Zobrazeno 1 - 10
of 13
pro vyhledávání: '"N H Manson"'
Publikováno v:
Circulation research. 53(5)
Human peripheral blood leukocytes, activated by phorbol myristate acetate, disrupt canine sarcoplasmic reticulum calcium transport, in vitro, by an oxygen-derived free radical mechanism. Activated leukocytes significantly depress Ca++ uptake activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f467f2a762e04ce051d0ce3fec347fe4
https://pubmed.ncbi.nlm.nih.gov/6138170
https://pubmed.ncbi.nlm.nih.gov/6138170
Autor:
J V, Boykin, N H, Manson
Publikováno v:
Virginia medical. 115(5)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::668045953caa6a2440465bc9a3164be2
https://pubmed.ncbi.nlm.nih.gov/3388964
https://pubmed.ncbi.nlm.nih.gov/3388964
Publikováno v:
Transplantation. 36(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::f19097baea92a696b53ee27f1e4f1f7c
https://pubmed.ncbi.nlm.nih.gov/6306881
https://pubmed.ncbi.nlm.nih.gov/6306881
Publikováno v:
Circulatory shock. 10(2)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e249203f44abac9b6a71283b62fa3fc1
https://pubmed.ncbi.nlm.nih.gov/6301705
https://pubmed.ncbi.nlm.nih.gov/6301705
Publikováno v:
Developments in Cardiovascular Medicine ISBN: 9781461296393
The following study has two principal aims: 1) the demonstration that oxygen free radicals possess negative inotropic properties and 2) the characterization of the role of the oxygen free radicals during myocardial ischemia in producing the disruptio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::831a0d1c61d4307c1e585a10181c8631
https://doi.org/10.1007/978-1-4613-2621-2_24
https://doi.org/10.1007/978-1-4613-2621-2_24
Autor:
M L, Hess, N H, Manson
Publikováno v:
Circulatory shock. 10(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::bec78c4d38c5b8000e5f1f2e15cc17fb
https://pubmed.ncbi.nlm.nih.gov/6299603
https://pubmed.ncbi.nlm.nih.gov/6299603
Autor:
M L, Hess, N H, Manson
Publikováno v:
Advances in myocardiology. 5
The pathways for the metabolism of molecular oxygen involve one electron-transfer reaction with the subsequent production of reduced-oxygen intermediates. These reduced-oxygen intermediates include the superoxide anion (.O2-), hydrogen peroxide (H2O2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a4800ad952a8c76d6a654483cf5332b8
https://pubmed.ncbi.nlm.nih.gov/2982205
https://pubmed.ncbi.nlm.nih.gov/2982205
Autor:
N H, Manson, M L, Hess
Publikováno v:
Circulatory shock. 10(3)
An initial event in gram-negative bacteremia is activation of the complement cascade with production of C5a. C5a, in turn, acts as a chemotactic stimulus for leukocytic aggregation and, in conjunction with bacterial products, stimulates the release o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c70788c2881b4186d338f69762a777d6
https://pubmed.ncbi.nlm.nih.gov/6851003
https://pubmed.ncbi.nlm.nih.gov/6851003
Publikováno v:
Oxygen Radicals in the Pathophysiology of Heart Disease ISBN: 9781461289791
Myocardial ischemia results in a series of metabolic events that include the autooxidation of catecholamines, a reduction in intracellular pH, the breakdown of ATP to hypoxanthine and xanthine, an increase in reducing equivalents and activation of th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::69a34c9b875692aab0de53073580c7b0
https://doi.org/10.1007/978-1-4613-1743-2_8
https://doi.org/10.1007/978-1-4613-1743-2_8
Publikováno v:
Advances in shock research. 6
The interrelationships between hemodynamics and hypoglycemia during the course of endotoxin shock (ES) has not been fully defined. In the following study, ES (E. coli, 1 mg/kg; n = 7) was induced in a canine model and systemic hemodynamics, glucose m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::878c17d470225f3358219b8e39e3fdd0
https://pubmed.ncbi.nlm.nih.gov/7349583
https://pubmed.ncbi.nlm.nih.gov/7349583