Zobrazeno 1 - 10
of 44
pro vyhledávání: '"N. de Bont"'
Autor:
N De Bont, Thierry Lejeune, Thierry Deltombe, V Halkin, M Rahamatali, Maxime Valet, P Hanson, C Selves
Publikováno v:
Acta neurologica Belgica, Vol. 121, no. 1, p. 181-189 (2021)
Post-stroke fatigue (PSF) is a common symptom associated with disability and decreased quality of life. Distinction can be made between perceived fatigue and fatigability. The first aim of this study was to evaluate the prevalence of perceived fatigu
Publikováno v:
Spinal cord series and cases. 4
STUDY DESIGN: Pilot retrospective study on the outcome of open surgery for grade III and IV haemorrhoids in patients with SCI. OBJECTIVE: Haemorrhoids and anal fissures are common in patients with spinal cord injury (SCI). Grade I to III haemorrhoids
Autor:
Pierre N.M. Demacker, N. de Bont, Mihai G. Netea, B.J. Kullberg, A.F.H. Stalenhoef, J.W.M. van der Meer
Publikováno v:
European Journal of Clinical Investigation. 30:818-822
Background All lipoproteins are able to bind to bacterial lipopolysaccharide (LPS), thereby neutralizing its deleterious effects. However, we demonstrated, recently, that in the absence of apolipoprotein E (apoE), eight-fold increased very-low-densit
Autor:
Tineke J. Smilde, Carl G. Figdor, N. de Bont, T.B.H. (Theo) Geijtenbeek, Mihai G. Netea, A.F.H. Stalenhoef, J.W.M. van der Meer, Pierre N.M. Demacker
Publikováno v:
European Journal of Clinical Investigation. 29:749-757
Background Low-density lipoproteins (LDL) can induce the adhesion of monocytes to endothelial cells. Monocytes of patients with familial hypercholesterolemia (FH) are exposed to high concentrations of LDL, and it has been reported that adhesiveness o
Autor:
Chantal P. Bleeker Rovers, N. de Bont, J.W.M. van der Meer, Cor Jacobs, Mihai G. Netea, A.F.H. Stalenhoef, B.J. Kullberg, P.N.M. Demacker
Publikováno v:
Atherosclerosis, 141, 99-105
ResearcherID
Atherosclerosis, 141, 1, pp. 99-105
Atherosclerosis, 141, pp. 99-105
Atherosclerosis, 141, 99-105. Elsevier sci ireland ltd
ResearcherID
Atherosclerosis, 141, 1, pp. 99-105
Atherosclerosis, 141, pp. 99-105
Atherosclerosis, 141, 99-105. Elsevier sci ireland ltd
It has been suggested that proinflammatory cytokines such as tumor necrosis factor-alpha (TNF) and interleukin-1beta (IL-1), as well as adhesion molecules such as beta2-integrins and CD14, play a role in the pathogenesis of atherosclerosis. Familial
Autor:
N. de Bont, Mihai G. Netea, Tineke J. Smilde, C.P. Rovers, J.W.M. van der Meer, Pierre N.M. Demacker, A.F.H. Stalenhoef
Publikováno v:
Atherosclerosis, 139, 147-152
Atherosclerosis, 139, 147-152. Elsevier sci ireland ltd
Atherosclerosis, 139, pp. 147-152
Atherosclerosis, 139, 1, pp. 147-152
Atherosclerosis, 139, 147-152. Elsevier sci ireland ltd
Atherosclerosis, 139, pp. 147-152
Atherosclerosis, 139, 1, pp. 147-152
Inflammatory processes play an important role in atherogenesis, and proinflammatory cytokines such as interleukin-1beta (IL-1beta), IL-6, and tumour necrosis factor alpha (TNFalpha) are thought to be mediators in this phenomenon. We have previously e
Autor:
M.G. Netea, E. Dharmana, Anneke Hijmans, J.P.H. Drenth, Pierre N.M. Demacker, J.W.M. van der Meer, Monique Keuter, N. de Bont
Publikováno v:
Cytokine, 8, 9, pp. 739-744
Cytokine, 8, 739-744
Cytokine, 8, 739-744
Whole blood cultures are used to study cytokine stimulation and release ex vivo. In the present study this method was compared with a more direct approach and a quantitative reverse transcriptase polymerase chain reaction (RT-PCR) was used to assess
Publikováno v:
European journal of clinical investigation. 30(9)
All lipoproteins are able to bind to bacterial lipopolysaccharide (LPS), thereby neutralizing its deleterious effects. However, we demonstrated, recently, that in the absence of apolipoprotein E (apoE), eight-fold increased very-low-density lipoprote
Autor:
I. Verschueren, J.W.M. van der Meer, A.F.H. Stalenhoef, B.J. Kullberg, N. de Bont, Pierre N.M. Demacker, Mihai G. Netea, K.W. van Dijk
Publikováno v:
Journal of Lipid Research, 40, 4, pp. 680-685
Journal of Lipid Research (Online), 40, 680-685
Journal of Lipid Research, Vol 40, Iss 4, Pp 680-685 (1999)
Journal of Lipid Research, 40, 680-685
Journal of Lipid Research (Online), 40, 680-685
Journal of Lipid Research, Vol 40, Iss 4, Pp 680-685 (1999)
Journal of Lipid Research, 40, 680-685
Lipoproteins are able to neutralize bacterial lipopolysaccharide (LPS) and thereby inhibit the proinflammatory cytokine response. In a previous study, we demonstrated that hypercholesterolemic low density lipoprotein receptor knock-out (LDLr–/–)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a1c3ca2f11e85af69694bae9d7f297fd
https://hdl.handle.net/2066/224165
https://hdl.handle.net/2066/224165
Autor:
Mihai G. Netea, B.J. Kullberg, Liesbeth E. H. Jacobs, J.W.M. van der Meer, N. de Bont, Anton F. H. Stalenhoef, Pierre N.M. Demacker, Trees J.G. Verver-Jansen
Publikováno v:
Infection and immunity. 66(5)
Lipoproteins can bind lipopolysaccharide (LPS) and decrease LPS-stimulated cytokine production. Lipoprotein(a) [Lp(a)] was as potent as low-density lipoproteins (LDL) in inhibiting LPS-stimulated tumor necrosis factor synthesis by human mononuclear c