Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Mikiko Tada"'
Autor:
Haruko Nakamura, Hiroshi Doi, Yosuke Miyaji, Taishi Wada, Erisa Takahashi, Mikiko Tada, Hiromi Fukuda, Atsushi Fujita, Yuichi Higashiyama, Yuri Nagao, Kazue Kimura, Masaharu Hayashi, Kyoko Hoshino, Naomichi Matsumoto, Fumiaki Tanaka
Publikováno v:
BMC Neurology, Vol 24, Iss 1, Pp 1-8 (2024)
Abstract Background Pathogenic variants in Gap junction protein beta 1 (GJB1), which encodes Connexin 32, are known to cause X-linked Charcot-Marie-Tooth disease (CMTX), the second most common form of CMT. CMTX presents with the following five centra
Externí odkaz:
https://doaj.org/article/0534b655aeef49dfa90fcf064f7ccf7e
Autor:
Takuya Ikeda, Keita Takahashi, Minatsu Higashi, Hiroyasu Komiya, Tetsuya Asano, Akihiro Ogasawara, Shun Kubota, Shunta Hashiguchi, Misako Kunii, Kenichi Tanaka, Mikiko Tada, Hiroshi Doi, Hideyuki Takeuchi, Kohtaro Takei, Fumiaki Tanaka
Publikováno v:
Cell Death Discovery, Vol 9, Iss 1, Pp 1-10 (2023)
Abstract Nogo–Nogo receptor 1 (NgR1) signaling is significantly implicated in neurodegeneration in amyotrophic lateral sclerosis (ALS). We previously showed that lateral olfactory tract usher substance (LOTUS) is an endogenous antagonist of NgR1 th
Externí odkaz:
https://doaj.org/article/6bc952e6666947de89b52f8fcf5a7f1c
Autor:
Yuko Kawamoto, Mikiko Tada, Tetsuya Asano, Haruko Nakamura, Aoi Jitsuki-Takahashi, Hiroko Makihara, Shun Kubota, Shunta Hashiguchi, Misako Kunii, Toshio Ohshima, Yoshio Goshima, Hideyuki Takeuchi, Hiroshi Doi, Fumio Nakamura, Fumiaki Tanaka
Publikováno v:
Frontiers in Neurology, Vol 13 (2022)
In amyotrophic lateral sclerosis (ALS), neurodegeneration is characterized by distal axonopathy that begins at the distal axons, including the neuromuscular junctions, and progresses proximally in a “dying back” manner prior to the degeneration o
Externí odkaz:
https://doaj.org/article/9485f55f46a343b097fec5aaa355cf66
Autor:
Shun Kubota, Hiroshi Doi, Shigeru Koyano, Kenichi Tanaka, Hiroyasu Komiya, Atsuko Katsumoto, Shingo Ikeda, Shunta Hashiguchi, Haruko Nakamura, Ryoko Fukai, Keita Takahashi, Misako Kunii, Mikiko Tada, Hideyuki Takeuchi, Fumiaki Tanaka
Publikováno v:
Molecular Brain, Vol 14, Iss 1, Pp 1-10 (2021)
Abstract Intracellular aggregates are a common pathological hallmark of neurodegenerative diseases such as polyglutamine (polyQ) diseases, amyotrophic lateral sclerosis (ALS), Parkinson’s disease (PD), and multiple system atrophy (MSA). Aggregates
Externí odkaz:
https://doaj.org/article/28ec26773d1e475c9691a8fbd146d1e6
Autor:
Hiroyasu Komiya, Hideyuki Takeuchi, Yuki Ogawa, Yuki Hatooka, Keita Takahashi, Atsuko Katsumoto, Shun Kubota, Haruko Nakamura, Misako Kunii, Mikiko Tada, Hiroshi Doi, Fumiaki Tanaka
Publikováno v:
Molecular Brain, Vol 13, Iss 1, Pp 1-4 (2020)
Abstract It remains controversial whether circulating monocytes expressing CCR2 infiltrate the central nervous system (CNS) and contribute to pathogenicity of amyotrophic lateral sclerosis (ALS). A previous report used conventional immunohistochemist
Externí odkaz:
https://doaj.org/article/20091d45a3274496a923e1de99d6684b
Autor:
Hiroyasu Komiya, Kohei Harada, Ryoji Morishita, Shunta Hashiguchi, Mikiko Tada, Kenichi Tanaka, Hiroshi Doi, Hideyuki Takeuchi, Fumiaki Tanaka
