Výsledky vyhledávání - "Mouchet, Nicolas"

Well-differentiated liver cancers reveal the potential link between ACE2 dysfunction and metabolic breakdown

Autor: Desquilles, Lise, Cano, Luis, Ghukasyan, Gevorg, Mouchet, Nicolas, Landreau, Clemence, Corlu, Anne, Clement, Bruno, Turlin, Bruno, Desert, Romain, Musso, Orlando

Publikováno v: Scientific Reports
Scientific Reports, 2022, 12 (1), pp.1859. ⟨10.1038/s41598-021-03710-0⟩
Scientific Reports, Vol 12, Iss 1, Pp 1-11 (2022)

International audience; Angiotensin-converting enzyme 2 (ACE2) is the receptor of the Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) causing Coronavirus disease 2019 (COVID-19). Transmembrane serine protease 2 (TMPRSS2) is a coreceptor.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::15a29d54228d1082b96f9db5af83b399
http://europepmc.org/articles/PMC8814043

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HLA-G Is Widely Expressed by Mast Cells in Regions of Organ Fibrosis in the Liver, Lung and Kidney

Autor: Mouchet, Nicolas, Vu, Nicolas, Turlin, Bruno, Rioux-Leclercq, Nathalie, Jouneau, Stéphane, Samson, Michel, Amiot, Laurence

Publikováno v: International Journal of Molecular Sciences
International Journal of Molecular Sciences, 2021, 22 (22), pp.12490. ⟨10.3390/ijms222212490⟩
Volume 22
Issue 22
International Journal of Molecular Sciences, MDPI, 2021, 22 (22), pp.12490. ⟨10.3390/ijms222212490⟩
International Journal of Molecular Sciences, Vol 22, Iss 12490, p 12490 (2021)

We previously demonstrated that mast cells expressing HLA-G are associated with regions of hepatitis C virus-induced liver fibrosis. Here, we aimed to determine whether HLA-G expression in mast cells is specific to viral etiology, the liver, or to th

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::e1e8f523a256bf4229318be6e4711c85
https://pubmed.ncbi.nlm.nih.gov/34830373

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USF1 defect drives p53 degradation during Helicobacter pylori infection and accelerates gastric carcinogenesis

Autor: Costa, Lionel, Corre, Sébastien, Michel, Valérie, Le Luel, Krysten, Fernandes, Julien, Ziveri, Jason, Jouvion, Grégory, Danckaert, Anne, Mouchet, Nicolas, da Silva Barreira, David, Torres, Javier, Camorlinga, Margarita, d'Elios, Mario Milco, Fiette, Laurence, de Reuse, Hilde, Galibert, Marie-Dominique, Touati, Eliette

Publikováno v: Gut
Gut, 2020, 69 (9), pp.1582-1591. ⟨10.1136/gutjnl-2019-318640⟩
Gut, BMJ Publishing Group, 2020, 69 (9), pp.1582-1591. ⟨10.1136/gutjnl-2019-318640⟩

International audience; Objective - Design - Human gastric epithelial cell lines were infected with 7.13, exposed or not to a DNA-damaging agent camptothecin (CPT), to mimic a genetic instability context. We quantified the expression of , and their t

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::f3595f54fee4f1e4a2af5e181612773f
https://hal-pasteur.archives-ouvertes.fr/pasteur-02418249

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Sustained activation of the Aryl hydrocarbon Receptor transcription factor promotes resistance to BRAF-inhibitors in melanoma

Autor: Corre, Sébastien, Tardif, Nina, Mouchet, Nicolas, Leclair, Héloïse M., Boussemart, Lise, Gautron, Arthur, Bachelot, Laura, Perrot, Anthony, Soshilov, Anatoly, Rogiers, Aljosja, Rambow, Florian, Dumontet, Erwan, Tarte, Karin, Bessede, Alban, Guillemin, Gilles J., Marine, Jean-Christophe, Denison, Michael S., Gilot, David, Galibert, Marie-Dominique

Publikováno v: Nature Communications, Vol 9, Iss 1, Pp 1-13 (2018)
Nature Communications
Nature Communications, 2018, 9 (1), pp.4775. ⟨10.1038/s41467-018-06951-2⟩
Corre, S; Tardif, N; Mouchet, N; Leclair, HM; Boussemart, L; Gautron, A; et al.(2018). Sustained activation of the Aryl hydrocarbon Receptor transcription factor promotes resistance to BRAF-inhibitors in melanoma. Nature Communications, 9(1), 4775. doi: 10.1038/s41467-018-06951-2. UC Office of the President: Research Grants Program Office (RGPO). Retrieved from: http://www.escholarship.org/uc/item/1jf4j4r5
Nature communications, vol 9, iss 1
Nature Communications, Nature Publishing Group, 2018, 9 (1), pp.4775. ⟨10.1038/s41467-018-06951-2⟩

BRAF inhibitors target the BRAF-V600E/K mutated kinase, the driver mutation found in 50% of cutaneous melanoma. They give unprecedented anti-tumor responses but acquisition of resistance ultimately limits their clinical benefit. The master regulators

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::d5841be9aefb4fd97a9496ed11115dc8
http://link.springer.com/article/10.1038/s41467-018-06951-2

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