Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Mohammed Z. Ferdaus"'
Autor:
Sathish K. Murali, Robert Little, Søren B. Poulsen, Mohammed Z. Ferdaus, David H. Ellison, James A. McCormick, Robert A. Fenton
Publikováno v:
Cells, Vol 11, Iss 1, p 95 (2021)
The thiazide-sensitive sodium chloride cotransporter (NCC) plays a vital role in maintaining sodium (Na+) and potassium (K+) homeostasis. NCC activity is modulated by with-no-lysine kinases 1 and 4 (WNK1 and WNK4), the abundance of which is controlle
Externí odkaz:
https://doaj.org/article/d432d2fc919446709970fa4315de27e0
Autor:
Mohammed Z. Ferdaus, Brittany D. K. Gratreak, Lauren Miller, Jinge Si, James A. McCormick, Chao‐Ling Yang, David H. Ellison, Andrew S. Terker
Publikováno v:
Physiological Reports, Vol 7, Iss 17, Pp n/a-n/a (2019)
Abstract The distal nephron is essential for calcium homeostasis. This is evidenced by disordered calcium transport following disrupted distal nephron function occurring in salt‐wasting tubulopathies or with diuretic use. A plethora of studies supp
Externí odkaz:
https://doaj.org/article/16255061e15a4fd3bcb1466e6fdfcdf7
Publikováno v:
American Journal of Physiology-Cell Physiology. 324:C1171-C1178
KCC3a expression is stimulated in alkalemia. This paper shows that bicarbonate itself is mediating this effect through a posttranscriptional mechanism. The paper also shows that this phenomenon is not mediated by aldosterone or angiotensin II.
Autor:
Yujiro Maeoka, Mohammed Z. Ferdaus, Ryan J. Cornelius, Avika Sharma, Xiao-Tong Su, Lauren N. Miller, Joshua A. Robertson, Susan B. Gurley, Chao-Ling Yang, David H. Ellison, James A. McCormick
Publikováno v:
J Am Soc Nephrol
BACKGROUND: Mutations in the ubiquitin ligase scaffold protein Cullin 3 (CUL3) gene cause the disease familial hyperkalemic hypertension (FHHt). In the kidney, mutant CUL3 (CUL3-Δ9) increases abundance of With-No-Lysine (K) Kinase 4 (WNK4), inapprop
Autor:
Andrew S. Terker, Olivier Staub, Jonathan W. Nelson, Dao Hong Lin, Anindit Mukherjee, Mohammed Z. Ferdaus, Lauren N. Miller, Philip J. Blatt, James A. McCormick
Publikováno v:
American Journal of Physiology-Renal Physiology. 317:F825-F838
Hypomagnesemia is associated with reduced kidney function and life-threatening complications and sustains hypokalemia. The distal convoluted tubule (DCT) determines final urinary Mg2+excretion and, via activity of the Na+-Cl−cotransporter (NCC), al
Autor:
Hiroki Ohara, Davis Ngarashi, Hasan M. Zahid, Toru Nabika, Mohammed Z. Ferdaus, Koichi Fujikawa
Publikováno v:
Hypertension Research. 42:981-989
Oxidative stress has been implicated in the pathophysiology of cerebral stroke. As NADPH oxidases (NOXs) play major roles in the regulation of oxidative stress, we hypothesized that reduction of NOX activity by depletion of p22phox, an essential subu
Autor:
Eric Abkin, Franco Puleo, Erica Comsti, Jesse D. Moreira, Alissa A. Frame, Kiyoung Kim, Mohammed Z. Ferdaus, Richard D Wainford, Kayla M. Nist, Kathryn R Walsh, Elizabeth Faudoa
Publikováno v:
Physiol Res
Increased sympathoexcitation and renal sodium retention during high salt intake are hallmarks of the salt sensitivity of blood pressure. The mechanism(s) by which excessive sympathetic nervous system release of norepinephrine influences renal sodium
Autor:
David H. Ellison, Gerardo Gamba, James A. McCormick, Andrew S. Terker, Lauren N. Miller, María Castañeda-Bueno, Ryan J. Cornelius, Chao-Ling Yang, Mohammed Z. Ferdaus, Kayla J. Erspamer, Wen-Hui Wang, Xiao-Tong Su
Publikováno v:
American Journal of Physiology-Renal Physiology. 315:F781-F790
With no lysine kinase 4 (WNK4) is essential to activate the thiazide-sensitive NaCl cotransporter (NCC) along the distal convoluted tubule, an effect central to the phenotype of familial hyperkalemic hypertension. Although effects on potassium and so
Autor:
Jessica Minnier, Sanjiv Kaul, Anthony P. Barnes, Jonathan W. Nelson, David H. Ellison, James A. McCormick, Mohammed Z. Ferdaus
Publikováno v:
Physiological Genomics. 50:104-116
Hypertension poses a significant challenge to vasculature homeostasis and stands as the most common cardiovascular disease in the world. Its effects are especially profound on endothelial cells that form the inner lining of the vasculature and are di
Autor:
Robert Little, James A. McCormick, Robert A. Fenton, David H. Ellison, Søren Brandt Poulsen, Mohammed Z. Ferdaus, Sathish K. Murali
Publikováno v:
Cells; Volume 11; Issue 1; Pages: 95
Cells, Vol 11, Iss 95, p 95 (2022)
Cells
Murali, S K, Little, R, Poulsen, S B, Ferdaus, M Z, Ellison, D H, McCormick, J A & Fenton, R A 2021, ' Potassium Effects on NCC Are Attenuated during Inhibition of Cullin E3-Ubiquitin Ligases ', Cells, vol. 11, no. 1, 95 . https://doi.org/10.3390/cells11010095
Cells, Vol 11, Iss 95, p 95 (2022)
Cells
Murali, S K, Little, R, Poulsen, S B, Ferdaus, M Z, Ellison, D H, McCormick, J A & Fenton, R A 2021, ' Potassium Effects on NCC Are Attenuated during Inhibition of Cullin E3-Ubiquitin Ligases ', Cells, vol. 11, no. 1, 95 . https://doi.org/10.3390/cells11010095
The thiazide sensitive sodium-chloride co-transporter (NCC) plays a vital role in maintaining sodium (Na+) and potassium (K+) homeostasis. NCC activity is modulated by the with-no-lysine kinases 1 and 4 (WNK1 and WNK4), the abundance of which are con