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Autor:
Faramarz Dehghani, Gunnar T. R. Hischebeth, Florentina Wirjatijasa, Horst-Werner Korf, Nils P. Hailer, Nadine Roser
Publikováno v:
European Journal of Neuroscience. 14:315-326
Acute CNS lesions lead to neuronal injury and a parallel glial activation that is accompanied by the release of neurotoxic substances. The extent of the original neuronal damage can therefore be potentiated in a process called secondary damage. As as