Publikováno v:
eNeurologicalSci, Vol 25, Iss , Pp 100377- (2021)
Externí odkaz:
https://doaj.org/article/5940f26f7dd14e8a82f114b574850b74
Autor:
Takuya Ikeda, Hideyuki Takeuchi, Keita Takahashi, Haruko Nakamura, Misako Kunii, Atsuko Katsumoto, Mikiko Tada, Yuichi Higashiyama, Takashi Hibiya, Shigeaki Suzuki, Ichizo Nishino, Shigeru Koyano, Hiroshi Doi, Fumiaki Tanaka
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Chronic tonsillitis has been attracted attention as a source of abnormal immune responses and a possible trigger of autoimmune diseases such as IgA nephritis, IgA vasculitis, palmoplantar pustulosis, psoriasis, rheumatoid arthritis, Behçet’s disea
Externí odkaz:
https://doaj.org/article/e6c1d0ba2a2e40aea0407f6221789b70
Autor:
Keita Takahashi, Hideyuki Takeuchi, Yuji Kurihara, Hiroshi Doi, Misako Kunii, Kenichi Tanaka, Haruko Nakamura, Ryoko Fukai, Atsuko Tomita-Katsumoto, Mikiko Tada, Yuichi Higashiyama, Hideto Joki, Shigeru Koyano, Kohtaro Takei, Fumiaki Tanaka
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-7 (2018)
Abstract Background Although inflammation in the central nervous system is responsible for multiple neurological diseases, the lack of appropriate biomarkers makes it difficult to evaluate inflammatory activities in these diseases. Therefore, a new b
Externí odkaz:
https://doaj.org/article/567adf62af5f46f28b68e61013f503bd
Ataxic phenotype with altered CaV3.1 channel property in a mouse model for spinocerebellar ataxia 42
Autor:
Shunta Hashiguchi, Hiroshi Doi, Misako Kunii, Yukihiro Nakamura, Misa Shimuta, Etsuko Suzuki, Shigeru Koyano, Masaki Okubo, Hitaru Kishida, Masaaki Shiina, Kazuhiro Ogata, Fumiko Hirashima, Yukichi Inoue, Shun Kubota, Noriko Hayashi, Haruko Nakamura, Keita Takahashi, Atsuko Katsumoto, Mikiko Tada, Kenichi Tanaka, Toshikuni Sasaoka, Satoko Miyatake, Noriko Miyake, Hirotomo Saitsu, Nozomu Sato, Kokoro Ozaki, Kiyobumi Ohta, Takanori Yokota, Hidehiro Mizusawa, Jun Mitsui, Hiroyuki Ishiura, Jun Yoshimura, Shinichi Morishita, Shoji Tsuji, Hideyuki Takeuchi, Kinya Ishikawa, Naomichi Matsumoto, Taro Ishikawa, Fumiaki Tanaka
Publikováno v:
Neurobiology of Disease, Vol 130, Iss , Pp 104516- (2019)
Spinocerebellar ataxia 42 (SCA42) is a neurodegenerative disorder recently shown to be caused by c.5144G > A (p.Arg1715His) mutation in CACNA1G, which encodes the T-type voltage-gated calcium channel CaV3.1. Here, we describe a large Japanese family
Externí odkaz:
https://doaj.org/article/159b1298c64f481c88470219971663c5
Autor:
Kenichi Tanaka, Mikiko Tada, Shingo Ikeda, Hiroyasu Komiya, Ryoko Fukai, Shunta Hashiguchi, Haruko Nakamura, Keita Takahashi, Shun Kubota, Fumiaki Tanaka, Atsuko Katsumoto, Hideyuki Takeuchi, Shigeru Koyano, Hiroshi Doi, Misako Kunii
Publikováno v:
Molecular Brain, Vol 14, Iss 1, Pp 1-10 (2021)
Molecular Brain
Molecular Brain
Intracellular aggregates are a common pathological hallmark of neurodegenerative diseases such as polyglutamine (polyQ) diseases, amyotrophic lateral sclerosis (ALS), Parkinson’s disease (PD), and multiple system atrophy (MSA). Aggregates are mainl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e21f19dd0ea57309a03c1fd3de23cff9
https://doaj.org/article/28ec26773d1e475c9691a8fbd146d1e6
https://doaj.org/article/28ec26773d1e475c9691a8fbd146d1e6