Výsledky vyhledávání - "Muller, D.N."

Enhanced Ca(2+) signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1h(M1560V/+))

Autor: Seidel, E., Schewe, J., Zhang, J., Dinh, H.A., Forslund, S.K., Markó, L., Hellmig, N., Peters, J., Muller, D.N., Lifton, R.P., Nottoli, T., Stölting, G., Scholl, U.I.

Gain-of-function mutations in the CACNA1H gene (encoding the T-type calcium channel Ca(V)3.2) cause autosomal-dominant familial hyperaldosteronism type IV (FH-IV) and early-onset hypertension in humans. We used CRISPR/Cas9 to generate Cacna1h(M1560V/

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::8789a3e2303e671ece26a1ec1921dadc
http://edoc.mdc-berlin.de/20206/1/20206oa.pdf

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The gut microbiome in hypertension: recent advances and future perspectives

Autor: Avery, E.G., Bartolomaeus, H., Maifeld, A., Marko, L., Wiig, H., Wilck, N., Rosshart, S.P., Forslund, S.K., Muller, D.N.

The pathogenesis of hypertension is known to involve a diverse range of contributing factors including genetic, environmental, hormonal, hemodynamic and inflammatory forces, to name a few. There is mounting evidence to suggest that the gut microbiome

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::973f4c86eb9b388a6506a9eb248d979a
http://edoc.mdc-berlin.de/20157/1/20157oa.pdf

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New role for the (pro)renin receptor in T cell development

Autor: Geisberger, S., Maschke, U., Gebhardt, M., Kleinewietfeld, M., Manzel, A., Linker, R.A., Chidgey, A., Dechend, R., Nguyen, G., Daumke, O., Muller, D.N., Wright, M.D., Binger, K.J.

The (pro)renin receptor (PRR) was originally thought to be important for regulating blood pressure via the renin-angiotensin system. However, it is now emerging that PRR has instead a generic role in cellular development. Here, we have specifically d

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::5f11fa496f48ca2f5e87d7f1a4ccb62b
http://edoc.mdc-berlin.de/14864/1/14864oa.pdf

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Skin sodium measured with (23)Na MRI at 7.0 T

Autor: Linz, P., Santoro, D., Renz, W., Rieger, J., Ruehle, A., Ruff, J., Deimling, M., Rakova, N., Muller, D.N., Luft, F.C., Titze, J., Niendorf, T.

Skin sodium (Na(+)) storage, as a physiologically important regulatory mechanism for blood pressure, volume regulation and, indeed, survival, has recently been rediscovered. This has prompted the development of MRI methods to assess Na(+) storage in

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::1874f2777dfb77a831419ec74619d098
http://edoc.mdc-berlin.de/14402/1/14402oa.pdf

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Autophagy and the (pro)renin receptor

Autor: Binger, K.J., Muller, D.N.

The (pro)renin receptor (PRR) is a newly reported member of the renin-angiotensin system (RAS); a hormonal cascade responsible for regulating blood pressure. Originally, identification of PRR was heralded as the next drug target of the RAS, of which

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::460e005b9b227aa4d7e5c79dc4e2d459
http://edoc.mdc-berlin.de/13475/1/13475oa.pdf

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Effect of cytochrome P450-dependent epoxyeicosanoids on Ristocetin-induced thrombocyte aggregation

Autor: Jung, F., Schulz, C., Blaschke, F., Muller, D.N., Mrowietz, C., Franke, R.P., Lendlein, A., Schunck, W.-H.

Publikováno v: Jung, F.; Schulz, C.; Blaschke, F.; Muller, D.N.; Mrowietz, C.; Franke, R.P.; Lendlein, A.; Schunck, W.-H.: Effect of cytochrome P450-dependent epoxyeicosanoids on Ristocetin-induced thrombocyte aggregation. In: Clinical Hemorheology and Microcirculation. Vol. 52 (2012) 2-4, 403-416. (DOI: 10.3233/CH-2012-1614)

Epoxyeicosatrienoic acids (EETs) produced by cytochrome P450 (CYP)-dependent epoxidation of arachidonic acid (AA) inhibit thrombocyte adhesion to the vascular wall. Upon dietary omega-3 fatty acid supplementation, EETs are partially replaced by eicos

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______2110::f8f73e20b2763d7b7e84fdc8888da607
https://publications.hzg.de/id/30346

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Peroxisome proliferator-activated receptor-gamma agonists suppress tissue factor overexpression in rat balloon injury model with Paclitaxel infusion

Autor: Park, J.B., Kim, B.K., Kwon, Y.W., Muller, D.N., Lee, H.C., Youn, S.W., Choi, Y.E., Lee, S.W., Yang, H.M., Cho, H.J., Park, K.W., Kim, H.S.

The role and underlying mechanisms of rosiglitazone, a peroxisome proliferator-activated receptor-gamma (PPAR-{gamma}) agonist, on myocardial infarction are poorly understood. We investigated the effects of this PPAR-{gamma} agonist on the expression

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=mdc______med::3372d5d8e585ea63ee31a05bdd2472a0
http://edoc.mdc-berlin.de/11931/1/11931oa.pdf

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