Zobrazeno 1 - 4
of 4
pro vyhledávání: '"MuGe QiLi"'
Autor:
Xuelian Li, GuoYuan Wang, MuGe QiLi, HaiHai Liang, TianShi Li, XiaoQiang E, Ying Feng, Ying Zhang, Xiao Liu, Ming Qian, BoZhi Xu, ZhiHang Shen, Samuel Chege Gitau, DanDan Zhao, HongLi Shan
Publikováno v:
Cellular Physiology and Biochemistry, Vol 45, Iss 5, Pp 1955-1965 (2018)
Background/Aims: Cardiac interstitial fibrosis is an abnormality of various cardiovascular diseases, including myocardial infarction, hypertrophy, and atrial fibrillation, and it can ultimately lead to heart failure. However, there is a lack of pract
Externí odkaz:
https://doaj.org/article/99fd1cc09654449d97e827b7bd412dda
Autor:
Ming Qian, Ying Feng, Muge Qili, Hongli Shan, Bozhi Xu, Yuhong Zhou, Nan Zheng, Zhenwei Pan, Baofeng Yang, Ying Zhang, Dandan Zhao, Xuelian Li, Haihai Liang, Xiao Liu, Qiuxia Wu
Publikováno v:
International Journal of Cardiology. 272:298-305
Background Sorting nexin 17 (SNX17) is a critical cytoplasmic adaptor protein that regulates endosomal trafficking of membrane proteins to determine their recycling and/or degradation. The potential role of SNX17 in cardiovascular pathophysiology has
Autor:
Tianshi Li, Hongli Shan, Dandan Zhao, Ming Qian, Ying Feng, Samuel Chege Gitau, Xiao Liu, Ying Zhang, Muge Qili, Xuelian Li, Haihai Liang, ZhiHang Shen, Guoyuan Wang, Xiaoqiang E, Bozhi Xu
Publikováno v:
Cellular Physiology and Biochemistry, Vol 45, Iss 5, Pp 1955-1965 (2018)
Background/Aims: Cardiac interstitial fibrosis is an abnormality of various cardiovascular diseases, including myocardial infarction, hypertrophy, and atrial fibrillation, and it can ultimately lead to heart failure. However, there is a lack of pract
Autor:
Dandan Zhao, Qiuxia Wu, Lu Wang, Zhenfeng Guo, Haihai Liang, Hongli Shan, Ming Qian, Tianshi Li, Chaoqian Xu, Bozhi Xu, Xuelian Li, Muge Qili, Longtao Huangfu, Xiaoqiang E, Samuel Chege Gitau
Publikováno v:
Scientific Reports
Although increases in cardiovascular load (pressure overload) are known to elicit ventricular remodeling including cardiomyocyte hypertrophy and interstitial fibrosis, the molecular mechanisms of pressure overload or AngII -induced cardiac interstiti