Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Motohiro Kamimura"'
Autor:
Hugo A. Katus, Alexander H. Dalpke, Erwin Blessing, Florian Bea, Christiane Viedt, Nigel Mackman, Motohiro Kamimura, Michael R. Preusch, Christian Weber, Michael E. Rosenfeld, David M. Cohen, Jörg Kreuzer
Publikováno v:
Circulation Research. 97:305-313
Atherosclerosis is considered to be an inflammatory disease. Tissue factor (TF), a prothrombotic molecule expressed by various cell types within atherosclerotic plaques, is thought to play an essential role in thrombus formation after atherosclerotic
Autor:
Hugo A. Katus, Christiane Viedt, Motohiro Kamimura, Jörg Kreuzer, Daniel Teupser, Ralf P. Brandes, H. I. Krieger-Brauer, Jianwei Fei
Publikováno v:
Journal of Molecular Medicine. 82:31-38
Reactive oxygen species (ROS) are involved in the transcriptional response to angiotensin (ANG) II. In this setting the role of NAD(P)H oxidase, an important source of ROS as second messengers, is not completely understood. In particular in human cel
Autor:
Suhail Ahmad, Minoru Oda, Hirotsugu Kobata, Masami Shimazaki, Takaya Abe, Hirohito Hasegawa, Kazuhiro Uchita, Motohiro Kamimura, Masaaki Fukunishi, Tomiya Abe, Tadao Akizawa, Hikari Orita
Publikováno v:
Kidney International. 64:1522-1528
Effect of β2-microglobulin adsorption column on dialysis-related amyloidosis.Backgroundβ2-microglobulin (β2-m) is considered a major pathogenic factor in dialysis-related amyloidosis (DRA), often seen in long-term dialysis patients. No effective t
Autor:
Hiroaki Ogata, Eriko Kinugasa, Shinji Sumikado, Tadao Akizawa, Shigeo Negi, Masahide Mizobuchi, Toshibumi Sakaguchi, Motohiro Kamimura, Kazuhiro Shiizaki
Publikováno v:
Nephrology. 8:S53-S57
SUMMARY: Hyperphosphatemia, vitamin D deficiency, and resulted hypocalcemia have been regarded as classical pathogeneses of secondary hyperparathyroidism. These factors have been treated by the administration of phosphorus binder and vitamin D deriva
Autor:
Karue Kimura, Masaki Matsuo, Tomiya Abe, Motohiro Kamimura, Tadao Akizawa, Tsuyoshi Nakaya, Takashi Miyahara, Kimiyo Matsumoto
Publikováno v:
Therapeutic Apheresis. 6:467-470
Peripheral arterial disease (PAD; arteriosclerosis obliterans) shows ischemic symptoms along the peripheral arteries due to reduced blood flow, and the number of patients with PAD is increasing. Several papers have reported on the clinical effect of
Autor:
Motohiro Kamimura, Stephan R Orth
Publikováno v:
Journal of Hypertension. 21:269-271
Autor:
Nobuhiko Narukawa, Hikari Orita, Motohiro Kamimura, Takaya Abe, Hirotsugu Kobata, Kazuhiro Shiizaki, Tadao Akizawa, Yukiko Kitabata
Publikováno v:
Therapeutic apheresis and dialysis : official peer-reviewed journal of the International Society for Apheresis, the Japanese Society for Apheresis, the Japanese Society for Dialysis Therapy. 9(1)
A 71-year-old woman was admitted to the Wakayama Medical University Hospital with dizziness and loss of body balance. She had started hemodialysis at the age of 70. During the 33 days before admission, she received oral tizanidine hydrochloride at 3
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 44(6)
Activation of vascular smooth muscle cells (SMCs) by platelet-derived growth factor (PDGF) is a seminal event in the initiation and progression of the atherosclerotic lesion and may contribute to atherosclerotic plaque instability with plaque rupture
Autor:
Tadao, Akizawa, Motohiro, Kamimura, Masahide, Mizobuchi, Kazuhiro, Shiizaki, Shinji, Sumikado, Toshibumi, Sakaguchi, Shigeo, Negi, Hiroaki, Ogata, Eriko, Kinugasa
Publikováno v:
Nephrology (Carlton, Vic.).
Hyperphosphatemia, vitamin D deficiency, and resulted hypocalcemia have been regarded as classical pathogeneses of secondary hyperparathyroidism. These factors have been treated by the administration of phosphorus binder and vitamin D derivatives. Ho
Autor:
Christiane Viedt, Jianwei Fei, Hugo A. Katus, Gertrud Maria Hänsch, Weili Shen, Jörg Kreuzer, Motohiro Kamimura
Publikováno v:
Basic research in cardiology. 98(6)
The terminal complement complex C5b-9 is known to participate in inflammatory processes including atherosclerosis. Inflammation appears to be a direct consequence of C5b-9-mediated cell stimulation. 3-Hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) red