Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Moritz Przybilla"'
Autor:
Vítor Teixeira, Kate Davies, Peter Campbell, Bart Vanhaesebroeck, Sam Janes, Kate Gowers, Amalie Kai Bentzen, Teerapon Sahwangarrom, Claudia Peinador Marin, Sam Gamble, Zoe Whiteman, Seng Ung, Lukas Kalinke, Ahmed Alhendi, Adam Pennycuick, Sandra Gomez Lopez, Andrei Enica, Amber Rogers, Kane Foster, Moritz Przybilla, James L Reading
Publikováno v:
Journal for ImmunoTherapy of Cancer, Vol 11, Iss Suppl 1 (2023)
Externí odkaz:
https://doaj.org/article/009acdb0f46049eeb93fb60f0226f250
Autor:
Moritz Przybilla, Simon Steiger, Alexandra M Poos, Isabelle Lander, Stefanie Huhn, Niels Weinhold, Oliver Stegle, Stephan M Tirier, Karsten Rippe, Nicola Giesen, Hartmut Goldschmidt, Carsten Müller-Tidow, Lukas John, Anna Luise Grab, Jan-Philipp Malllm, Marc S. Raab, Nina Prokoph, Katharina Bauer, Anja Baumann
Publikováno v:
Clinical Lymphoma Myeloma and Leukemia. 21:S5
Introduction Despite new treatment approaches, in most patients with multiple myeloma (MM) resistant subclones expand, finally resulting in the development of relapsed/refractory disease (RRMM). Recent single-cell transcriptomic (scRNA-seq) studies h
Autor:
Anja Baumann, Oliver Stegle, Hartmut Goldschmidt, Nicola Casiraghi, Niels Weinhold, Katharina Bauer, Carsten Mueller-Tidow, Lukas John, Hana Susak, Stephan M Tirier, Jan-Philipp Mallm, Moritz Przybilla, Benedikt Brors, Karsten Rippe, Isabelle Lander, Marc S. Raab, Alexandra M Poos, Nicola Giesen, Anja Kunze, Jing Xu
Publikováno v:
Blood. 134:575-575
Introduction: Multiple myeloma (MM) is a heterogeneous malignancy of clonal plasma cells that accumulate in the bone marrow (BM). Despite new treatment approaches, in most patients resistant subclones are selected by therapy, resulting in the develop
Autor:
Johannes Gebert, Elisabeth Pfaffendorf, Jukka-Pekka Mecklin, Simon Kalteis, Moritz Przybilla, Julia Krzykalla, Florian Seidler, Sanne W. ten Broeke, Markus Draxlbauer, Daniel Heid, Aysel Ahadova, Sarah Schott, Axel Benner, Hendrik Bläker, Maria Bonsack, Maartje Nielsen, Michael Jendrusch, Magnus von Knebel Doeberitz, Sonja Krausert, Alexej Ballhausen, Angelika B. Riemer, Matthias Kloor, Toni T. Seppälä
Publikováno v:
Cancer Research. 79:571-571
The immune system can recognize and attack cancer cells and their precursors, especially those with a high load of mutation-induced neoantigens. Such neoantigens are particularly abundant in DNA mismatch repair (MMR)-deficient cancers. MMR deficiency