Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Muriel, Auberson"'
Autor:
Elodie Ehret, Sévan Stroh, Muriel Auberson, Frédérique Ino, Yannick Jäger, Marc Maillard, Roman Szabo, Thomas H. Bugge, Simona Frateschi, Edith Hummler
Publikováno v:
Cells, Vol 12, Iss 19, p 2342 (2023)
The serine proteases CAP1/Prss8 and CAP3/St14 are identified as ENaC channel-activating proteases in vitro, highly suggesting that they are required for proteolytic activation of ENaC in vivo. The present study tested whether CAP3/St14 is relevant fo
Externí odkaz:
https://doaj.org/article/c6b5a932c8c94f73bb776dd9d47fb1a1
Autor:
Daniela Cabuzu, Suresh K. Ramakrishnan, Matthias B. Moor, Dusan Harmacek, Muriel Auberson, Fanny Durussel, Olivier Bonny
Publikováno v:
PLoS ONE, Vol 17, Iss 10 (2022)
Kidney stone is one of the most frequent urinary tract diseases, affecting 10% of the population and displaying a high recurrence rate. Kidney stones are the result of salt supersaturation, including calcium and oxalate. We have previously identified
Externí odkaz:
https://doaj.org/article/a3a0e3df4eae43cea203c1d24fe4ba20
Publikováno v:
Revue Médicale Suisse. 18:379-384
Publikováno v:
Revue medicale suisse. 18(771)
Hyperuricemia is often encountered as glomerular filtration rate decreased. It is associated with a more rapid decline of the renal function, but causality has not been demonstrated. Recent studies showed that treatment of hyperuricemia did not affec
Publikováno v:
American journal of physiology. Renal physiology. 316(5)
The objective of the present study was to theoretically investigate the mechanisms underlying uric acid transport in the proximal tubule (PT) of rat kidneys, and their modulation by factors, including Na+, parathyroid hormone, ANG II, and Na+-glucose
Publikováno v:
Inflammatory bowel diseases, vol. 22, no. 12, pp. 2824-2839
Inflammatory bowel diseases (IBD) including ulcerative colitis and Crohn's disease are diseases with impaired epithelial barrier function. We aimed to investigate whether mutated prostasin and thus, reduced colonic epithelial sodium channel activity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::712969a8899849db870d40a3fdccf255
https://serval.unil.ch/notice/serval:BIB_A9B4457513BE
https://serval.unil.ch/notice/serval:BIB_A9B4457513BE
Autor:
Giuseppe Albano, Candice Stoudmann, Daniel Guido Fuster, Olivier Bonny, Muriel Auberson, Wilhelm Hofstetter, Annie Mercier-Zuber, Mark Siegrist, Silvia Dolder
Publikováno v:
Albano, Giuseppe; Dolder, Silvia; Siegrist, Mark; Mercier-Zuber, Annie; Auberson, Muriel; Stoudmann, Candice; Hofstetter, Wilhelm; Bonny, Olivier; Fuster, Daniel Guido (2017). Increased bone resorption by osteoclast-specific deletion of the sodium/calcium exchanger isoform 1 (NCX1). Pflügers Archiv : European journal of physiology, 469(2), pp. 225-233. Springer 10.1007/s00424-016-1923-5
Calcium is a key component of the bone mineral hydroxyapatite. During osteoclast-mediated bone resorption, hydroxyapatite is dissolved and significant quantities of calcium are released. Several calcium transport systems have previously been identifi
Autor:
Laurent Schild, Ivan Gautschi, Kimmo Kontula, Heidi Fodstad, Muriel Auberson, Heikki Swan, Johannes Loffing
Publikováno v:
Journal of Molecular and Cellular Cardiology. 37:593-602
Mutations of the KCNJ2 gene encoding the potassium channel Kir2.1 were previously shown to cause Andersen's syndrome (AS), a multisystem disease manifesting with developmental abnormalities, cardiac arrhythmias and periodic paralyses. We conducted a
Autor:
Muriel Auberson, Maja B. Hoegg-Beiler, Miguel López de Heredia, Ian J. Orozco, Svea Hohensee, Sònia Sirisi, Thomas J. Jentsch, Virginia Nunes, Raúl Estévez, Clara Vilches, Kathrin Gödde, Isidre Ferrer
Publikováno v:
Recercat. Dipósit de la Recerca de Catalunya
instname
Dipòsit Digital de la UB
Universidad de Barcelona
instname
Dipòsit Digital de la UB
Universidad de Barcelona
Defects in the astrocytic membrane protein MLC1, the adhesion molecule GlialCAM or the chloride channel ClC-2 underlie human leukoencephalopathies. Whereas GlialCAM binds ClC-2 and MLC1, and modifies ClC-2 currents in vitro, no functional connections
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53b9ef7191b28dc40c3ec25de7795c47
http://hdl.handle.net/2445/129370
http://hdl.handle.net/2445/129370
Autor:
Michaela Schweizer, Thomas J. Jentsch, Christian A. Hübner, Hannes Maier, Judith Blanz, Adrian Muenscher, Muriel Auberson
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 27(24)
ClC-2 is a broadly expressed plasma membrane chloride channel that is modulated by voltage, cell swelling, and pH. A human mutation leading to a heterozygous loss of ClC-2 has previously been reported to be associated with epilepsy, whereas the disru