Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Milo Careaga"'
Autor:
Elizabeth L. Berg, Tianna M. Ching, Donald A. Bruun, Josef K. Rivera, Milo Careaga, Jacob Ellegood, Jason P. Lerch, Markus Wöhr, Pamela J. Lein, Jill L. Silverman
Publikováno v:
Journal of Neurodevelopmental Disorders, Vol 12, Iss 1, Pp 1-15 (2020)
Abstract Background Neurodevelopmental disorders (NDDs), including intellectual disability, attention deficit hyperactivity disorder (ADHD), and autism spectrum disorder (ASD), are pervasive, lifelong disorders for which pharmacological interventions
Externí odkaz:
https://doaj.org/article/740a18ca2df0470a99cf6e1fbaa83ca8
Publikováno v:
Brain Sciences, Vol 12, Iss 2, p 220 (2022)
The prevalence of autism spectrum disorder (ASD) has starkly increased, instigating research into risk factors for ASD. This research has identified immune risk factors for ASD, along with evidence of immune dysfunction and excess inflammation freque
Externí odkaz:
https://doaj.org/article/1a44f3d4720c4342a536ed199f46cfab
T cell populations in children with autism spectrum disorder and co-morbid gastrointestinal symptoms
Autor:
Destanie R. Rose, Houa Yang, Milo Careaga, Kathy Angkustsiri, Judy Van de Water, Paul Ashwood
Publikováno v:
Brain, Behavior, & Immunity - Health, Vol 2, Iss , Pp 100042- (2020)
Children with ASD are more likely to experience gastrointestinal (GI) symptoms than typically-developed children. Numerous studies have reported immune abnormalities and inflammatory profiles in the majority of individuals with ASD. Immune dysfunctio
Externí odkaz:
https://doaj.org/article/cac3264dd66c467da70745df08137c30
Autor:
Tyler A. Lesh, Milo Careaga, Destanie R. Rose, A. Kimberley McAllister, Judy Van de Water, Cameron S. Carter, Paul Ashwood
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-11 (2018)
Abstract Background Over the past 30 years, evidence has been accumulating for an immunological component to schizophrenia etiology, including genetic links to the major histocompatibility complex, microglia activation, and dysregulated cytokine prof
Externí odkaz:
https://doaj.org/article/e7df5f660c98468898c2600397751f81
Publikováno v:
PLoS ONE, Vol 9, Iss 4, p e94475 (2014)
BACKGROUND:Increased rates of autoinflammatory and autoimmune disorders have been observed in female premutation carriers of CGG repeat expansion alleles of between 55-200 repeats in the fragile X mental retardation 1 (FMR1) gene. To determine whethe
Externí odkaz:
https://doaj.org/article/d2c71af971cf4866bf282dc9288e59f7
Publikováno v:
Mediators of Inflammation, Vol 2013 (2013)
Maternal immune activation (MIA) is a potential risk factor for autism spectrum disorder (ASD) and schizophrenia (SZ). In rodents, MIA results in changes in cytokine profiles and abnormal behaviors in the offspring that model these neuropsychiatric c
Externí odkaz:
https://doaj.org/article/03967e6899f3417fb1b2759278a4b66b
Publikováno v:
Mediators of Inflammation, Vol 2013 (2013)
Autism spectrum disorders (ASD) are characterized by impairments in communication, social interactions, and repetitive behaviors. While the etiology of ASD is complex and likely involves the interplay of genetic and environmental factors, growing evi
Externí odkaz:
https://doaj.org/article/850048dce2af418ea5a53030a663ffe9
Autor:
Joseph C. Boktor, Mark D. Adame, Destanie R. Rose, Cynthia M. Schumann, Karl D. Murray, Melissa D. Bauman, Milo Careaga, Sarkis K. Mazmanian, Paul Ashwood, Brittany D. Needham
Publikováno v:
Molecular psychiatry, vol 27, iss 12
Mol Psychiatry
Mol Psychiatry
Epidemiological evidence implicates severe maternal infections as risk factors for neurodevelopmental disorders, such as ASD and schizophrenia. Accordingly, animal models mimicking infection during pregnancy, including the maternal immune activation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::871944e03223b1a96446dedd15fceb21
https://escholarship.org/uc/item/51r9k74s
https://escholarship.org/uc/item/51r9k74s
Publikováno v:
Brain Behav Immun
The BTBR T(+)Itpr3(tf)/J (BTBR) mouse has been used as a complex genetic model of Autism Spectrum Disorders (ASD). While the specific mechanisms underlying BTBR behavioral phenotypes are poorly understood, prior studies have implicated profound diffe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::961c9d5a164a832214e74ffbf675c801
https://escholarship.org/uc/item/644203vt
https://escholarship.org/uc/item/644203vt
The BTBR T+Itpr3tf/J (BTBR) mouse has been used as a complex genetic model of Autism Spectrum Disorders (ASD). While the specific mechanisms underlying BTBR behavioral phenotypes are poorly understood, prior studies have implicated profound differenc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9338bb10bda76704e2b3907bf72bc2b