Zobrazeno 1 - 10
of 25
pro vyhledávání: '"N. J. Cairns"'
Publikováno v:
Neuroscience. 248
The regulation of D₃ receptor has not been well documented in diffuse Lewy body disease (DLBD). In this study, a novel D₃-preferring radioligand [(3)H]WC-10 and a D₂-preferring radioligand [(3)H]raclopride were used and the absolute densities o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::7c9cd6885a0e16813d44598fcab8219d
https://pubmed.ncbi.nlm.nih.gov/23732230
https://pubmed.ncbi.nlm.nih.gov/23732230
Autor:
Felicity Nicholson, P. Lantos, Henry J. Kaminski, N. J. Cairns, G. T. Spencer, E. J. Thompson, P. Muir, J. E. Banatvala, M. K. Sharief
Publikováno v:
Annals of the New York Academy of Sciences. 753:219-232
It has been suggested that late onset neurological deterioration after poliomyelitis may be due in some cases to persistent poliovirus infection of the central nervous system. In view of this, we decide to determine whether polioviruses and other ent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79080336f5a09ff9b2207a6eb3690dc9
https://doi.org/10.1111/j.1749-6632.1995.tb27548.x
https://doi.org/10.1111/j.1749-6632.1995.tb27548.x
Publikováno v:
Neuropathology and applied neurobiology. 38(1)
To further characterize the neuropathology of the heterogeneous molecular disorder frontotemporal lobar degeneration (FTLD) with transactive response (TAR) DNA-binding protein of 43 kDa (TDP-43) proteinopathy (FTLD-TDP).We quantified the neuronal cyt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::54854c36c3175225a426c6674b32952b
https://pubmed.ncbi.nlm.nih.gov/21696412
https://pubmed.ncbi.nlm.nih.gov/21696412
Autor:
R A, Armstrong, N J, Cairns
Publikováno v:
Clinical neuropathology. 28(5)
To determine the laminar distribution of the pathological changes in the cerebral cortex in progressive supranuclear palsy (PSP).The distribution of the abnormally enlarged neurons (EN), surviving neurons, neurofibrillary tangles (NFT), glial inclusi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::10388f6ea6159fbde3d59f1d8a55df50
https://pubmed.ncbi.nlm.nih.gov/19788050
https://pubmed.ncbi.nlm.nih.gov/19788050
Autor:
R A, Armstrong, N J, Cairns
Publikováno v:
Clinical neuropathology. 25(5)
To determine the laminar distribution of the pathological changes in the frontal and temporal lobe in neuronal intermediate filament inclusion disease (NIFID).The distribution of the alpha-internexin-positive neuronal cytoplasmic inclusions (NCI), su
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9267afc9bb71bcd168ecfb4738702f0d
https://pubmed.ncbi.nlm.nih.gov/17007442
https://pubmed.ncbi.nlm.nih.gov/17007442
Publikováno v:
Clinical neuropathology. 24(5)
To determine the distribution of the pathological changes in the neocortex in multiple-system atrophy (MSA).The vertical distribution of the abnormal neurons (neurons with enlarged or atrophic perikarya), surviving neurons, glial cytoplasmic inclusio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::609049292e11c912f0608826ecf187c5
https://pubmed.ncbi.nlm.nih.gov/16167547
https://pubmed.ncbi.nlm.nih.gov/16167547
Autor:
N J, Cairns, R A, Armstrong
Publikováno v:
Clinical neuropathology. 23(3)
To quantify the densities of neurofilament inclusions (NI), swollen achromatic neurons, surviving neurons and glial cells in a novel neurofilamentopathy neurofilament inclusion disease (NID).Sections of temporal lobe from 4 cases of NID stained with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::11a33434723f5c2b3926217fd0a33448
https://pubmed.ncbi.nlm.nih.gov/15200288
https://pubmed.ncbi.nlm.nih.gov/15200288
Publikováno v:
Clinical neuropathology. 21(6)
To study the spatial patterns of the vacuolation ("spongiform change") in the subcortical white matter in the "classical" form of sporadic Creutzfeldt-Jakob disease (sCJD).Frontal, parietal, occipital and temporal lobes of 11 cases of sCJD.Spatial pa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::65d05a66eb17790183e490117b5bc1a4
https://pubmed.ncbi.nlm.nih.gov/12489678
https://pubmed.ncbi.nlm.nih.gov/12489678
Autor:
N J, Cairns
Publikováno v:
Journal of neural transmission. Supplementum. (61)
Down syndrome (DS) is a complex, clinically heterogeneous disorder which shows both impairment of neurodevelopement and the neurodegenerative changes of Alzheimer's disease (AD). The phenotype of DS is caused by triplication of chromosome 21 and tran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ffdc00502ff416326f7d0436c155ebd1
https://pubmed.ncbi.nlm.nih.gov/11771752
https://pubmed.ncbi.nlm.nih.gov/11771752
Autor:
D M, Mann, A, Takeuchi, S, Sato, N J, Cairns, P L, Lantos, M N, Rossor, M, Haltia, H, Kalimo, T, Iwatsubo
Publikováno v:
Neuropathology and applied neurobiology. 27(3)
The pattern of deposition of amyloid beta protein (Abeta) was investigated, using the monoclonal antibodies BA27 and BC05 detecting the C-terminal species Abeta40 and Abeta42(43), in six cases of Alzheimer's disease (AD) due to deletions in exon 9 of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1f82d50f4ae40252f4c51296c3165bcb
https://pubmed.ncbi.nlm.nih.gov/11489138
https://pubmed.ncbi.nlm.nih.gov/11489138