Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Misa Saijo"'
Publikováno v:
Japanese Journal of Thrombosis and Hemostasis. 26:290-296
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 85:2116-2121
Blocking-type TSH-binding inhibitor Igs (TBIIs) are known to cause hypothyroidism and an atrophic thyroid gland in patients with primary myxedema. They can block the activity of thyroid-stimulating antibodies (TSAbs) in Graves' patients as well as th
Autor:
Megumi Inuzuka, Naohisa Tamura, Nobuko Yamada, Goro Katsuura, Naofumi Oyamada, Kousaku Ohinata, Masakatsu Sone, Yasutomo Fukunaga, Daisuke Taura, Takuhiro Sonoyama, Katsutoshi Kojima, Kyoko Honda, Yudai Takeuchi, Eri Kondo, Toshihiro Fujii, Takuo Nambu, Misa Saijo, Masako Miura, Naotetsu Kanamoto, Akihiro Yasoda, Hiroshi Arai, Kazuwa Nakao
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f96cb3ff8e81e503840da07a4e9deea1
https://doi.org/10.1210/endo-meetings.2010.part3.p9.p3-413
https://doi.org/10.1210/endo-meetings.2010.part3.p9.p3-413
Autor:
Naotetsu Kanamoto, Takashi Akamizu, Yuji Hataya, Yoshiyuki Hattori, Tomohiko Mori, Kenji Moriyama, Fumihiko Matsuda, Kazuwa Nakao, Misa Saijo, Jyoji Okuda
Publikováno v:
Journal of endocrinological investigation. 26(11)
Anti-TSH receptor antibodies (TRAbs) have been known to be involved in Graves’ disease and primary hypothyroidism. We previously isolated and reconstituted immunoglobulin (Ig) genes of Epstein-Barr virus-transformed B cell clones producing monoclon
Autor:
Takashi Akamizu, Kenji Moriyama, Hideshi Kuzuya, Naotetsu Kanamoto, Tetsuya Tagami, Kazuwa Nakao, Misa Saijo, Yuji Hataya, Akira Shimatsu, Takeshi Usui
Publikováno v:
The Journal of clinical endocrinology and metabolism. 87(11)
Bisphenol A (BPA), a monomer of polycarbonate plastics, has been shown to possess estrogenic properties and act as an agonist for the estrogen receptors. Although an epidemiologically based investigation has suggested that some chemicals could disrup
Autor:
Naotetsu Kanamoto, Yuji Hataya, Kazuwa Nakao, Akira Shimatsu, Kenji Moriyama, Misa Saijo, Kazuhiko Takaya, Hiroshi Hosoda, Kenji Kangawa, Takashi Akamizu, Hiroyuki Ariyasu, Masayasu Kojima, Kiminori Hosoda
Publikováno v:
The Journal of clinical endocrinology and metabolism. 86(10)
Ghrelin, an endogenous ligand for the GH secretagogue receptor, is a novel acylated peptide produced in the gastrointestinal endocrine cells as well as neuroendocrine cells in the hypothalamus. The Ser(3) residue of ghrelin is modified by n-octanoic
Autor:
Takashi Akamizu, Kenji Moriyama, Yoshiyuki Hattori, Kazuwa Nakao, Naotetsu Kanamoto, Misa Saijo, Nobuyuki Ito
Publikováno v:
Molecular and cellular endocrinology. 182(2)
Thyrotropin receptor (TSHR) is a member of the glycoprotein hormone receptor family and an autoantigen of Graves’ disease. Various attempts have been made to obtain a large amount of soluble ectodomain of TSHR in insect or mammalian cells, but most
Autor:
Jaeduk Yoshimura Noh, Yoshihiko Saito, Takashi Akamizu, Hitorni Hiratani, Kazuwa Nakao, Naotetsu Kanamoto, Donald W. Bowden, Yoshihiro Miyamoto, Misa Saijo, Stephen S. Rich, Michèle M. Sale
Publikováno v:
Thyroid : official journal of the American Thyroid Association. 10(10)
In a previous study we identified a microsatellite marker near the thyrotropin receptor (TSHR) gene. Studies with this marker, TSHR-CA, revealed a significant association between autoimmune thyroid disease (AITD) in Japanese patients and one specific
Autor:
Masako Miura, Kenji Moriyama, Mitsuyoshi Umemoto, Takashi Akamizu, Katsumi Taniguchi, Kazuwa Nakao, Misa Saijo
Publikováno v:
Endocrine journal. 46(5)
We present the case of a 66-year-old woman with Hashimoto's thyroiditis, who showed extremely high concentrations of serum thyroglobulin (Tg). Serum Tg levels were markedly elevated following a slight elevation of serum thyrotropin (TSH) (22,000 ng/m
Publikováno v:
Endocrinology. 140(4)
Anti-TSH receptor autoantibodies (TSHRAbs) are known to be involved in Graves' disease. To elucidate the molecular mechanism of the pathogenesis of Graves' disease, we previously isolated and reconstituted the Ig genes of two B cell clones (101-2 and