Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Mina Kazemzadeh"'
Autor:
Nazila Valatabar, Fatemeh Oroojalian, Mina Kazemzadeh, Amir Ali Mokhtarzadeh, Reza Safaralizadeh, Amirhossein Sahebkar
Publikováno v:
Journal of Nanobiotechnology, Vol 22, Iss 1, Pp 1-50 (2024)
Abstract Gene therapy is a therapeutic option for mitigating diseases that do not respond well to pharmacological therapy. This type of therapy allows for correcting altered and defective genes by transferring nucleic acids to target cells. Notably,
Externí odkaz:
https://doaj.org/article/9409387ef82c434c95e358e29f471a73
Autor:
Vincent De Smet, Elif Gürbüz, Nathalie Eysackers, Liza Dewyse, Ayla Smout, Mina Kazemzadeh Dastjerd, Pierre Lefesvre, Nouredin Messaoudi, Hendrik Reynaert, Stefaan Verhulst, Inge Mannaerts, Leo A. van Grunsven
Publikováno v:
JHEP Reports, Vol 6, Iss 5, Pp 101036- (2024)
Background & Aims: Chronic liver disease (CLD) remains a global health issue associated with a significant disease burden. Liver fibrosis, a hallmark of CLD, is characterised by the activation of hepatic stellate cells (HSCs) that gain profibrotic ch
Externí odkaz:
https://doaj.org/article/a220db56f1c24cd99ae3f6a1a5825e81
Autor:
Vincent De Smet, Nathalie Eysackers, Vincent Merens, Mina Kazemzadeh Dastjerd, Georg Halder, Stefaan Verhulst, Inge Mannaerts, Leo A. van Grunsven
Publikováno v:
Cell Death and Disease, Vol 12, Iss 12, Pp 1-10 (2021)
Abstract Activated hepatic stellate cells (aHSC) are the main source of extra cellular matrix in liver fibrosis. Activation is classically divided in two phases: initiation and perpetuation. Currently, HSC-based therapeutic candidates largely focus o
Externí odkaz:
https://doaj.org/article/8ea65b2a9036464990e291a1c81feff9
Publikováno v:
International Journal of Genomics, Vol 2014 (2014)
Discovered in 1993, micoRNAs (miRNAs) are now recognized as one of the major regulatory gene families in eukaryotes. To date, 24521 microRNAs have been discovered and there are certainly more to come. It was primarily acknowledged that miRNAs result
Externí odkaz:
https://doaj.org/article/9d4571ae7d154fa7ba85a0fd0eca7c08
Akademický článek
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Autor:
Georg Halder, Nathalie Eysackers, Inge Mannaerts, Stefaan Verhulst, Leo A. van Grunsven, Mina Kazemzadeh Dastjerd, Vincent Merens, Vincent De Smet
Publikováno v:
Cell Death and Disease, Vol 12, Iss 12, Pp 1-10 (2021)
Cell Death & Disease
Cell Death & Disease
Activated hepatic stellate cells (aHSC) are the main source of extra cellular matrix in liver fibrosis. Activation is classically divided in two phases: initiation and perpetuation. Currently, HSC-based therapeutic candidates largely focus on targeti
Autor:
Alireza Khabbazi, Hossein Daghagh, Maryam Nasiri Aghdam, Yousef Daneshmandpour, Haniyeh Rahbar Kafshboran, Ebrahim Sakhinia, Jafar Nouri Nojadeh, Hamid Hamzeiy, Mina Kazemzadeh
Publikováno v:
Immunology Letters. 221:27-32
Autoinflammation and PLCG2-associated antibody deficiency and immune dysregulation (APLAID) is an autosomal dominant autoinflammatory disease characterized by episodic skin, musculoskeletal, ophthalmic and gastrointestinal tract symptoms. Here we rep
Akademický článek
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Autor:
De Smet, Vincent, Verhulst, Stefaan, Dewyse, Liza, Dastjerd, Mina Kazemzadeh, Reynaert, Hendrik, Halder, Georg, Mannaerts, Inge, van Grunsven, Leo A.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______3848::40eed96fba83edae45fe9c92bec71703
https://biblio.vub.ac.be/vubir/a-profibrotic-role-for-the-orphan-gprotein-coupled-receptor-176-during-hepatic-stellate-cell-activation(954b5346-4c34-4da5-941c-08f038f27782).html
https://biblio.vub.ac.be/vubir/a-profibrotic-role-for-the-orphan-gprotein-coupled-receptor-176-during-hepatic-stellate-cell-activation(954b5346-4c34-4da5-941c-08f038f27782).html
Autor:
Reza Safaralizadeh, Shirin Azarbarzin, Alavieh Fateh, Mina Kazemzadeh, Mohammad Ali Hosseinpour Feizi
Publikováno v:
Biochemical Genetics. 55:244-252
MicroRNAs, a class of gene expression regulatory non-coding RNAs, participate in the pathogenic mechanisms of gastric cancer which is one of the life-treating cancers. Due to its aberrant expression in some types of human cancer, miR-383 has the valu