Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Misao Iizuka"'
Autor:
Masashi Satoh, Misao Iizuka, Masataka Majima, Chizuru Ohwa, Akito Hattori, Luc Van Kaer, Kazuya Iwabuchi
Publikováno v:
ImmunologyREFERENCES. 165(4)
Obesity is accompanied by and accelerated with chronic inflammation in adipose tissue, especially visceral adipose tissue (VAT). This low-level inflammation predisposes the host to the development of metabolic disease, most notably type 2 diabetes. W
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::38d07cec956dcd57388bd7a6707b53a8
https://pubmed.ncbi.nlm.nih.gov/35137411
https://pubmed.ncbi.nlm.nih.gov/35137411
Autor:
Natsumi Tajiri, Masaru Taniguchi, Nobuyoshi Kitaichi, Kazuya Iwabuchi, Misao Iizuka, Taiki Kato, Masashi Satoh
Publikováno v:
Experimental eye research. 203
Experimental autoimmune uveoretinitis (EAU) in mice provides a useful platform to study the pathogenesis and experimental therapeutics of human uveitis. One often used EAU model employs C57BL/6 (B6) mice sensitized with a peptide residue having 1 to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7735dc5c17af36cd826084029e4f336
https://pubmed.ncbi.nlm.nih.gov/33347870
https://pubmed.ncbi.nlm.nih.gov/33347870
Autor:
Koki Fujita, Masashi Satoh, Misao Iizuka, Miyuki Hoshino, Kazuya Iwabuchi, Satoshi Fujii, Luc Van Kaer, Christopher Stuart Clingan
Publikováno v:
Scientific Reports
It has been shown that CD1d expression and glycolipid-reactive, CD1d-restricted NKT cells exacerbate the development of obesity and insulin resistance in mice. However, the relevant CD1d-expressing cells that influence the effects of NKT cells on the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2ccd142439244ea121de96ea63a3c0e5
https://pubmed.ncbi.nlm.nih.gov/27329323
https://pubmed.ncbi.nlm.nih.gov/27329323
Publikováno v:
Molecular Immunology. 42:71-78
Using a panel of transfectant B lymphoma cells expressing varying amounts of the mutant Fas together with the endogenous wild type Fas, semi-quantitative studies on the dominant negative effect of a murine mutant Fas molecule lacking death domain wer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da4b4fc5f4c99d7eace4e9bf46fd61cc
https://doi.org/10.1016/j.molimm.2004.07.042
https://doi.org/10.1016/j.molimm.2004.07.042
Publikováno v:
Journal of Clinical & Cellular Immunology.
Objective: Kidney involvement is one of the most important clinical features of systemic lupus erythematosus (SLE). Therefore, histopathological analysis of renal sections is necessary to evaluate the magnitude of lupus-like diseases. However, histop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::99968f0180ff69f20e18cc7da832c3f4
https://doi.org/10.4172/2155-9899.1000209
https://doi.org/10.4172/2155-9899.1000209
Autor:
Mushtaq A. Siddiqi, Makoto Tachibana, Yoshiko Shirota-Someya, Misao Iizuka, Nobukata Shinohara, Atsushi Koito, Yuko Ikegami, Satoko Tahara-Hanaoka, Koji Eshima
Publikováno v:
Journal of Biological Chemistry. 275:20288-20294
Despite extensive mutational studies on the human CD4 molecule and its affinity to human immunodeficiency virus (HIV) envelope glycoprotein gp120, coreceptor functions of such mutant molecules have only been examined by indirect measurement of their
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c1d44412c3013e53d6c5ac1226413767
https://doi.org/10.1074/jbc.m001917200
https://doi.org/10.1074/jbc.m001917200
Autor:
Kazuya Iwabuchi, Nobukata Shinohara, Harumi Suzuki, Koji Eshima, Hirosuke Kobayashi, Misao Iizuka, Kenichi Kokubo, Sayuri Chiba
Publikováno v:
Immunology letters. 144(1-2)
During viral infection, CD8(+) cytotoxic T lymphocytes (CTL) play a central role to eliminate viruses by destructing virus-infected cells utilizing two cytolytic pathways, i.e., perforin/granzyme pathway and FasL-Fas pathway. It has been shown that e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::88ee882f070c5f73809e57f38df9d6a1
https://pubmed.ncbi.nlm.nih.gov/22425747
https://pubmed.ncbi.nlm.nih.gov/22425747