Zobrazeno 1 - 10
of 39
pro vyhledávání: '"Milan Basta"'
Autor:
Mark P. Mattson, Milan Basta, Yi-Lin Cheng, Thiruma V. Arumugam, Jong Hwan Lee, Xinzhi Chen, Srinivasulu Chigurupati
Publikováno v:
NeuroMolecular Medicine. 20:63-72
Acute ischemic stroke causes a high rate of deaths and permanent neurological deficits in survivors. Current interventional treatment, in the form of enzymatic thrombolysis, benefits only a small percentage of patients. Brain ischemia triggers mobili
Autor:
Milan Basta
Publikováno v:
Immune Aspects of Biopharmaceuticals and Nanomedicines ISBN: 9780203731536
Immune Aspects of Biopharmaceuticals and Nanomedicines
Immune Aspects of Biopharmaceuticals and Nanomedicines
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b2da5f1ee688b6be5fc22fda382da00e
https://doi.org/10.1201/b22372-10
https://doi.org/10.1201/b22372-10
Autor:
Silvia Manzanero, David C. Reutens, Tim Magnus, Mathias Gelderblom, Sic L. Chan, Milan Basta, Alexander Widiapradja, Sung-Chun Tang, Viktor Vegh, Mark P. Mattson, Thiruma V. Arumugam, Christopher G. Sobey, Ker Zhing Lok, Yi-Lin Cheng, John Thundyil, Dong-Gyu Jo, Dale Pavlovski
Publikováno v:
Journal of Neurochemistry. 122:321-332
Intravenous immunoglobulin (IVIg) preparations obtained by fractionating blood plasma, are increasingly being used increasingly as an effective therapeutic agent in treatment of several inflammatory diseases. Its use as a potential therapeutic agent
Autor:
Sung-Chun Tang, Thiruma V. Arumugam, Aiwu Cheng, Dong-Gyu Jo, Alexander O. Vortmeyer, Daniel N. Granger, Mark P. Mattson, Justin D. Lathia, Mohamed R. Mughal, Tim Magnus, Sic L. Chan, Srinivasulu Chigurupati, Milan Basta, Xin Ouyang, David P. Fairlie
Publikováno v:
Proceedings of the National Academy of Sciences. 104:14104-14109
Stroke is among the three leading causes of death worldwide and the most frequent cause of permanent disability. Brain ischemia induces an inflammatory response involving activated complement fragments. Here we show that i.v. Ig (IVIG) treatment, whi
Autor:
Terez Shea-Donohue, Jimie Anderson, George C. Tsokos, Scott T. Rehrig, Sherry D. Fleming, Milan Basta
Publikováno v:
Clinical Immunology. 114:137-146
Intravenous immunoglobulin (IVIG) has been found useful in the treatment of various clinical entities and its effect has been associated with inhibition of complement-mediated tissue damage. The aim of this study was to determine the ability of IVIG
Publikováno v:
Pediatric research. 75(5)
Neonatal asphyxia is one of the leading causes of death in newborn and permanent neurological disabilities in surviving children. The underlying hypoxic-ischemic (HI) injury triggers an inflammatory response lading to neuronal damage. Here, we tested
Autor:
Milan Basta
Publikováno v:
Scopus-Elsevier
Summary High-dose intravenous immune globulin (IVIG) exerts a beneficial effect in a variety of immune disorders. One possible underlying mechanism of this effect could be interference with the complement system. This conclusion was based on the resu
Publikováno v:
Hybridoma. 15:69-75
A monoclonal antibody designated B1.9.E-2 was produced and characterized to facilitate study of the immunizing antigen-a serum glycoprotein of 120 kDa (sgp 120) of unknown function. The antibody, produced in mice, reacts with 1-2 epitopes located in
Autor:
Alexander, Widiapradja, Viktor, Vegh, Ker Zhing, Lok, Silvia, Manzanero, John, Thundyil, Mathias, Gelderblom, Yi-Lin, Cheng, Dale, Pavlovski, Sung-Chun, Tang, Dong-Gyu, Jo, Tim, Magnus, Sic L, Chan, Christopher G, Sobey, David, Reutens, Milan, Basta, Mark P, Mattson, Thiruma V, Arumugam
Publikováno v:
Journal of neurochemistry. 122(2)
Intravenous immunoglobulin (IVIg) preparations obtained by fractionating blood plasma, are increasingly being used increasingly as an effective therapeutic agent in treatment of several inflammatory diseases. Its use as a potential therapeutic agent
Autor:
Milan Basta, Marinos C. Dalakas
Publikováno v:
Journal of Clinical Investigation. 94:1729-1735
In patients with dermatomyositis (DM) the earliest lesion is microvasculopathy mediated by deposition of C5b-C9 membranolytic attack complex (MAC) on intramuscular capillaries. This leads sequentially to muscle ischemia, necrosis of muscle fibers, an