Výsledky vyhledávání - "N V, Krishtal'"

[The participation of the renin-angiotensin-aldosterone system in the pathogenesis of acute kidney failure in endotoxemia]

Autor: N V, Krishtal', A F, Frolov, A L, Kukharchuk

Publikováno v: Likars'ka sprava. (3-4)

It was found in acute experiments on white rats that injection of Salmonella typhimurium lipopolysaccharide activates renin-angiotensin-aldosterone system (RAAS) and causes oligohydruria form of acute renal failure (ARF). Enalapril, angiotensin-conve

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::40c47c12846150f6cdf1b484078564f2
https://pubmed.ncbi.nlm.nih.gov/8819932

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[Mechanisms of renal adaptation to disorders of water-electrolyte balance in metabolic acidosis]

Autor: N V, Krishtal', A I, Gozhenko, E G, Gareeva

Publikováno v: Patologicheskaia fiziologiia i eksperimental'naia terapiia. (3)

The albino rat experiments have demonstrated that despite the type, metabolic acidosis causes an increase in plasma and urinary osmolarity, hyperkalemia and hypernatremia, enhanced urinary excretion of potassium, phosphates, and titrated acids with a

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::f7a92317a653206db68c29beca12b0dc
https://pubmed.ncbi.nlm.nih.gov/7824347

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[The mechanisms of disorders of carbohydrate metabolism in changes to the acid-base balance]

Autor: N V, Krishtal', E G, Gareeva

Publikováno v: Biulleten' eksperimental'noi biologii i meditsiny. 116(9)

Experiments on white rats found that both chronic acidosis and chronic alkalosis cause fasting hyperglycemia and decreases glucose tolerance. In hypophysectomized animals, alkalosis causes similar effects and acidosis leads to hypoglycemia. Acute aci

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::aa1fce47c01e64f5fe7f205ea4540827
https://pubmed.ncbi.nlm.nih.gov/8117990

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[Endocrine regulation of chlorine ion transport in rat kidneys]

Autor: N V, Krishtal'

Publikováno v: Problemy endokrinologii. 39(3)

Eighty-five intact, adrenalectomized and hypophysectomized white male rats fed low-caloric diets were used in experiments. Hormonal effects on urinary excretion of chlorides and sodium were studied in hyperhydration conditions; these hormones' levels

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::35ae2c295682d92badd4739d4ce99739
https://pubmed.ncbi.nlm.nih.gov/8058684

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[Effect of prostaglandin E2 on ion regulating and acid excretory function of the kidneys]

Autor: N V, Krishtal'

Publikováno v: Problemy endokrinologii. 38(2)

Prostaglandin E2 action on ion regulating and acid excretory function of the kidneys was investigated in acute experiments on intact male rats whose angiotensin-converting enzyme (ACE) had been blocked by enalapril. It was shown that i.p. two-fold PG

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::0fd23beb2574ba47a9027c706bf87e21
https://pubmed.ncbi.nlm.nih.gov/1513778

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[The adrenergic and dopaminergic regulation of kidney acid-excreting function]

Autor: N V, Krishtal'

Publikováno v: Fiziologicheskii zhurnal. 38(2)

It has been found in experiments on white rats that the activation of peripheral alpha 1-adrenoreceptors and dopamine receptors of type I stimulates acid-excretory renal function with inhibition of the angiotensin-aldosterone system. The blockade of

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::03a3ba14dd5efb93829ca1654b4c9a29
https://pubmed.ncbi.nlm.nih.gov/1568499

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[Adrenergic effects on the renal function and their mechanisms]

Autor: N V, Krishtal'

Publikováno v: Fiziologicheskii zhurnal. 37(3)

The experiments on rats have shown that the activation of alpha-adrenoceptors decreases the glomerular filtration as well as sodium and water reabsorption against a background of a decrease in the blood content of vasopressin, renin, angiotensin II,

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::3d12e0a23df501f36363c539c7808625
https://pubmed.ncbi.nlm.nih.gov/1654283

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