Zobrazeno 1 - 10
of 86
pro vyhledávání: '"Mizuho Yamada"'
Autor:
Yasufumi Kaneda, Yutaka Kawakami, Megumi Nishioka, Toshiharu Sakurai, Atsuhiro Saito, Akira Myoui, Eiji Kiyohara, Toshihiro Nakajima, Mizuho Yamada, Kazuma Sakura, Atsushi Tanemura, Akinori Yokomi, Aya Tanaka, Ichiro Katayama
Publikováno v:
Cancer Immunology, Immunotherapy. 69:1131-1140
Hemagglutinating virus of Japan (HVJ; Sendai virus) is an RNA virus that has cell fusion activity. HVJ-envelope (HVJ-E) is a UV-irradiated HVJ particle that loses viral replication and protein synthesis activity but retains cell fusion activity. We r
Autor:
Kenichi Kato, Yuiko Nagata, Mizuho Yamada, Aya Tanaka, Emi Ono, Ichiro Katayama, Atsushi Tanemura
Publikováno v:
Journal of Cosmetics, Dermatological Sciences and Applications. :135-140
Although the administration of systemic steroid for nonsegmental vitiligo in the progressive stage is a recommended treatment according to guidelines, the clinical efficacy of this regimen has not been fully established. In this study, we evaluated t
Autor:
Naoki Hamada, Kazuyoshi Kuwano, Jyutaro Fukumoto, Yoichi Nakanishi, Tomonobu Kawaguchi, Michihiro Yoshimi, Mizuho Yamada, Takashige Maeyama
Publikováno v:
Respirology. 13:639-645
Background and objective: Erythropoietin (EPO) has recently been demonstrated to have a tissue protective role by acting as an anti-apoptotic agent in various tissues, such as brain, spinal cord, heart and kidney. The purpose of this study was to det
Autor:
Michael Kasper, Mizuho Yamada, Michihiro Yoshimi, Takashige Maeyama, Kazuyoshi Kuwano, Naoki Hamada, Yoichi Nakanishi
Publikováno v:
Histochemistry and Cell Biology. 129:453-462
Epithelial-mesenchymal transition (EMT) has been considered to be involved in organ fibrogenesis. However, there is few direct evidence of this process in the pathophysiology of pulmonary fibrosis in vivo. Therefore, we tried to verify the involvemen
Autor:
Kensuke Egashira, Jutaro Fukumoto, Yoichi Nakanishi, Kenichi Hiasa, Takashige Maeyama, Michihiro Yoshimi, Naoki Hamada, Mizuho Yamada, Kazuyoshi Kuwano, Koichi Takayama
Publikováno v:
Journal of Clinical Pathology. 60:916-920
Background: Transforming growth factor-β1 (TGF-β1) has the potential to induce acute inflammation and apoptosis in lung epithelial cells and plays a central role in subsequent fibrosis. Aims: To examine a new anti-TGF-β1 therapy against lung injur
Autor:
Nobutaka Nakashima, Yoichi Nakanishi, Kenichi Hiasa, Michihiro Yoshimi, Mizuho Yamada, Kazuyoshi Kuwano, Takashige Maeyama, Naoki Hagimoto, Kensuke Egashira, Naoki Hamada
Publikováno v:
The Journal of Immunology. 175:1224-1231
Vascular endothelial growth factor (VEGF) is an angiogenesis factor with proinflammatory roles. Flt-1 is one of the specific receptors for VEGF, and soluble flt-1 (sflt-1) binds to VEGF and competitively inhibits it from binding to the receptors. We
Autor:
Mizuho Yamada, Yoichi Nakanishi, Takashige Maeyama, Michihiro Yoshimi, Kazuyoshi Kuwano, Naoki Hamada
Publikováno v:
Medicinal Chemistry. 1:49-56
Evidence that apoptosis plays an important role in the pathophysiology of lung diseases has been accumulated. Apoptosis signaling is classically composed of two principle pathways. One is a direct pathway from death receptor ligation to caspase casca
Publikováno v:
The Journal of Dermatology. 21:172-177
Cutaneous lesions of T-cell proliferative disorders are characterized by epidermotropic infiltration of the neoplastic cells and expression of intercellular adhesion molecule-1 (ICAM-1) and HLA-DR by lesional keratinocytes. Using cloned HTLV-1-infect
Publikováno v:
The Journal of Dermatology. 41:185-186
Autor:
Ryota Souzaki, Yoichi Nakanishi, Saiko Ogata-Suetsugu, Kazuyoshi Kuwano, Tomonobu Kawaguchi, Takashige Maeyama, Mizuho Yamada, Tatsuro Tajiri, Chika Harada, Naoki Hamada, Tomoaki Taguchi
Publikováno v:
American journal of respiratory and critical care medicine. 183(6)
Epidermal growth factor receptor (EGFR) and its ligands play important roles in the regeneration of damaged epithelium and proliferation of various epithelial tumors. Although the EGFR-tyrosine kinase inhibitor gefitinib is effective against advanced