Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Mohamed Hosam Mourad"'
Autor:
Usama Roshdy El Safy, Manar Mohamed Fathy, Tamer Hasan Hassan, Marwa Zakaria, Mohamed Abdel Kader Al Malky, Mohamed Arafa, Hany El Sayed, Ashgan Al Ghobashy, Boshra Zaho, Attia Abdel Wahab, Mohamed Hosam Mourad
Publikováno v:
International Breastfeeding Journal, Vol 12, Iss 1, Pp 1-5 (2017)
Abstract Background Thalassemia major or Cooley’s anemia is the most severe form of beta thalassemia in which the complete lack of beta protein in the hemoglobin causes a life-threatening anemia requiring regular blood transfusions and extensive on
Externí odkaz:
https://doaj.org/article/94ee44733e134b9daa63cc67a2fa6d92
Autor:
Soha A Elhawari, Mohamed Hosam Mourad
Publikováno v:
Afro-Egyptian Journal of Infectious and Endemic Diseases. 5:265-270
Background and study aim: Typhoid fever is consideredan endemic disease in Egypt. Widal test has been used for many years for diagnosing typhoid fever in the developing countries but it has serious doubts regarding its validity. TUBEX test is a simpl
Autor:
Mohamed A. Arafa, Tamer Hassan, Marwa Zakaria, Manar Fathy, Usama El Safy, Boshra Zaho, Ashgan Al Ghobashy, Mohamed Abdel Kader Al Malky, Hany El Sayed, Mohamed Hosam Mourad, Attia Abdel Wahab
Publikováno v:
International Breastfeeding Journal
International Breastfeeding Journal, Vol 12, Iss 1, Pp 1-5 (2017)
International Breastfeeding Journal, Vol 12, Iss 1, Pp 1-5 (2017)
Background Thalassemia major or Cooley’s anemia is the most severe form of beta thalassemia in which the complete lack of beta protein in the hemoglobin causes a life-threatening anemia requiring regular blood transfusions and extensive ongoing med
Autor:
Usama El-Safy, Ahmed Al-Akhras, Mohamed Hosam Mourad, Joaquin Brintrup, Tamer Hassan, Marwa Zakaria, Elisabeth Kohne, Hadeel M. Abdelrahman, M. A. Badr
Publikováno v:
Biomedical reports. 4(6)
In β-thalassemia, certain mutations cause a complete absence of β-globin chain synthesis, termed β 0 -thalassemia, while others may allow certain β-globin production and are termed β + - or β ++ -thalassemia. The homozygous state results in sev