Zobrazeno 1 - 10
of 45
pro vyhledávání: '"Morten G. Ræder"'
Autor:
Geir Christensen, Ulla H. Enger, William E. Louch, Kristin B. Andersson, Xiu Hua Liu, Morten G. Ræder, Zhen Ying Zhang, Cathrine Husberg
Publikováno v:
Cell Calcium. 49:201-207
Reduced sarco(endo)plasmic reticulum (SR) Ca(2+) ATPase (SERCA2) contributes to the impaired cardiomyocyte Ca(2+) homeostasis observed in heart failure. We hypothesized that a reduction in SERCA2 also elicits myocardial ER/SR stress responses, includ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9228a68e7e96007c280260f95bcba73d
https://doi.org/10.1016/j.ceca.2010.09.009
https://doi.org/10.1016/j.ceca.2010.09.009
Autor:
Kristin Eiklid, Knut Erik Berge, Olaug K. Rødningen, Morten G. Ræder, Knut Jørgen Labori, Sigve Nakken, Karl Esten Nakken
Publikováno v:
Liver International. 29:743-747
Background and Aims: Mutations in the gene encoding the ABCB4 [adenosine triphosphate (ATP)-binding cassette, sub-family B (MDR/TAP), member 4] transporter lower phosphatidylcholine output into bile and contribute to cholesterol gallstone formation b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26ad809fec093ac048589761fb2a5434
https://doi.org/10.1111/j.1478-3231.2008.01914.x
https://doi.org/10.1111/j.1478-3231.2008.01914.x
Autor:
Knut Erik Berge, Ståle Nygård, Karl Esten Nakken, Annlaug Ødegaard, Terese Haaland, Morten G. Ræder, Knut Jørgen Labori
Publikováno v:
Scandinavian Journal of Gastroenterology. 44:211-218
Abcb4 (-/-) mice secrete phosphatidylcholine-deficient bile and develop sclerosing cholangitis (SC), a condition that involves differential hepatic transcription of genes governing inflammation, tissue remodelling and fibrosis. The objective of this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d0cd65b75b4f1eb174b484889ba1c0d
https://doi.org/10.1080/00365520802400867
https://doi.org/10.1080/00365520802400867
Autor:
Annlaug Ødegaard, K. Arnkværn, Terese Haaland, Morten G. Ræder, Knut Jørgen Labori, Knut Erik Berge, Ståle Nygård, Karl Esten Nakken
Publikováno v:
Scandinavian Journal of Gastroenterology. 42:1245-1255
Abcb4 (-/-) mice secrete phosphatidylcholine-free, cytotoxic bile and develop chronic cholangitis. The aim of this study was to identify differentially transcribed genes whose products contribute to the liver tissue pathology during this disease.Hepa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2809dae885563ac03de239cfa98de4fd
https://doi.org/10.1080/00365520701320521
https://doi.org/10.1080/00365520701320521
Autor:
Morten G. Ræder, Knut Jørgen Labori
Publikováno v:
Scandinavian Journal of Surgery. 93:176-183
Background and Aims: Jaundice in trauma patients may reflect serious underlying pathology. The aim of this review was to determine the appropriate diagnostic approach to the patient with jaundice following trauma. Methods: A MEDLINE search was perfor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23d193309da44bdbb1883acd9c0aa2ce
https://doi.org/10.1177/145749690409300302
https://doi.org/10.1177/145749690409300302
Autor:
B. Woldseth, T. Christensen, E. Hvattum, Bjørn Atle Bjørnbeth, Torstein Lyberg, Morten G. Ræder, Knut Jørgen Labori
Publikováno v:
Scandinavian Journal of Gastroenterology. 34:1042-1049
Infusion of large intravenous bilirubin loads in bile acid-depleted pigs reduces P-glycoprotein-dependent biliary phospholipid secretion and increases the cytotoxicity of bile. The reasons for the diminution of biliary phospholipid secretion and the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ed9015131fe35e7ade92c94d74023c3
https://doi.org/10.1080/003655299750025165
https://doi.org/10.1080/003655299750025165
Publikováno v:
Scandinavian Journal of Gastroenterology. 33:201-208
Large intravenous bilirubin loads cause loss of hepatic canalicular membrane microvilli and cholestasis. This study examines whether these untoward effects might be due to canalicular membrane injury from cytotoxic bile.The cytotoxicity of bile was a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ea1868bab6d824c7cfd121b3a0e2b900
https://doi.org/10.1080/00365529850166950
https://doi.org/10.1080/00365529850166950
Publikováno v:
Gastroenterology. 108:850-859
Background/Aims: Secretin stimulates pancreatic ductules to secrete HCO3− into pancreatic juice and H+ into interstitial fluid. The aim of the present study was first to examine whether ductular H+ secretion is inhibited by micromolar concentration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9bd1c47729bde5f88f824e29f377f07f
https://doi.org/10.1016/0016-5085(95)90460-3
https://doi.org/10.1016/0016-5085(95)90460-3
Publikováno v:
Scandinavian Journal of Gastroenterology. 30:1186-1193
This study was undertaken to elucidate why large bilirubin loads cause canalicular cholestasis and whether bile acid infusions protect against bilirubin-induced cholestasis.The effects of bilirubin infusion on canalicular bile secretion and canalicul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f588b2fbc64176a0d4d40a40a64922b3
https://doi.org/10.3109/00365529509101629
https://doi.org/10.3109/00365529509101629
Autor:
Morten G. Ræder
Publikováno v:
Gastroenterology. 103:1674-1684
In recent years, there has been a rapid growth in knowledge about the subcellular mechanisms involved in pancreatic ductal secretion of bicarbonate. The mechanisms governing anion transport across the luminal membrane of duct cells have been well cha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ce92decda70965a56a2d8c618c5d5822
https://doi.org/10.1016/0016-5085(92)91195-a
https://doi.org/10.1016/0016-5085(92)91195-a