Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Mourad Matmati"'
Publikováno v:
Annals of Biomedical Engineering. 41:894-903
Cartilage defects can be addressed with replacement strategies such as autologous chondrocyte implantation (ACI). Expansion of autologous chondrocytes in vitro is an essential step to obtain the necessary cell numbers required for ACI. A major proble
Publikováno v:
McGill Science Undergraduate Research Journal. 7:42-48
Introduction: autologous chondrocyte implantation (aci) is one successful treatment for osteoarthritic defects (1-4). However, chondrocytes undergo a conversion to a fibroblastic phenotype during their expansion in vitro. This process can lead to deg
Autor:
Jörg Hamann, Mark J. Kwakkenbos, Else N. Kop, Martin Stacey, Hsi-Hsien Lin, Siamon Gordon, Mourad Matmati
Publikováno v:
Immunogenetics. 55(10):655-666
With the human and mouse genome projects now completed, the receptor repertoire of mammalian cells has finally been elucidated. The EGF-TM7 receptors are a family of class B seven-span transmembrane (TM7) receptors predominantly expressed by cells of
Autor:
Mark J. Kwakkenbos, Mourad Matmati, Ole Madsen, Walter Pouwels, YongYi Wang, Ronald E. Bontrop, Peter J. Heidt, Robert M. Hoek, Jörg Hamann
Publikováno v:
FASEB journal, 20(14), 2582-2584. FASEB
The epidermal growth factor (EGF)-TM7 receptors CD97, EMR1, EMR2, EMR3, and EMR4 form a group of adhesion class heptahelical molecules predominantly expressed by cells of the immune system. These receptors bind cellular ligands through EGF-like domai
Autor:
Derek H. Rosenzweig, Mourad Matmati, Sidharth Chaudhry, Thomas M. Quinn, Boris Hinz, Ghazaleh Khayat
Expansion of autologous chondrocytes in vitro is used to generate adequate populations for cell-based therapies. However, standard (SD) culture methods cause loss of chondrocyte phenotype and dedifferentiation to fibroblast-like cells. Here, we use a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6381a209b311b925a6a98974c49ee65c
https://europepmc.org/articles/PMC3501116/
https://europepmc.org/articles/PMC3501116/
Autor:
Lars Vereecke, Dirk Elewaut, Lies Geboes, Patrick Matthys, Conor Mc Guire, Louis Boon, Peggy Jacques, Mirjam Kool, Eveline Verheugen, Geert van Loo, Rudi Beyaert, Yuanyuan Chu, Els Louagie, Steven Staelens, Manolis Pasparakis, Jonathan Maelfait, Mourad Matmati, Marc Schmidt-Supprian, Mozes Sze, Bart N. Lambrecht
Publikováno v:
Nature genetics
A20 (TNFAIP3) is a protein that is involved in the negative feedback regulation of NF-κB signaling in response to specific proinflammatory stimuli in different cell types and has been suggested as a susceptibility gene for rheumatoid arthritis. To d
Publikováno v:
Immunology Letters, 123, 160-8
Immunology Letters, 123, 2, pp. 160-8
Immunology letters, 123(2), 160-168. Elsevier
Immunology Letters, 123, 2, pp. 160-8
Immunology letters, 123(2), 160-168. Elsevier
Contains fulltext : 80460.pdf (Publisher’s version ) (Closed access) CD97 is a large heptahelical EGF-TM7 receptor broadly expressed on hematopoietic cells as three isoforms with respectively three, four, or five epidermal growth factor (EGF)-like
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3fdb7fed8a0547d02731499d17b2b43e
http://hdl.handle.net/2066/80460
http://hdl.handle.net/2066/80460
Autor:
Paul A. Baars, Mireille T. M. Vossen, Mourad Matmati, René A. W. van Lier, Taco W. Kuijpers, Mi-Ran Gent, Kirsten M. L. Hertoghs, Jörg Hamann, Georges Leclercq
Publikováno v:
Journal of immunology (Baltimore, Md., 180(6), 3739-3745. American Association of Immunologists
The absence of the TNF-receptor family member CD27 marks the stable acquisition of cytolytic effector functions by both CD4+ and CD8+ T cells. We found that the majority of circulating human NK cells was CD27−. These cells were largely CD56dim, con
Autor:
Arthur J. Verhoeven, Machiel H. Jansen, Mourad Matmati, Walter Pouwels, Jörg Hamann, Robert M. Hoek, Robin van Bruggen
Publikováno v:
Journal of leukocyte biology, 81(2), 440-448. FASEB
EMR3 is a member of the epidermal growth factor-seven-transmembrane (EGF-TM7) family of adhesion class TM7 receptors. This family also comprises CD97, EMR1, EMR2, and EMR4. To characterize human EMR3 at the protein level, we generated Armenian hamste
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::858f266f5f768b07e089e8909c0d14f9
https://pure.amc.nl/en/publications/the-human-egftm7-receptor-emr3-is-a-marker-for-mature-granulocytes(5d72f495-b462-4046-a64a-a060e48e6d56).html
https://pure.amc.nl/en/publications/the-human-egftm7-receptor-emr3-is-a-marker-for-mature-granulocytes(5d72f495-b462-4046-a64a-a060e48e6d56).html
Autor:
Mark J. Kwakkenbos, Hsi-Hsien Lin, René A. W. van Lier, Siamon Gordon, Walter Pouwels, Martin Stacey, Jörg Hamann, Mourad Matmati
Publikováno v:
Journal of leukocyte biology, 77(1), 112-119. FASEB
The EGF-TM7 receptors CD97 and EMR2 are heptahelical molecules predominantly expressed on leukocytes. A characteristic of these receptors is their ability to interact with cellular ligands via the N-terminal epidermal growth factor (EGF)-like domains