Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Miki Kamiyama"'
Autor:
Toko Shinkai, Kouji Masumoto, Miki Kamiyama, Yudai Goto, Yasunari Tanaka, Kentaro Ono, Tomohiro Aoyama, Kazuki Shirane, Hinako Horiguchi, Nao Tanaka, Takato Sasaki, Fumiko Chiba, Takahiro Jimbo, Yasuhisa Urita, Naoya Sakamoto
Publikováno v:
Journal of Pediatric Surgery Case Reports, Vol 91, Iss , Pp 102573- (2023)
Background: Stenosis of the permanent tracheostoma causes the patient reduced quality of life. We herein report a treatment of permanent tracheostomal stenosis with local steroid injection after laryngotracheal separation in a patient with severe mot
Externí odkaz:
https://doaj.org/article/6afe1c643be24d93b45e3aa3b38598e1
Autor:
Mahak Singhal, Nicolas Gengenbacher, Silvia La Porta, Stephanie Gehrs, Jingjing Shi, Miki Kamiyama, Diane M Bodenmiller, Anthony Fischl, Benjamin Schieb, Eva Besemfelder, Sudhakar Chintharlapalli, Hellmut G Augustin
Publikováno v:
EMBO Molecular Medicine, Vol 12, Iss 6, Pp 1-9 (2020)
Abstract The angiopoietin (Ang)–Tie pathway has been intensely pursued as candidate second‐generation anti‐angiogenic target. While much of the translational work has focused on the ligand Ang2, the clinical efficacy of Ang2‐targeting drugs i
Externí odkaz:
https://doaj.org/article/890060853bf4435fbf75eea21ec4070f
Autor:
Miki Kamiyama, Yoshihiro Hayakawa, Natsuki Furukawa, Takehiro Sato, Makoto Fujimoto, Kohsuke Takeda, Hidenori Ichijo, Takeru Odawara, Hiroki Ryuno, Tetsuro Watabe, Yasuhiro Yoshimatsu, Isao Naguro, Michaela Prchal-Murphy, Kosuke Fuse, Veronika Sexl, Hideaki Tahara
Publikováno v:
Cancer Science
Tumor metastasis is the leading cause of death worldwide and involves an extremely complex process composed of multiple steps. Our previous study demonstrated that apoptosis signal‐regulating kinase 1 (ASK1) deficiency in mice attenuates tumor meta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::da4dd71d070ba779a7bf8f802d326fac
http://europepmc.org/articles/PMC8019214
http://europepmc.org/articles/PMC8019214
Autor:
Martin R. Sprick, Carolin Mogler, Mathias Heikenwalder, Stephen E. Moss, Jeroen Krijgsveld, Matthias Schlesner, David Kallenberg, Mahak Singhal, Hellmut G. Augustin, Barbara Leuchs, Ling Hai, Shubhada Rajabhau Kulkarni, Junhao Hu, Danijela Heide, Bianca J. Kuhn, Stephanie F. Preuß, John A. Greenwood, Nicolas Gengenbacher, Eva Besemfelder, Andreas Trumpp, Ashik Ahmed Abdul Pari, Elisa Espinet, Miki Kamiyama, Carlotta Camilli
Publikováno v:
Science Translational Medicine. 13
Metastasis is the primary cause of cancer-related mortality. Tumor cell interactions with cells of the vessel wall are decisive and potentially rate-limiting for metastasis. The molecular nature of this cross-talk is, beyond candidate gene approaches
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c83359326057a69aeb1abdc8de17fdcf
https://doi.org/10.1126/scitranslmed.abe6805
https://doi.org/10.1126/scitranslmed.abe6805
Publikováno v:
YAKUGAKU ZASSHI. 139:743-751
Stress-responsive signaling pathways convert cellular stresses into various physiological responses, such as cell proliferation, apoptosis, and inflammation. Signal pathway dysfunction thus induces abnormal cellular behaviors that may lead to tumorig
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10a48314160548b689d4afccf0723560
https://doi.org/10.1248/yakushi.18-00185-2
https://doi.org/10.1248/yakushi.18-00185-2
Autor:
Miki Kamiyama, Hellmut G. Augustin
Publikováno v:
Cancer research. 81(1)
Angiopoietin (ANPGT)–TIE signaling serves as a critical regulator of vessel maturation controlling vascular quiescence, maintenance, and homeostasis (primarily through ANGPT1–TIE2 signaling), as well as enabling vascular plasticity and responsive
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba0b15a852e3affc78cafa572e2e037a
https://pubmed.ncbi.nlm.nih.gov/33037065
https://pubmed.ncbi.nlm.nih.gov/33037065
Autor:
Isao Naguro, Takehiro Sato, Shogo Tamura, Hidenori Ichijo, Kei Takahashi, Kohei Miyazono, Yoshihiro Hayakawa, Shogo Ehata, Miki Kamiyama, Katsue Suzuki-Inoue, Toshiaki Shirai, Kohsuke Takeda
Publikováno v:
Cell Death & Differentiation. 24:2066-2076
Tumor metastasis is the major cause of deaths in cancer patients and is modulated by intertwined stress-responsive signaling cascades. Here we demonstrate that deletion of stress-responsive apoptosis signal-regulating kinase 1 (Ask1) in platelets res
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::fb2f987306ab554038722235ca669578
https://doi.org/10.1038/cdd.2017.114
https://doi.org/10.1038/cdd.2017.114
Over the course of tumor progression, tumor cells in primary lesions spawn their progenies to initiate metastatic dissemination and colonization, resulting in successful tumor metastasis. More than...
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1e78d3476fdb081b2c31c14e1e4f6569
https://europepmc.org/articles/PMC5884391/
https://europepmc.org/articles/PMC5884391/
In vivo gene manipulation reveals the impact of stress-responsive MAPK pathways on tumor progression
Publikováno v:
Cancer Science
It has been widely accepted that tumor cells and normal stromal cells in the host environment coordinately modulate tumor progression. Mitogen-activated protein kinase pathways are the representative stress-responsive cascades that exert proper cellu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7962c40c150592576770182fbf9b8844
http://europepmc.org/articles/PMC4520628
http://europepmc.org/articles/PMC4520628
Publikováno v:
Advances in biological regulation. 66
Cancer is a major problem in public health and is one of the leading causes of mortality worldwide. Many types of cancer cells exhibit aberrant cellular signal transduction in response to stress, which often leads to oncogenesis. Mitogen-activated pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d09a1a712d77398b5c5c4e355579ead0
https://pubmed.ncbi.nlm.nih.gov/28552579
https://pubmed.ncbi.nlm.nih.gov/28552579