Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Moritz Osterholt"'
Autor:
Martin Braun, Niko Hettinger, Christoph Koentges, Katharina Pfeil, Maria C Cimolai, Michael M Hoffmann, Moritz Osterholt, Torsten Doenst, Christoph Bode, Heiko Bugger
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0119416 (2015)
Adiponectin deficiency leads to increased myocardial infarct size following ischemia reperfusion and to exaggerated cardiac hypertrophy following pressure overload, entities that are causally linked to mitochondrial dysfunction. In skeletal muscle, l
Externí odkaz:
https://doaj.org/article/1fa183b82a0e46ed9b2d41a20cee6ad2
Publikováno v:
The Thoracic and Cardiovascular Surgeon. 65:S1-S110
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cc809b9da57f3d6ff90f56d1a5a17285
https://doi.org/10.1055/s-0037-1598722
https://doi.org/10.1055/s-0037-1598722
Autor:
Andrea Schrepper, Michael Schwarzer, Moritz Osterholt, Torsten Doenst, Anne Lunkenbein, Paulo A. Amorim
Publikováno v:
The Journal of Physiology. 592:3767-3782
We investigated the impact of cardiac reactive oxygen species (ROS) during the development of pressure overload-induced heart failure. We used our previously described rat model where transverse aortic constriction (TAC) induces compensated hypertrop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::92a06b822bf8d667f764ed9904180a1c
https://doi.org/10.1113/jphysiol.2014.274704
https://doi.org/10.1113/jphysiol.2014.274704
Autor:
Christophe Beauloye, Jessica M. Berthiaume, Luc Bertrand, David I. Brown, Torsten Doenst, Jan F.C. Glatz, Louis Hue, Peter J. Kennel, Terje S. Larsen, Craig A. Lygate, Miranda Nabben, Tien Dung Nguyen, Bernd Niemann, Moritz Osterholt, Linda R. Peterson, Susanne Rohrbach, Andrea Schrepper, Paul Christian Schulze, Michael Schwarzer, Marc van Bilsen, Christina Werner, Monte S. Willis, Martin E. Young
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::90c87ef115369093cca5c0c78b88ea12
https://doi.org/10.1016/b978-0-12-802394-5.00020-0
https://doi.org/10.1016/b978-0-12-802394-5.00020-0
Cardiac metabolism encompasses all biochemical processes that result in the conversion of substrates or intermediates of metabolic pathways and cycles for the purpose of cell function, growth, and contraction. Methods investigating metabolism must th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::741318437df5c4225952afa01fb678f6
https://doi.org/10.1016/b978-0-12-802394-5.00007-8
https://doi.org/10.1016/b978-0-12-802394-5.00007-8
Publikováno v:
Heart Failure Reviews. 18:645-656
Normal cardiac function requires high and continuous supply with ATP. As mitochondria are the major source of ATP production, it is apparent that mitochondrial function and cardiac function need to be closely related to each other. When subjected to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cc04b6a40b8282f1e91dfae5fe4bce60
https://doi.org/10.1007/s10741-012-9346-7
https://doi.org/10.1007/s10741-012-9346-7
Autor:
Katharina Pfeil, Sophia Kersting, Christoph Bode, Maximilian E. Hölscher, Tilman Schnick, Michael M. Hoffmann, Lutz Hein, Adam R. Wende, Maria C. Cimolai, Torsten Doenst, Christoph Koentges, Moritz Osterholt, Alexandra König, Katja E. Odening, Heiko Bugger, Andrea Schrepper, Judith Asal, E. Dale Abel
Publikováno v:
Basic Research in Cardiology. 110
Hypoadiponectinemia is an independent predictor of cardiovascular disease, impairs mitochondrial function in skeletal muscle, and has been linked to the pathogenesis of Type 2 diabetes. In models of Type 2 diabetes, myocardial mitochondrial function
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cf58680ca0e0b6035d87976df5b5e9f1
https://doi.org/10.1007/s00395-015-0495-4
https://doi.org/10.1007/s00395-015-0495-4
Autor:
Niko Hettinger, Maria C. Cimolai, Christoph Koentges, Heiko Bugger, Moritz Osterholt, Torsten Doenst, Michael M. Hoffmann, Katharina Pfeil, Martin Braun, Christoph Bode
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 3, p e0119416 (2015)
PLoS ONE, Vol 10, Iss 3, p e0119416 (2015)
Adiponectin deficiency leads to increased myocardial infarct size following ischemia reperfusion and to exaggerated cardiac hypertrophy following pressure overload, entities that are causally linked to mitochondrial dysfunction. In skeletal muscle, l
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e9fb548bc6cb0a5f45d3bfcea0ad40c7
http://europepmc.org/articles/PMC4364743
http://europepmc.org/articles/PMC4364743
Autor:
Michael, Schwarzer, Moritz, Osterholt, Anne, Lunkenbein, Andrea, Schrepper, Paulo, Amorim, Torsten, Doenst
Publikováno v:
The Journal of physiology. 592(17)
We investigated the impact of cardiac reactive oxygen species (ROS) during the development of pressure overload-induced heart failure. We used our previously described rat model where transverse aortic constriction (TAC) induces compensated hypertrop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::cab6ddd59dbde4ca7fb2c1cfb2e18ce7
https://pubmed.ncbi.nlm.nih.gov/24951621
https://pubmed.ncbi.nlm.nih.gov/24951621
Autor:
Yasushige Shingu, Paulo A. Amorim, Moritz Osterholt, TD Nguyen, Michael Schwarzer, Torsten Doenst
Publikováno v:
The Thoracic and cardiovascular surgeon. 61(8)
Background There is currently no standard for the assessment of contractile function in animals. We aimed to determine whether transthoracic echocardiography in rats with chronic pressure overload allows determining the stage of hypertrophy and heart
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f27be1eb94e08fcf1c231a4055f96099
https://pubmed.ncbi.nlm.nih.gov/23344767
https://pubmed.ncbi.nlm.nih.gov/23344767