Zobrazeno 1 - 10
of 37
pro vyhledávání: '"Mya Mya Mu"'
Autor:
Jargalsaikhan Dagvadorj, Ferdaus Hassan, Naoki Koide, Shamima Islam, Yoshikazu Naiki, Gantsetseg Tumurkhuu, Tomoaki Yoshida, Isamu Mori, Takashi Yokochi, Mya Mya Mu
Publikováno v:
Journal of Endotoxin Research. 13:167-175
Lipopolysaccharide (LPS) enhances the production of nitric oxide (NO) in interferon (IFN)-gamma-stimulated vascular endothelial cells. We studied the mechanism by which LPS enhances IFN-gamma-induced NO production by using the murine vascular endothe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a03eff62c1c3bb03aeb08ab87895d7e2
https://doi.org/10.1177/0968051907080894
https://doi.org/10.1177/0968051907080894
Autor:
Isamu Mori, Takashi Yokochi, Tomoaki Yoshida, Shamima Islam, Hiroyasu Ito, Naoki Koide, Ferdaus Hassan, Mya Mya Mu
Publikováno v:
Molecular Cancer Research. 3:373-379
The effect of lipopolysaccharide on doxorubicin-induced cell death was studied by using mouse RAW 264.7 macrophage cells. Pretreatment with lipopolysaccharide at 10 ng/mL prevented doxorubicin-induced cell death and the inhibition was roughly depende
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e270d47aad60b8cfadcc646d53b6bba
https://doi.org/10.1158/1541-7786.mcr-05-0046
https://doi.org/10.1158/1541-7786.mcr-05-0046
Autor:
Tomoaki Yoshida, Isamu Mori, Mya Mya Mu, Takashi Yokochi, Shamima Islam, Ferdaus Hassan, Hiroyasu Ito, Naoki Koide
Publikováno v:
Microbiology and Immunology. 48:807-815
The role of p38 mitogen-activated protein kinase (MAPK) on vacuole formation in lipopolysaccharide (LPS)-stimulated RAW 264.7 cells was examined. LPS definitely induced the formation of vacuoles in RAW 264.7 cells and SB202190 as a p38 specific inhib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f78c05f9a4180f3e63717fecd79840b6
https://doi.org/10.1111/j.1348-0421.2004.tb03612.x
https://doi.org/10.1111/j.1348-0421.2004.tb03612.x
Autor:
Naoki Koide, Takashi Yokochi, Shamima Islam, Hiroyasu Ito, Ferdaus Hassan, Isamu Mori, Tomoaki Yoshida, Mya Mya Mu
Publikováno v:
Microbiology and Immunology. 48:729-736
The effect of piceatannol on lipopolysaccharide (LPS)-induced nitric oxide (NO) production was examined. Piceatannol significantly inhibited NO production in LPS-stimulated RAW 264.7 cells. The inhibition was due to the reduced expression of an induc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d4768f4460bd955248a58552a44a547
https://doi.org/10.1111/j.1348-0421.2004.tb03598.x
https://doi.org/10.1111/j.1348-0421.2004.tb03598.x
Autor:
Teruaki Hamano, Takashi Yokochi, Isamu Mori, Naoki Koide, Tsuyoshi Sugiyama, Tomoaki Yoshida, Mya Mya Mu
Publikováno v:
Microbiology and Immunology. 47:669-679
The in vitro effect of gamma interferon (IFN-gamma) on nitric oxide (NO) production in a mouse CD5+ B1-like cell line, TH2.52, was studied. The TH2.52 cell line is the hybridoma line between mouse B lymphoma line and mouse splenic B cells and express
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fac24e18301e305f020438f121092714
https://doi.org/10.1111/j.1348-0421.2003.tb03430.x
https://doi.org/10.1111/j.1348-0421.2003.tb03430.x
Publikováno v:
Journal of Endotoxin Research. 9:85-90
The effect of C(2)-ceramide, a membrane-permeable ceramide analogue, on nitric oxide (NO) production in lipopolysaccharide (LPS)-stimulated RAW 264.7 cells was studied. The non-toxic concentration of C(2)-ceramide inhibited LPS-induced NO production.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::782e76c1a52439562928c91a8575f90d
https://doi.org/10.1179/096805103125001450
https://doi.org/10.1179/096805103125001450
Publikováno v:
Journal of Endotoxin Research. 9:85-90
The effect of C2-ceramide, a membrane-permeable ceramide analogue, on nitric oxide (NO) production in lipopolysaccharide (LPS)-stimulated RAW 264.7 cells was studied. The non-toxic concentration of C2-ceramide inhibited LPS-induced NO production. It
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f8eab10454013226abac26c4d18ab2eb
https://doi.org/10.1177/09680519030090020201
https://doi.org/10.1177/09680519030090020201
Autor:
Naoki Koide, Tomoaki Yoshida, Takashi Yokochi, Isamu Mori, Tsuyoshi Sugiyama, Mya Mya Mu, Dipshikha Chakravortty, Kazuko Takahashi
Publikováno v:
Journal of Endotoxin Research. 7:431-438
The effect of quercetin on lipopolysaccharide (LPS)-induced nitric oxide (NO) production was studied. Quercetin pretreatment significantly inhibited NO production in an LPS-stimulated RAW 264.7 murine macrophage cell line. Post-treatment with quercet
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bf904a43bb07dfa2407ae6ad125d0ab8
https://doi.org/10.1177/09680519010070060601
https://doi.org/10.1177/09680519010070060601
Autor:
Naoki Koide, Takashi Yokochi, Kazuko Takahashi, Akiko Morikawa, Mya Mya Mu, Yutaka Kato, Tomoaki Yoshida, T. Sugiyama
Publikováno v:
FEMS Immunology & Medical Microbiology. 31:29-33
The protective effect of flavonoids on two types of lethal endotoxic shock was studied. A lethal endotoxic shock was induced by administration of lipopolysaccharide (LPS) into D-galactosamine (D-GalN)-sensitized mice and another one was done by admin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90141c645e088bf1ddf95240c33818c1
https://doi.org/10.1111/j.1574-695x.2001.tb01582.x
https://doi.org/10.1111/j.1574-695x.2001.tb01582.x
Autor:
Naoki Koide, Takashi Yokochi, Tomoaki Yoshida, Yutaka Kato, Tsuyoshi Sugiyama, Dipshika Chakravortty, Mya Mya Mu
Publikováno v:
Journal of Endotoxin Research. 7:223-226
The surface expression of CD14 on mouse B-1 cells and its role on their response to lipopolysaccharide (LPS) were studied by using the murine TH2.52 B-1 cell line and peritoneal B-1 cells. TH2.52 cells with the B-1 phenotype were found to express mem
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9f63a7fea3547b42ad15aacf72c496df
https://doi.org/10.1179/096805101101532710
https://doi.org/10.1179/096805101101532710