Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Miklos Sahin-Toth"'
Autor:
Miklos Sahin-Toth, Alexandra Demcsak
Publikováno v:
Gastroenterology. 163:761-763
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e89dcafd019696b06331ed2af1a44322
https://doi.org/10.1053/j.gastro.2022.06.001
https://doi.org/10.1053/j.gastro.2022.06.001
Autor:
Alexandra Demcsak, Miklos Sahin-Toth
Publikováno v:
Gastroenterology. 162:S-245
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c31b682ffb73f1ffb1664c83e7525e19
https://doi.org/10.1016/s0016-5085(22)60583-3
https://doi.org/10.1016/s0016-5085(22)60583-3
Autor:
Alexandra Demcsák, Miklós Sahin-Tóth
Publikováno v:
Cellular and Molecular Gastroenterology and Hepatology, Vol 18, Iss 3, Pp 101361- (2024)
Background & Aims: Heterozygous SPINK1 mutations are strong risk factors for chronic pancreatitis in humans, yet heterozygous disruption of mouse Spink1 yielded no pancreatic phenotype. To resolve this contradiction, we used CRISPR/Cas9-mediated geno
Externí odkaz:
https://doaj.org/article/72300ff5086f44febbbc45b26aa3f12f
Autor:
Heiko, Witt, Miklos Sahin Toth, Olfert, Landt, Jian Min Chen, Thilo, Kahne, Drenth, Joost P. H., Zoltan, Kukor, Edit, Szepessy, Walter, Halangk, Stefan, Dahm, Klaus, Rohde, Hans Ulrich Schulz, Cedric Le Marechal, Nejat, Akar, Ammann, Rudolf W., Kaspar, Truninger, Mario, Bargetzi, Eesh, Bhatia, Carlo, Castellani, Giulia Martina Cavestro, Milos, Cerny, DESTRO-BISOL, Giovanni, Spedini, Gabriella, Hans, Eiberg, Jansen, Jan B. M. J., Monika, Koudova, Eva, Rausova, Milan, Macek, Macek Jr, M., Nuria, Malats, Real, Francisco X., Hans Jurgen Menzel, Pedro, Moral, Roberta, Galavotti, Pier Franco Pignatti, Olga, Rickards, Julius, Spicak, Narcis Octavian Zarnescu, Wolfgang, Bock, Gress, Thomas M., Helmut, Friess, Johann, Ockenga, Hartmut, Schmidt, Roland, Pfutzer, Matthias, Lohr, Peter, Simon, Frank Ulrich Weiss, Lerch, Markus M., Niels, Teich, Volker, Keim, Thomas, Berg, Bertram, Wiedenmann, Werner, Luck, David Alexander Groneberg, Michael, Becker, Thomas, Keil, Andreas, Kage, Jana, Bernardova, Markus, Braun, Claudia, Guldner, Juliane, Halangk, Jonas, Rosendahl, Ulrike, Witt, Matthias, Treiber, Renate, Nickel, Claude, Ferec
Publikováno v:
Nature Genetics, 38, 6, pp. 668-73
Recercat. Dipósit de la Recerca de Catalunya
instname
Nature Genetics, 38, 668-73
Karolinska Institutet
Recercat. Dipósit de la Recerca de Catalunya
instname
Nature Genetics, 38, 668-73
Karolinska Institutet
Chronic pancreatitis is a common inflammatory disease of the pancreas. Mutations in the genes encoding cationic trypsinogen (PRSS1) and the pancreatic secretory trypsin inhibitor (SPINK1) are associated with chronic pancreatitis. Because increased pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c84ef25b3b8f04b9a05573390b501145
https://doi.org/10.1038/ng1797
https://doi.org/10.1038/ng1797
Autor:
Andrea Parniczky, Balazs Kui, Andrea Szentesi, Anita Balazs, Akos Szucs, Dora Mosztbacher, Szilard Godi, Tamas Takacs, Ferenc Izbeki, Adrienn Halasz, Janos Novak, Istvan Hritz, Csaba Gog, Janos Sumegi, Marta Varga, Barnabas Bod, Jozsef Hamvas, Monika Muller Varga, Miklos Sahin-Toth, Peter Hegyi
Publikováno v:
Pancreatology. 16:S46-S47
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::84d7212cd5c1880c9bc9ca301f3088b7
https://doi.org/10.1016/j.pan.2016.06.175
https://doi.org/10.1016/j.pan.2016.06.175
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-11 (2022)
Abstract Genetic mutations in pancreatic digestive enzymes may cause protein misfolding, endoplasmic reticulum (ER) stress and chronic pancreatitis. The CPA1 N256K mouse model carries the human p.N256K carboxypeptidase A1 (CPA1) mutation, a classic e
Externí odkaz:
https://doaj.org/article/3fe704728e884b17b3d6eec0669d960f
Autor:
Zsófia Gabriella Pesei, Zsanett Jancsó, Alexandra Demcsák, Balázs Csaba Németh, Sandor Vajda, Miklós Sahin-Tóth
Publikováno v:
JCI Insight, Vol 7, Iss 21 (2022)
Pancreatitis, the inflammatory disorder of the pancreas, has no specific therapy. Genetic, biochemical, and animal model studies revealed that trypsin plays a central role in the onset and progression of pancreatitis. Here, we performed biochemical a
Externí odkaz:
https://doaj.org/article/8dbf9e72515a47ec83d1da9b27a95b5f
Publikováno v:
Pancreatology. 15:S51
s / Pancreatology 15 (2015) S1eS141 S51 stomach and identified in the pancreas. GHRL has been demonstrated to protect pancreatic tissue from caerulein-induced pancreatitis (CIP). Aims: To investigate the effect of GHRL and caerulein on gene expressio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::503221ba6b1726eda3ac9984faeea9bf
https://doi.org/10.1016/j.pan.2015.05.203
https://doi.org/10.1016/j.pan.2015.05.203
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Akademický článek
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