Výsledky vyhledávání - "Mounira Djerbi"

Glycogen synthase kinase-3α/β inhibition promotes in vivo amplification of endogenous mesenchymal progenitors with osteogenic and adipogenic potential and their differentiation to the osteogenic lineage

Autor: Chandan K. Nagaraju, Patrick J O'Shea, James Pilling, Alessandra Gambardella, Peter I. Croucher, Sindhu T. Mohanty, Michael Sullivan, Lucksy Kottam, Mounira Djerbi, Witte Koopmann, Ilaria Bellantuono

Publikováno v: Journal of Bone and Mineral Research. 26:811-821

Small molecules are attractive therapeutics to amplify and direct differentiation of stem cells. They also can be used to understand the regulation of their fate by interfering with specific signaling pathways. Mesenchymal stem cells (MSCs) have the

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f1cb9f80aa735c18697524f5875eea8b
https://doi.org/10.1002/jbmr.266

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Simultaneous Expression and Detection of Multiple Retroviral Constructs in Haematopoietic Cells After Bone Marrow Transplantation

Autor: Mounira Djerbi, Alf Grandien, A. M. Nyakeriga, M. M. Malinowski

Publikováno v: Scandinavian Journal of Immunology. 61:545-550

Due to the complexity and redundancy of molecular processes governing the development and function of haematopoietic cells, experimental procedures allowing simultaneous alteration in gene expression of multiple genes in vivo are needed. Here, we des

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4e39754ea8ddc9aa4d7a948a28467f5c
https://doi.org/10.1111/j.1365-3083.2005.01607.x

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Expression of the Long Form of Human FLIP by Retroviral Gene Transfer of Hemopoietic Stem Cells Exacerbates Experimental Autoimmune Encephalomyelitis

Autor: Mounira Djerbi, Alf Grandien, Tomas Olsson, Manuchehr Abedi-Valugerdi, Robert A. Harris, Khairul-Bariah Abdul-Majid

Publikováno v: Karolinska Institutet

Subsidence of inflammation and clinical recovery in experimental autoimmune encephalomyelitis (EAE) is postulated to involve apoptosis of inflammatory cells. To test this concept, we examined the effects of overexpressing the long form of human FLICE

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::807b4c0d219df3c2ad798ae38d4232b1
https://doi.org/10.4049/jimmunol.170.4.2064

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Characterization of the Human FLICE-Inhibitory Protein Locus and Comparison of the Anti-Apoptotic Activity of Four Different FLIP Isoforms

Autor: Mounira Djerbi, Taher Darreh-Shori, Boris Zhivotovsky, Alf Grandien

Publikováno v: Scandinavian Journal of Immunology. 54:180-189

Death receptor-mediated apoptosis is involved in the regulation of immune responses and in the maintenance of immunological tolerance. FLICE-inhibitory proteins (FLIPs) are important modulators of death receptor-mediated apoptosis. To date, the FLIP

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::b12a05d38d33167972ffed4b9378e157
https://doi.org/10.1046/j.1365-3083.2001.00941.x

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The Inhibitor of Death Receptor Signaling, Flice-Inhibitory Protein Defines a New Class of Tumor Progression Factors

Autor: Valentina Screpanti, Mounira Djerbi, Bjarne Bogen, Alf Grandien, Peter Biberfeld, Anca I. Catrina

Publikováno v: The Journal of Experimental Medicine

Death receptor–mediated apoptosis can be modulated by several antiapoptotic proteins, such as the FLICE (FADD [Fas-associated death domain]-like IL-1β–converting enzyme)-inhibitory proteins (FLIPs). The FLIP family includes both cellular and vir

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b8c00630b2e4752906457dc781487194
https://doi.org/10.1084/jem.190.7.1025

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Participation of FLIP, RIP and Bcl-x(L) in Fas-mediated T-cell death

Autor: Mounira Djerbi, Alf Grandien, Boris Zhivotovsky, M. M. Malinowski, Hideo Yagita

Publikováno v: Scandinavian journal of immunology. 66(4)

Apart from the conventional Fas signalling pathway, alternative pathways including the mitochondrial caspase-dependent and RIP-mediated cell death routes have been proposed to operate during Fas-mediated cell death. To evaluate the contribution of di

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a9bc12382aba39ddba35d6d740c5220
https://pubmed.ncbi.nlm.nih.gov/17850585

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Exposure to mercuric chloride during the induction phase and after the onset of collagen-induced arthritis enhances immune/autoimmune responses and exacerbates the disease in DBA/1 mice

Autor: Joseph W. DePierre, Håkan Mellstedt, Manuchehr Abedi-Valugerdi, Farhad Gharibdoost, Monika Hansson, Mounira Djerbi, Moustapha Hassan, Hodjattallah Rabbani

Publikováno v: Immunology. 114(3)

In susceptible mice, mercuric chloride induces a systemic autoimmune response that is characterized by elevated serum levels of immunoglobulin G1 (IgG1) and immunoglobulin E (IgE), production of anti-nucleolar antibodies (ANolAs) and the formation of

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c730851eaabee0db5ce79778c37f225f
https://pubmed.ncbi.nlm.nih.gov/15720444

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