containing prepro-endothelin-1, endothelin-converting enzyme-1, and endothelin receptors A and B has been described. The endotheli
Autor:
Ruth H. Strasser, Wen Chen, Jens Waschke, Viacheslav O. Nikolaev, Birgit Gaßner, Nadine Steinbronn, Nicolas Schlegel, Sebastian Börner, Michaela Kuhn
Publikováno v:
Cardiovascular Research
Aims Cardiac atrial natriuretic peptide (ANP) participates in the maintenance of arterial blood pressure and intravascular volume homeostasis. The hypovolaemic effects of ANP result from coordinated actions in the kidney and systemic microcirculation
Autor:
Cathleen, Petzold, Nadine, Steinbronn, Marcus, Gereke, Ruth H, Strasser, Tim, Sparwasser, Dunja, Bruder, Robert, Geffers, Sonja, Schallenberg, Karsten, Kretschmer
Publikováno v:
European journal of immunology. 44(12)
Under physiological conditions, studies on the biology of naturally induced Foxp3(+) Treg cells of intra- and extrathymic origin have been hampered by the lack of unambiguous markers to discriminate the mature progeny of such developmental Treg-cell
Autor:
Annette Ebner, Aljoscha Brandt, Anja Hofmann, Mathias Forkmann, Bernd Ebner, Ruth H. Strasser, Anne-Kathrin Tausche, Henning Morawietz, Nadine Steinbronn, Anne-Katrin Polanski, Carsten Wunderlich
Publikováno v:
Journal of cardiovascular pharmacology. 61(6)
Caveolin-1-deficient (cav1) mice display a severely diseased cardiac phenotype with systolic and diastolic heart failure. Accumulating evidence supports a causative role of uncoupled endothelial nitric oxide synthase in the development of these abnor
Autor:
Nadine Steinbronn, Anne-Kathrin Tausche, Ruth H. Strasser, Carsten Schwencke, Ruediger C. Braun-Dullaeus, Christian Heerwagen, Carsten Wunderlich, Aljoscha Brandt, Michael Kasper, Alexander Schmeisser, Kristin Schober
Publikováno v:
Journal of molecular and cellular cardiology. 44(5)
Recently generated caveolin-1 deficient mice (cav-1(-/-)) display several physiological alterations such as severe heart failure and lung fibrosis. The molecular mechanisms how the loss of caveolin-1 (cav-1) mediates these alterations are currently u
Publikováno v:
Cardiovascular Research. 103:S142.3-S142
Mice lacking Caveolin-1 (cav1ko) develop a severe hypertrophic cardiomyopathy (HCM). The HCM in this model is induced by an uncoupling of the endothelial NO-synthase, which leads to the enhanced production of radicals. The main characteristic of this
Autor:
Roland Jung, Ruth H. Strasser, Nadine Steinbronn, Paula Geidel, Meinolf Suttorp, Josephine Tabea Tauer, Ivo Leuschner
Publikováno v:
Blood. 122:3984-3984
Background Treatment of chronic myeloid leukemia (CML) by tyrosine kinase inhibitors (TKIs, e.g. imatinib, dasatinib) can result in cardiac failure as these drugs exert off-target effects via so far ill-defined mechanisms. Especially in pediatric pat
Autor:
Carsten Wunderlich, Ruth H. Strasser, David M. Poitz, Nadine Steinbronn, Bernd Ebner, Annette Ebner, J. Mitschke
Publikováno v:
European Heart Journal. 34:P5042-P5042
Purpose: Heart failure is one of the most prevalent causes of death in the western world. Caveolin1 knockout mice (cav1-/-) develop a severe hypertrophic cardiomyopathy with functional impairment especially in cardiac output and ejection fraction. Be