Zobrazeno 1 - 10
of 10
pro vyhledávání: '"N. S. Mantorova"'
Autor:
A. B. Salmina, V. V. Salmin, O. V. Frolova, D. I. Laletin, M. A. Fursov, D. P. Skomorokha, A. A. Fursov, M. A. Kondrashov, N. N. Medvedeva, N. A. Malinovskaya, N. S. Mantorova
Publikováno v:
Анналы клинической и экспериментальной неврологии, Vol 5, Iss 3, Pp 32-39 (2017)
The paper contains results of the investigation of the laser-inducedfluorescence detection method for the assessment ofbrain metabolism in situ through the dura mater. Models ofanoxia and acute brain ischemia were used for the evaluation ofreliabilit
Externí odkaz:
https://doaj.org/article/a0c69751938d48e9b675d9d420ab45c7
Publikováno v:
Общая реаниматология, Vol 4, Iss 1 (2008)
The present views of the pathogenesis of neuronal dysfunction in critical conditions are analyzed, by taking into account of impairments of cellular NAD+ metabolism, the activity of NAD+-converting enzymes, including ADP-ribosyl cyclase/CD38, the pos
Externí odkaz:
https://doaj.org/article/0a1946cacedc497eacd81c3a27fda393
Autor:
N. S. Mantorova, S. V. Mikhutkina, O. S. Okuneva, Alla B. Salmina, A. V. Morgun, T. E. Taranushenko, Natalia A. Malinovskaya
Publikováno v:
Neurochemical Journal. 2:215-221
Using a model of perinatal hypoxic-ischemic injury of rat brain we have found the patterns of changes in the activity of ADP ribosyl cyclase in brain cells that determine the specific features of programmed cell death and maintenance of the intracell
Autor:
S. V. Mikhutkina, Alla B. Salmina, A. A. Fursov, K. Y. Linev, S. V. Shakhmaeva, Viktor Moroz, N. S. Mantorova
Publikováno v:
Bulletin of Experimental Biology and Medicine. 147:570-572
The pathogenesis of neuronal dysfunction was evaluated from the viewpoint of cellular disturbances in NAD(+) metabolism and changes in activity of NAD(+)-utilizing enzymes (e.g., ADP-ribosyl cyclase/CD38). S-100B concentration and CD38 expression on
Autor:
Iu A, Uspenskaia, A B, Salmina, N A, Malinovskaia, N S, Mantorova, E F, Vaĭs, L A, Kushnir, A D, Klimova
Publikováno v:
Eksperimental'naia i klinicheskaia farmakologiia. 70(1)
Mechanisms of the myelotoxic action of doxorubicin associated with changes in the expression and functional activity of P2X7 receptors have been assessed. The acute and subacute exposure of bone marrow cells to doxorubicin in vivo changed the express
Publikováno v:
Ukrainskii biokhimicheskii zhurnal (1. 63(2)
Activation of purified glutathione transferase (CE 2.5.1.18) from the rabbit liver by cAMP-dependent protein kinase (protein kinase A) and activation of glutathione transferase from the rat liver and heart by cAMP preparations have been studied. A co
Publikováno v:
Patologicheskaia fiziologiia i eksperimental'naia terapiia. (4)
Immobilization of rats for 20 minutes and for 4 hours activates glutathione peroxidase in the heart, liver, and kidneys and glutathione transferase in the heart and liver, but inhibits gamma-glutamyl transferase in the liver and kidneys. Most of thes
Publikováno v:
Ukrainskii biokhimicheskii zhurnal (1. 62(4)
The phenobarbital and ionol administration to rats and mice increases considerably the glutathione transferase, glutathione reductase and gamma-glutamyl transferase activities in the liver. The induction of these enzymes has been observed in a number
Publikováno v:
Voprosy onkologii. 33(3)
Tumors were studied in 38 patients, tissues located far away from tumor being used as control. Breast and stomach tumors revealed relatively high levels of glutathione reductase, glutathione-S-transferase (I) and, particularly, gamma-glutamyl transfe
Publikováno v:
Voprosy meditsinskoi khimii. 33(3)
Emotional stress during 20 min induced the stimulation of the glutathione peroxidase activity in heart, liver and kidney tissues and the activation of glutathione-S-transferase in heart and liver tissues. gamma-Glutamyl transferase activity was inhib