Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Monica Ropolo"'
Autor:
Enrico Cappelli, Fotios Kalfas, Paolo Severi, Donatella Vecchio, Valeria Capra, Alessandro Poggi, Alessandro Raso, Roberto Biassoni, Paolo Nozza, Guido Frosina, Samantha Mascelli, Monica Ropolo
Publikováno v:
Brain Pathology. 22:677-688
Previous studies have shown that tumor-driving glioma stem cells (GSC) may promote radio-resistance by constitutive activation of the DNA damage response started by the ataxia telangiectasia mutated (ATM) protein. We have investigated whether GSC may
Autor:
Paolo Degan, Gianluca Damonte, Alessandro Poggi, Monica Ropolo, Mara Foresta, Guido Frosina, Yoke W. Kow, Ilaria Pettinati
Publikováno v:
Free Radical Biology and Medicine. 48:681-690
Repair of the oxidized purine 8-oxo-7,8-dihydro-2'-deoxyguanosine is inefficient in cells belonging to both complementation groups A and B of Cockayne syndrome (CS), a developmental and neurological disorder characterized by defective transcription-c
Autor:
Monica Ropolo, Paolo Degan, Eugenia Dogliotti, Denise Lasigliè, Guido Frosina, Simonetta Zupo, Gianluigi Casartelli, Mara Foresta, Alessandro Poggi, Mariarosaria D’Errico
Publikováno v:
Free Radical Biology and Medicine. 42:1807-1817
Repair of the oxidized purine 8-oxo-7,8-dihydroguanine (8-oxoGua) is inefficient in cells belonging to the B complementation group of Cockayne syndrome (CS-B), a developmental and neurological disorder characterized by defective transcription-coupled
Autor:
Claudia Bolognesi, Monica Ropolo, Armen Nersesyan, Philip Thomas, Michael Fenech, Maryam Hor, Paola Roggieri, Siegfried Knasmueller
Publikováno v:
Mutagenesis. 30(4)
The buccal micronucleus cytome (BMCyt) assay is a minimally invasive approach for measuring DNA damage, cell proliferation, cell differentiation and cell death in exfoliated buccal cells. The main limitation for its use is the lack of knowledge about
Autor:
Vittorio Lodi, Monica Ropolo, Paola Silingardi, Annamaria Colacci, Claudia Bolognesi, Francesco Saverio Violante, Paola Roggieri, Maria Concetta Nucci, Cristina Balia
Publikováno v:
Mutation research. 747(1)
The health risk associated with low levels of ionizing radiation is still a matter of debate. A number of factors, such as non-target effects, adaptive responses and low-dose hypersensitivity, affect the long-term outcome of low-dose exposures. Cytog
Autor:
Enrico Cappelli, Mara Foresta, Guido Frosina, Monica Ropolo, Luca Proietti-De-Santis, Alessandro Poggi
Publikováno v:
IUBMB life. 63(4)
We have previously shown that DNA repair of oxidized bases (either purines or pyrimidines) is inefficient in cells from patients with Cockayne syndrome (cs), a rare developmental and neurological genetic disorder. Here, we show for the first time tha
Autor:
Renato Spaziante, Giorgio Corte, Gianluigi Casartelli, Enrico Cappelli, Mara Foresta, Monica Ropolo, Gianluigi Zona, Fabrizio Griffero, Guido Frosina, Antonio Daga, Alessandro Poggi, Annalisa Zunino
Publikováno v:
Molecular cancer research : MCR. 7(3)
It has been reported that cancer stem cells may contribute to glioma radioresistance through preferential activation of the DNA damage checkpoint response and an increase in DNA repair capacity. We have examined DNA repair in five stem and nonstem gl
Autor:
Massimo Teson, Paolo Degan, Donata Orioli, Pawel Jaruga, Tiziana Lemma, Ivano Iavarone, Monica Ropolo, Giovanna Zambruno, A. Calcagnile, Eugenia Dogliotti, Mariarosaria D’Errico, Guido Frosina, Miral Dizdaroglu, Antonia M. Pedrini, Eleonora Parlanti, Miria Stefanini
Publikováno v:
Oncogene. 26(30)
Cockayne syndrome (CS) is a rare genetic disease characterized by severe growth, mental retardation and pronounced cachexia. CS is most frequently due to mutations in either of two genes, CSB and CSA. Evidence for a role of CSB protein in the repair
Autor:
Guido, Frosina, Enrico, Cappelli, Monica, Ropolo, Paola, Fortini, Barbara, Pascucci, Eugenia, Dogliotti
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 314
Base excision repair (BER) is the main pathway for removal of endogenous DNA damage. This repair mechanism is initiated by a specific DNA glycosylase that recognizes and removes the damaged base through N-glycosylic bond hydrolysis. The generated apu
Autor:
Paola Fortini, Eugenia Dogliotti, Barbara Pascucci, Guido Frosina, Monica Ropolo, Enrico Cappelli
Publikováno v:
DNA Repair Protocols : Mammalian Systems, edited by Daryl S. Henderson, pp. 377–396, 2006
info:cnr-pdr/source/autori:Frosina G.; Cappelli E.; Ropolo M.; Fortini P.; Pascucci B.; and Dogliotti E./titolo:In vitro base excision repair assay using mammalian cell extracts/titolo_volume:DNA Repair Protocols : Mammalian Systems/curatori_volume:Daryl S. Henderson/editore:/anno:2006
Methods in Molecular Biology ISBN: 9781588295132
info:cnr-pdr/source/autori:Frosina G.; Cappelli E.; Ropolo M.; Fortini P.; Pascucci B.; and Dogliotti E./titolo:In vitro base excision repair assay using mammalian cell extracts/titolo_volume:DNA Repair Protocols : Mammalian Systems/curatori_volume:Daryl S. Henderson/editore:/anno:2006
Methods in Molecular Biology ISBN: 9781588295132
Base excision repair (BER) is the main pathway for removal of endogenous DNA damage. This repair mechanism is initiated by a specific DNA glycosylase that recognizes and removes the damaged base through N-glycosylic bond hydrolysis. The generated apu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::602a66e7b1de3f97d4a6869e05bf966c
https://publications.cnr.it/doc/134607
https://publications.cnr.it/doc/134607